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1
A Key Role for Lysine Residues in Amyloid β-Protein Folding, Assembly, and Toxicity
A Key Role for Lysine Residues in Amyloid β-Protein Folding, Assembly, and Toxicity
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A Key Role for Lysine Residues in Amyloid β-Protein Folding, Assembly, and Toxicity

Sinha, Sharmistha ; Lopes, Dahabada H. J ; Bitan, Gal

ACS chemical neuroscience, 2012-06, Vol.3 (6), p.473-481 [Periódico revisado por pares]

United States: American Chemical Society

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2
Peripherally derived angiotensin converting enzyme-enhanced macrophages alleviate Alzheimer-related disease
Peripherally derived angiotensin converting enzyme-enhanced macrophages alleviate Alzheimer-related disease
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Peripherally derived angiotensin converting enzyme-enhanced macrophages alleviate Alzheimer-related disease

Koronyo-Hamaoui, Maya ; Sheyn, Julia ; Hayden, Eric Y ; Li, Songlin ; Fuchs, Dieu-Trang ; Regis, Giovanna C ; Lopes, Dahabada H J ; Black, Keith L ; Bernstein, Kenneth E ; Teplow, David B ; Fuchs, Sebastien ; Koronyo, Yosef ; Rentsendorj, Altan

Brain (London, England : 1878), 2020-01, Vol.143 (1), p.336-358 [Periódico revisado por pares]

England: Oxford University Press

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3
Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia
Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia
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Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia

Marques-Lopes, J ; Pinto, M ; Pinho, D ; Morato, M ; Patinha, D ; Albino-Teixeira, A ; Tavares, I

Neuroscience, 2009-02, Vol.158 (4), p.1301-1310 [Periódico revisado por pares]

Amsterdam: Elsevier Ltd

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4
Decrease in the expression of N-methyl-D-aspartate receptors in the nucleus tractus solitarii induces antinociception and increases blood pressure
Decrease in the expression of N-methyl-D-aspartate receptors in the nucleus tractus solitarii induces antinociception and increases blood pressure
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Decrease in the expression of N-methyl-D-aspartate receptors in the nucleus tractus solitarii induces antinociception and increases blood pressure

Marques-Lopes, J. ; Martins, I. ; Pinho, D. ; Morato, M. ; Wilson, S.P. ; Albino-Teixeira, A. ; Tavares, I.

Journal of neuroscience research, 2012-02, Vol.90 (2), p.356-366 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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5
Physical training exerts neuroprotective effects in the regulation of neurochemical factors in an animal model of Parkinson’s disease
Physical training exerts neuroprotective effects in the regulation of neurochemical factors in an animal model of Parkinson’s disease
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Physical training exerts neuroprotective effects in the regulation of neurochemical factors in an animal model of Parkinson’s disease

Tuon, T ; Valvassori, S.S ; LOPES-BORGES, J ; Luciano, T ; Trom, C.B ; Silva, L.A ; Quevedo, J ; Souza, C.T ; Lira, F.S ; Pinho, R.A

Neuroscience, 2012-12, Vol.227, p.305-312 [Periódico revisado por pares]

Amsterdam: Elsevier Ltd

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6
Sex differences in NMDA GluN1 plasticity in rostral ventrolateral medulla neurons containing corticotropin-releasing factor type 1 receptor following slow-pressor angiotensin II hypertension
Sex differences in NMDA GluN1 plasticity in rostral ventrolateral medulla neurons containing corticotropin-releasing factor type 1 receptor following slow-pressor angiotensin II hypertension
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Sex differences in NMDA GluN1 plasticity in rostral ventrolateral medulla neurons containing corticotropin-releasing factor type 1 receptor following slow-pressor angiotensin II hypertension

Van Kempen, T.A ; Dodos, M ; Woods, C ; Marques-Lopes, J ; Justice, N.J ; Iadecola, C ; Pickel, V.M ; Glass, M.J ; Milner, T.A

Neuroscience, 2015-10, Vol.307, p.83-97 [Periódico revisado por pares]

United States: Elsevier Ltd

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7
Cell Cycle Re-Entry in Alzheimers Disease: A Major Neuropathological Characteristic?
Cell Cycle Re-Entry in Alzheimers Disease: A Major Neuropathological Characteristic?
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Cell Cycle Re-Entry in Alzheimers Disease: A Major Neuropathological Characteristic?

Lopes, J. ; Oliveira, C. ; Agostinho, P.

Current Alzheimer research, 2009-06, Vol.6 (3), p.205-212 [Periódico revisado por pares]

Bentham Science Publishers Ltd

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8
Distribution of angiotensin type 1a receptor-containing cells in the brains of bacterial artificial chromosome transgenic mice
Distribution of angiotensin type 1a receptor-containing cells in the brains of bacterial artificial chromosome transgenic mice
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Distribution of angiotensin type 1a receptor-containing cells in the brains of bacterial artificial chromosome transgenic mice

Gonzalez, A.D ; Wang, G ; Waters, E.M ; Gonzales, K.L ; Speth, R.C ; Van Kempen, T.A ; Marques-Lopes, J ; Young, C.N ; Butler, S.D ; Davisson, R.L ; Iadecola, C ; Pickel, V.M ; Pierce, J.P ; Milner, T.A

Neuroscience, 2012-12, Vol.226, p.489-509 [Periódico revisado por pares]

Amsterdam: Elsevier Ltd

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9
Antisense peptide nucleic acid‐mediated knockdown of the p75 neurotrophin receptor delays motor neuron disease in mutant SOD1 transgenic mice
Antisense peptide nucleic acid‐mediated knockdown of the p75 neurotrophin receptor delays motor neuron disease in mutant SOD1 transgenic mice
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Antisense peptide nucleic acid‐mediated knockdown of the p75 neurotrophin receptor delays motor neuron disease in mutant SOD1 transgenic mice

Turner, Bradley J. ; Cheah, Irwin K. ; Macfarlane, Katherine J. ; Lopes, Elizabeth C. ; Petratos, Steven ; Langford, Steven J. ; Cheema, Surindar S.

Journal of neurochemistry, 2003-11, Vol.87 (3), p.752-763 [Periódico revisado por pares]

Oxford, UK: Blackwell Science Ltd

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10
Chemical sensory deafferentation abolishes hypothalamic pituitary activation induced by noxious stimulation or electroacupuncture but only decreases that caused by immobilization stress. A c- fos study
Chemical sensory deafferentation abolishes hypothalamic pituitary activation induced by noxious stimulation or electroacupuncture but only decreases that caused by immobilization stress. A c- fos study
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Chemical sensory deafferentation abolishes hypothalamic pituitary activation induced by noxious stimulation or electroacupuncture but only decreases that caused by immobilization stress. A c- fos study

Pan, B ; Castro-Lopes, J.M ; Coimbra, A

Neuroscience, 1997-06, Vol.78 (4), p.1059-1068 [Periódico revisado por pares]

Oxford: Elsevier Ltd

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