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Refinado por: assunto: Epigenetics remover
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Insulator dysfunction and oncogene activation in IDH mutant gliomas
Insulator dysfunction and oncogene activation in IDH mutant gliomas
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Insulator dysfunction and oncogene activation in IDH mutant gliomas

Flavahan, William A ; Drier, Yotam ; Liau, Brian B ; Gillespie, Shawn M ; Venteicher, Andrew S ; Stemmer-Rachamimov, Anat O ; Suvà, Mario L ; Bernstein, Bradley E

Nature (London), 2016-01, Vol.529 (7584), p.110-114 [Periódico revisado por pares]

England: Nature Publishing Group

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2
Isocitrate dehydrogenase mutations in myeloid malignancies
Isocitrate dehydrogenase mutations in myeloid malignancies
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Isocitrate dehydrogenase mutations in myeloid malignancies

Medeiros, B C ; Fathi, A T ; DiNardo, C D ; Pollyea, D A ; Chan, S M ; Swords, R

Leukemia, 2017-02, Vol.31 (2), p.272-281 [Periódico revisado por pares]

England: Nature Publishing Group

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3
Isocitrate dehydrogenase variants in cancer — Cellular consequences and therapeutic opportunities
Isocitrate dehydrogenase variants in cancer — Cellular consequences and therapeutic opportunities
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Isocitrate dehydrogenase variants in cancer — Cellular consequences and therapeutic opportunities

Liu, Shuang ; Cadoux-Hudson, Tom ; Schofield, Christopher J.

Current opinion in chemical biology, 2020-08, Vol.57, p.122-134 [Periódico revisado por pares]

England: Elsevier Ltd

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4
Treatment with a Small Molecule Mutant IDH1 Inhibitor Suppresses Tumorigenic Activity and Decreases Production of the Oncometabolite 2-Hydroxyglutarate in Human Chondrosarcoma Cells
Treatment with a Small Molecule Mutant IDH1 Inhibitor Suppresses Tumorigenic Activity and Decreases Production of the Oncometabolite 2-Hydroxyglutarate in Human Chondrosarcoma Cells
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Treatment with a Small Molecule Mutant IDH1 Inhibitor Suppresses Tumorigenic Activity and Decreases Production of the Oncometabolite 2-Hydroxyglutarate in Human Chondrosarcoma Cells

Li, Luyuan ; Paz, Ana C ; Wilky, Breelyn A ; Johnson, Britt ; Galoian, Karina ; Rosenberg, Andrew ; Hu, Guozhi ; Tinoco, Gabriel ; Bodamer, Olaf ; Trent, Jonathan C Mills, Ken

PloS one, 2015-09, Vol.10 (9), p.e0133813-e0133813 [Periódico revisado por pares]

United States: Public Library of Science

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5
Natural and synthetic 2-oxoglutarate derivatives are substrates for oncogenic variants of human isocitrate dehydrogenase 1 and 2
Natural and synthetic 2-oxoglutarate derivatives are substrates for oncogenic variants of human isocitrate dehydrogenase 1 and 2
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Natural and synthetic 2-oxoglutarate derivatives are substrates for oncogenic variants of human isocitrate dehydrogenase 1 and 2

Liu, Xiao ; Reinbold, Raphael ; Liu, Shuang ; Herold, Ryan A. ; Rabe, Patrick ; Duclos, Stéphanie ; Yadav, Rahul B. ; Abboud, Martine I. ; Thieffine, Sandrine ; Armstrong, Fraser A. ; Brewitz, Lennart ; Schofield, Christopher J.

