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1
Neutrophil recruitment and articular hyperalgesia in antigen-induced arthritis are modulated by the cholinergic anti-inflammatory pathway
Neutrophil recruitment and articular hyperalgesia in antigen-induced arthritis are modulated by the cholinergic anti-inflammatory pathway
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Neutrophil recruitment and articular hyperalgesia in antigen-induced arthritis are modulated by the cholinergic anti-inflammatory pathway

Alexandre Kanashiro Jhimmy Talbot; Raphael S Peres; Larissa G Pinto; Gabriel S Bassi; Thiago Mattar Cunha; Fernando de Queiroz Cunha

Basic and Clinical Pharmacology and Toxicology West Sussex v. 119, n. 5, p. 453-457, 2016

West Sussex 2016

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2839830 Estantes deslizantes )(Acessar)

2
Expression and activity of NOD1 and NOD2/RIPK2 signalling in mononuclear cells from patients with rheumatoid arthritis
Expression and activity of NOD1 and NOD2/RIPK2 signalling in mononuclear cells from patients with rheumatoid arthritis
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Expression and activity of NOD1 and NOD2/RIPK2 signalling in mononuclear cells from patients with rheumatoid arthritis

R. F. O. Franca S. M Vieira; J Talbot; R. S Peres; L. G Pinto; Dario Simões Zamboni; Paulo Louzada Júnior; Fernando de Queiroz Cunha; Thiago Mattar Cunha

Scandinavian Journal of Rheumatology Abingdon v. 45, n. 1, p. 8-12, 2016

Abingdon 2016

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2752633 Estantes Deslizantes )(Acessar)

3
Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes
Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes
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Succinate receptor deficiency attenuates arthritis by reducing dendritic cell traffic and expansion of Th17 cells in the lymph nodes

André L. Saraiva Flávio P Veras; Raphael S Peres; Jhimmy Talbot; Kalil A. de Lima; João P Luiz; José M Carballido; Thiago Mattar Cunha; Fernando de Queiroz Cunha; Bernhard Ryffel; José Carlos Farias Alves Filho

The FASEB Journal Bethesda v. 32, n. 12, p. 6550-6558, 2018

Bethesda 2018

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2939385 Estantes Deslizantes )(Acessar)

4
Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis
Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis
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Citrullinated human fibrinogen triggers arthritis through an inflammatory response mediated by IL-23/IL-17 immune axis

André Lopes Saraiva Raphael Sanches Peres; Flávio Protasio Veras; Jhimmy Talbot; Kalil Alves de Lima; João Paulo Mesquita Luiz; Thiago Mattar Cunha; Paulo Louzada-Junior; Fernando Queiróz Cunha; José Carlos Alves-Filho

International Immunopharmacology London v. 101, part. B, art. 108363, 2021

London 2021

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 3085979 Acervo Digital )(Acessar)

5
Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production
Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production
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Joint production of IL-22 participates in the initial phase of antigen-induced arthritis through IL-1β production

Larissa G. Pinto Jhimmy Talbot; Raphael S Peres; Rafael F Franca; Sérgio Henrique Ferreira; Bernhard Ryffel; José Carlos Farias Alves Filho; Florêncio Figueiredo; Thiago Mattar Cunha; Fernando de Queiroz Cunha

Arthritis Research & Therapy London v. 17, art. 235, 2015

London 2015

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2746811 Estantes Deslizantes )(Acessar)

6
TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism
TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism
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TGFβ1 signaling sustains aryl hydrocarbon receptor (AHR) expression and restrains the pathogenic potential of TH17 cells by an AHR-independent mechanism

Kalil Alves de Lima Paula Barbim Donate; Jhimmy Talbot; Marcela Davoli-Ferreira; Raphael Sanches Peres; Thiago Mattar Cunha; José Carlos Farias Alves Filho; Fernando de Queiroz Cunha

Cell Death and Disease London v. 9, art. 1130, 2018

London 2018

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2936690 Estantes Deslizantes )(Acessar)

