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Refinado por: Nome da Publicação: Atherosclerosis remover
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Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps
Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps
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Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps

Raul Dias dos Santos Filho Luca Valenti; Stefano Romeo

Atherosclerosis v. 282, p. 110-120, 2019

Clare 2019

Acesso online. A biblioteca também possui exemplares impressos.

2
Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps
Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps
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Does nonalcoholic fatty liver disease cause cardiovascular disease? Current knowledge and gaps

Santos, Raul D. ; Valenti, Luca ; Romeo, Stefano

Atherosclerosis, 2019-03, Vol.282, p.110-120 [Periódico revisado por pares]

Ireland: Elsevier B.V

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3
Progression of carotid vascular damage and cardiovascular events in non-alcoholic fatty liver disease patients compared to the general population during 10 years of follow-up
Progression of carotid vascular damage and cardiovascular events in non-alcoholic fatty liver disease patients compared to the general population during 10 years of follow-up
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Progression of carotid vascular damage and cardiovascular events in non-alcoholic fatty liver disease patients compared to the general population during 10 years of follow-up

Fracanzani, Anna Ludovica ; Tiraboschi, Silvia ; Pisano, Giuseppina ; Consonni, Dario ; Baragetti, Andrea ; Bertelli, Cristina ; Norata, Danilo ; Valenti, Luca ; Grigore, Liliana ; Porzio, Marianna ; Catapano, Alberico ; Fargion, Silvia

Atherosclerosis, 2016-03, Vol.246, p.208-213 [Periódico revisado por pares]

Ireland: Elsevier Ireland Ltd

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4
Downregulation of interleukin 32 reduce intracellular triglyceride levels in liver spheroids
Downregulation of interleukin 32 reduce intracellular triglyceride levels in liver spheroids
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Downregulation of interleukin 32 reduce intracellular triglyceride levels in liver spheroids

Sasidharan, K. ; Caddeo, A.M. ; Jamialahmadi, O. ; Baselli, G. ; Malvestiti, F. ; Pingitore, P. ; Valenti, L. ; Romeo, S.

Atherosclerosis, 2021-08, Vol.331, p.e143-e143 [Periódico revisado por pares]

Elsevier B.V

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5
Mutation in candidate genes account for a small minority of hypobetalipoproteinemias and NGS analysis support polygenicity in mutation-negative patients
Mutation in candidate genes account for a small minority of hypobetalipoproteinemias and NGS analysis support polygenicity in mutation-negative patients
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Mutation in candidate genes account for a small minority of hypobetalipoproteinemias and NGS analysis support polygenicity in mutation-negative patients

Giammanco, A. ; Scrimali, C. ; Spina, R. ; Ingrassia, V. ; Brucato, F. ; Valenti, V. ; Cefalù, A.B. ; Misiano, G. ; Altieri, G.I. ; Noto, D. ; Barbagallo, C.M. ; Ganci, A. ; Fayer, F. ; Averna, M.

Atherosclerosis, 2020-12, Vol.315, p.e45-e45 [Periódico revisado por pares]

Elsevier B.V

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6
PCSK9-D374Y mediated LDL-R degradation can be functionally inhibited by EGF-A and truncated EGF-A peptides. An in vitro study
PCSK9-D374Y mediated LDL-R degradation can be functionally inhibited by EGF-A and truncated EGF-A peptides. An in vitro study
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PCSK9-D374Y mediated LDL-R degradation can be functionally inhibited by EGF-A and truncated EGF-A peptides. An in vitro study

Scrimali, C. ; Spina, R. ; Ingrassia, V. ; Brucato, F. ; Altieri, G.I. ; Noto, D. ; Valenti, V. ; Misiano, G. ; Giammanco, A. ; Fayer, F. ; Barbagallo, C.M. ; Ganci, A. ; Cefalù, A.B. ; Averna, M.

Atherosclerosis, 2020-12, Vol.315, p.e258-e258 [Periódico revisado por pares]

Elsevier B.V

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7
Leptin and resistin affect PCSK9 expression: In vitro and in vivo evidence
Leptin and resistin affect PCSK9 expression: In vitro and in vivo evidence
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Leptin and resistin affect PCSK9 expression: In vitro and in vivo evidence

Macchi, C. ; Botta, M. ; Marchiano, S. ; Dongiovanni, P. ; Valenti, L. ; Cicero, A. ; Magni, P. ; Corsini, A. ; Ferri, N. ; Ruscica, M.

Atherosclerosis, 2018-08, Vol.275, p.e18-e18 [Periódico revisado por pares]

Elsevier B.V

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8
Identification Of P.Leu167Del Apoe Gene Mutation By Next Generation Sequencing In A Large Hypercholesterolemic Family
Identification Of P.Leu167Del Apoe Gene Mutation By Next Generation Sequencing In A Large Hypercholesterolemic Family
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Identification Of P.Leu167Del Apoe Gene Mutation By Next Generation Sequencing In A Large Hypercholesterolemic Family

Scrimali, C. ; Spina, R. ; Ingrassia, V. ; Cefalù, A.B. ; Brucato, F. ; Misiano, G. ; Valenti, V. ; Noto, D. ; Altieri, G.I. ; Fayer, F. ; Giammanco, A. ; Barbagallo, C. ; Averna, M.

Atherosclerosis, 2019-08, Vol.287, p.e213-e213 [Periódico revisado por pares]

Elsevier B.V

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9
Metabolic disturbances and risk of cancer in the 25 years follow-up of the “Ventimiglia Heart Study” epidemiological project
Metabolic disturbances and risk of cancer in the 25 years follow-up of the “Ventimiglia Heart Study” epidemiological project
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Metabolic disturbances and risk of cancer in the 25 years follow-up of the “Ventimiglia Heart Study” epidemiological project

Noto, D ; Cefalù, A.B ; Barbagallo, C.M ; Giammanco, A ; Fayer, F ; Palesano, O ; Altieri, G.I ; Spina, R ; Valenti, V ; Pagano, M ; Cavera, G ; Averna, M.R

Atherosclerosis, 2016-09, Vol.252, p.e86-e86 [Periódico revisado por pares]

Elsevier B.V

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10
Identification and characterization of two novel mutations in the LPL gene causing type I hyperlipoproteinemia
Identification and characterization of two novel mutations in the LPL gene causing type I hyperlipoproteinemia
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Identification and characterization of two novel mutations in the LPL gene causing type I hyperlipoproteinemia

Pingitore, P ; Lepore, S.M ; Pirazzi, C ; Mancina, R.M ; Motta, B.M ; Valenti, L ; Berge, K.E ; Retterstøl, K ; Leren, T.P ; Wiklund, O ; Romeo, S

Atherosclerosis, 2016-09, Vol.252, p.e73-e74 [Periódico revisado por pares]

Elsevier B.V

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