The Journal of biological chemistry, 2023-02, Vol.299 (2), p.102873-102873, Article 102873 [Periódico revisado por pares]

United States: Elsevier Inc

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6
Loss of 5-Hydroxymethylcytosine Is an Epigenetic Hallmark of Melanoma
Loss of 5-Hydroxymethylcytosine Is an Epigenetic Hallmark of Melanoma
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Loss of 5-Hydroxymethylcytosine Is an Epigenetic Hallmark of Melanoma

Lian, Christine Guo ; Xu, Yufei ; Ceol, Craig ; Wu, Feizhen ; Larson, Allison ; Dresser, Karen ; Xu, Wenqi ; Tan, Li ; Hu, Yeguang ; Zhan, Qian ; Lee, Chung-wei ; Hu, Di ; Lian, Bill Q. ; Kleffel, Sonja ; Yang, Yijun ; Neiswender, James ; Khorasani, Abraham J. ; Fang, Rui ; Lezcano, Cecilia ; Duncan, Lyn M. ; Scolyer, Richard A. ; Thompson, John F. ; Kakavand, Hojabr ; Houvras, Yariv ; Zon, Leonard I. ; Mihm, Martin C. ; Kaiser, Ursula B. ; Schatton, Tobias ; Woda, Bruce A. ; Murphy, George F. ; Shi, Yujiang G.

Cell, 2012-09, Vol.150 (6), p.1135-1146 [Periódico revisado por pares]

United States: Elsevier Inc

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7
Distinct and opposite effects of leukemogenic Idh and Tet2 mutations in hematopoietic stem and progenitor cells
Distinct and opposite effects of leukemogenic Idh and Tet2 mutations in hematopoietic stem and progenitor cells
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Distinct and opposite effects of leukemogenic Idh and Tet2 mutations in hematopoietic stem and progenitor cells

Fortin, Jerome ; Chiang, Ming-Feng ; Meydan, Cem ; Foox, Jonathan ; Ramachandran, Parameswaran ; Leca, Julie ; Lemonnier, François ; Li, Wanda Y ; Gams, Miki S ; Sakamoto, Takashi ; Chu, Mandy ; Tobin, Chantal ; Laugesen, Eric ; Robinson, Troy M ; You-Ten, Annick ; Butler, Daniel J ; Berger, Thorsten ; Minden, Mark D ; Levine, Ross L ; Guidos, Cynthia J ; Melnick, Ari M ; Mason, Christopher E ; Mak, Tak W

Proceedings of the National Academy of Sciences - PNAS, 2023-01, Vol.120 (4), p.e2208176120-e2208176120 [Periódico revisado por pares]

United States: National Academy of Sciences

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8
Efficient induction of differentiation and growth inhibition in IDH1 mutant glioma cells by the DNMT Inhibitor Decitabine
Efficient induction of differentiation and growth inhibition in IDH1 mutant glioma cells by the DNMT Inhibitor Decitabine
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Efficient induction of differentiation and growth inhibition in IDH1 mutant glioma cells by the DNMT Inhibitor Decitabine

Turcan, Sevin ; Fabius, Armida W M ; Borodovsky, Alexandra ; Pedraza, Alicia ; Brennan, Cameron ; Huse, Jason ; Viale, Agnes ; Riggins, Gregory J ; Chan, Timothy A

Oncotarget, 2013-10, Vol.4 (10), p.1729-1736

United States: Impact Journals LLC

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9
Metabolic reprogramming and epithelial‐to‐mesenchymal transition in cancer
Metabolic reprogramming and epithelial‐to‐mesenchymal transition in cancer
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Metabolic reprogramming and epithelial‐to‐mesenchymal transition in cancer

Sciacovelli, Marco ; Frezza, Christian

The FEBS journal, 2017-10, Vol.284 (19), p.3132-3144 [Periódico revisado por pares]

England: Blackwell Publishing Ltd

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10
IDH1 mutant glioma is preferentially sensitive to the HDAC inhibitor panobinostat
IDH1 mutant glioma is preferentially sensitive to the HDAC inhibitor panobinostat
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IDH1 mutant glioma is preferentially sensitive to the HDAC inhibitor panobinostat

Sears, Thomas K. ; Horbinski, Craig M. ; Woolard, Kevin D.

Journal of neuro-oncology, 2021-09, Vol.154 (2), p.159-170 [Periódico revisado por pares]

New York: Springer US

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