7
CCR2 expression in neutrophils plays a critical role in their migration into the joints in rheumatoid arthritis
CCR2 expression in neutrophils plays a critical role in their migration into the joints in rheumatoid arthritis
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CCR2 expression in neutrophils plays a critical role in their migration into the joints in rheumatoid arthritis

Jhimmy Talbot Francine Junta Bianchini; Daniele C Nascimento; Rene D. R Oliveira; Fabricio O Souto; Larissa G Pinto; Raphael S Peres; Jaqueline R Silva; Sergio C. L Almeida; Paulo Louzada Júnior; Thiago Mattar Cunha; Fernando de Queiroz Cunha; José Carlos Farias Alves Filho

Arthritis & Rheumatology Hoboken v. 67, n. 7, p. 1751-1759, 2015

Hoboken 2015

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2746902 Estantes Deslizantes )(Acessar)

8
Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells
Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells
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Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells

Jhimmy Talbot Raphael S Peres; Larissa G Pinto; Rene D. R Oliveira; Kalil A Lima; Paula B Donate; Jaqueline R Silva; Bernard Ryffel; Thiago Mattar Cunha; José Carlos Farias Alves Filho; Foo Y Liew; Paulo Louzada Júnior; Fernando de Queiroz Cunha

Arthritis Research & Therapy London v.20, n.1, art. 119, 2018

London 2018

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2934191 Estantes Deslizantes )(Acessar)

9
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis
TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis
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TGF-β signalling defect is linked to low CD39 expression on regulatory T cells and methotrexate resistance in rheumatoid arthritis

Raphael Sanches Peres Paula Barbim Donate; Jhimmy Talbot; Nerry Tatiana Cecílio; Patricia Rolim Mendonça Lobo; Caio Cavalcante Machado; Kalil William Alves de Lima; Renê Donizete Ribeiro de Oliveira; Vanessa Carregaro; Helder Takashi Imoto Nakaya; Thiago Mattar Cunha; José Carlos Farias Alves Filho; Foo Yew Liew; Paulo Louzada Júnior; Fernando Queiróz Cunha

Journal of Autoimmunity London v. 90, p. 49-58, 2018

London 2018

Localização: FMRP - Fac. Medicina de Ribeirão Preto    (pcd 2874186 Estantes Deslizantes )(Acessar)

10
Low expression of CD39 on regulatory T cells as a biomarker for resistance to methotrexate therapy in rheumatoid arthritis
Low expression of CD39 on regulatory T cells as a biomarker for resistance to methotrexate therapy in rheumatoid arthritis
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Low expression of CD39 on regulatory T cells as a biomarker for resistance to methotrexate therapy in rheumatoid arthritis

Raphael Sanches Peres Foo Y Liew; Jhimmy Talbot; Vanessa Carregaro; Rene D Oliveira; Sergio L Almeida; Rafael F. O França; Paula B Donate; Larissa G Pinto; Flavia I. S Ferreira; Diego L Costa; Daniel P Demarque; Dayana Rubio Gouvea; Norberto Peporine Lopes; Regina Helena Costa Queiroz; João Santana da Silva; Florêncio Figueiredo; José Carlos Alves-Filho; Thiago Mattar Cunha; Sérgio Henrique Ferreira; Paulo Louzada Junior; Fernando de Queiroz Cunha

Proceedings of the National Academy of Sciences of the United States of America Washington v. 112, n. 8, p. 2509-2514, 2015

Washington 2015

Localização: FCFRP - Fac. Ciên. Farm. Ribeirão Preto    (pcd 2708338 Estantes Deslizantes )(Acessar)

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Deste Autor:

  1. Cunha, F
  2. Peres, R
  3. Talbot, J
  4. Cunha, T
  5. Alves Filho, J

Neste Assunto:

  1. Artrite Reumatoide
  2. Hidrocarbonetos
  3. Neutrófilos
  4. Inflamação
  5. Modelos Animais De Doenças

  • Realização: ABCD-USP USP   Logos de Redes Sociais: Freepik

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