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Genetically engineered mouse models of PI3K signaling in breast cancer
Genetically engineered mouse models of PI3K signaling in breast cancer
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Genetically engineered mouse models of PI3K signaling in breast cancer

Klarenbeek, Sjoerd ; van Miltenburg, Martine H. ; Jonkers, Jos

Molecular oncology, 2013-04, Vol.7 (2), p.146-164 [Periódico revisado por pares]

United States: Elsevier B.V

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2
Biochemical characterization of a novel oxidatively stable, halotolerant, and high‐alkaline subtilisin from Alkalihalobacillus okhensis Kh10‐101T
Biochemical characterization of a novel oxidatively stable, halotolerant, and high‐alkaline subtilisin from Alkalihalobacillus okhensis Kh10‐101T
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Biochemical characterization of a novel oxidatively stable, halotolerant, and high‐alkaline subtilisin from Alkalihalobacillus okhensis Kh10‐101T

Falkenberg, Fabian ; Rahba, Jade ; Fischer, David ; Bott, Michael ; Bongaerts, Johannes ; Siegert, Petra

FEBS open bio, 2022-10, Vol.12 (10), p.1729-1746 [Periódico revisado por pares]

Amsterdam: John Wiley & Sons, Inc

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3
Emerging functions of the EGFR in cancer
Emerging functions of the EGFR in cancer
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Emerging functions of the EGFR in cancer

Sigismund, Sara ; Avanzato, Daniele ; Lanzetti, Letizia

Molecular oncology, 2018-01, Vol.12 (1), p.3-20 [Periódico revisado por pares]

United States: John Wiley & Sons, Inc

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4
Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment
Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment
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Induced PTF1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment

Jakubison, Brad L. ; Schweickert, Patrick G. ; Moser, Sarah E. ; Yang, Yi ; Gao, Hongyu ; Scully, Kathleen ; Itkin‐Ansari, Pamela ; Liu, Yunlong ; Konieczny, Stephen F.

Molecular oncology, 2018-06, Vol.12 (7), p.1104-1124 [Periódico revisado por pares]

United States: John Wiley & Sons, Inc

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5
High HER2 protein levels correlate with increased survival in breast cancer patients treated with anti-HER2 therapy
High HER2 protein levels correlate with increased survival in breast cancer patients treated with anti-HER2 therapy
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High HER2 protein levels correlate with increased survival in breast cancer patients treated with anti-HER2 therapy

Nuciforo, Paolo ; Thyparambil, Sheeno ; Aura, Claudia ; Garrido-Castro, Ana ; Vilaro, Marta ; Peg, Vicente ; Jimenez, José ; Vicario, Rocio ; Cecchi, Fabiola ; Hoos, William ; Burrows, Jon ; Hembrough, Todd ; Ferreres, Juan Carles ; Perez-Garcia, José ; Arribas, Joaquin ; Cortes, Javier ; Scaltriti, Maurizio

Molecular oncology, 2016-01, Vol.10 (1), p.138-147 [Periódico revisado por pares]

United States: Elsevier B.V

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6
Platycodin D inhibits autophagy and increases glioblastoma cell death via LDLR upregulation
Platycodin D inhibits autophagy and increases glioblastoma cell death via LDLR upregulation
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Platycodin D inhibits autophagy and increases glioblastoma cell death via LDLR upregulation

Lee, Sol Ji ; Choi, Yu‐Jeong ; Kim, Hyo In ; Moon, Hyo Eun ; Paek, Sun Ha ; Kim, Tai Young ; Ko, Seong‐Gyu

Molecular oncology, 2022-01, Vol.16 (1), p.250-268 [Periódico revisado por pares]

United States: John Wiley & Sons, Inc

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7
IL-8 induces miR-424-5p expression and modulates SOCS2/STAT5 signaling pathway in oral squamous cell carcinoma
IL-8 induces miR-424-5p expression and modulates SOCS2/STAT5 signaling pathway in oral squamous cell carcinoma
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IL-8 induces miR-424-5p expression and modulates SOCS2/STAT5 signaling pathway in oral squamous cell carcinoma

Peng, Hsuan-Yu ; Jiang, Shih-Sheng ; Hsiao, Jenn-Ren ; Hsiao, Michael ; Hsu, Yuan-Ming ; Wu, Guan-Hsun ; Chang, Wei-Min ; Chang, Jang-Yang ; Jin, Shiow-Lian Catherine ; Shiah, Shine-Gwo

Molecular oncology, 2016-06, Vol.10 (6), p.895-909 [Periódico revisado por pares]

United States: Elsevier B.V

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8
Hepsin promotes breast tumor growth signaling via the TGFβ‐EGFR axis
Hepsin promotes breast tumor growth signaling via the TGFβ‐EGFR axis
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Hepsin promotes breast tumor growth signaling via the TGFβ‐EGFR axis

Belitškin, Denis ; Munne, Pauliina ; Pant, Shishir M. ; Anttila, Johanna M. ; Suleymanova, Ilida ; Belitškina, Kati ; Kirchhofer, Daniel ; Janetka, James ; Käsper, Taivo ; Jalil, Sami ; Pouwels, Jeroen ; Tervonen, Topi A. ; Klefström, Juha

Molecular oncology, 2024-03, Vol.18 (3), p.547-561 [Periódico revisado por pares]

United States: John Wiley & Sons, Inc

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9
Dexmedetomidine inhibits the growth and metastasis of esophageal cancer cells by down‐regulation of lncRNA MALAT1
Dexmedetomidine inhibits the growth and metastasis of esophageal cancer cells by down‐regulation of lncRNA MALAT1
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Dexmedetomidine inhibits the growth and metastasis of esophageal cancer cells by down‐regulation of lncRNA MALAT1

Zhang, Wei ; Zhang, Li ; Cai, Xiao‐Jun ; Li, Dong ; Cao, Feng‐Jun ; Zuo, Zhi‐Gang ; Song, Ying ; Yu, Xiong‐Jie ; Liu, Shan

The Kaohsiung journal of medical sciences, 2022-06, Vol.38 (6), p.585-593 [Periódico revisado por pares]

BP, Asia: Wiley Publishing Asia Pty Ltd

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10
Induced PTF 1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment
Induced PTF 1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment
Material Type:
Artigo 		Adicionar ao Meu Espaço

Induced PTF 1a expression in pancreatic ductal adenocarcinoma cells activates acinar gene networks, reduces tumorigenic properties, and sensitizes cells to gemcitabine treatment

Jakubison, Brad L. ; Schweickert, Patrick G. ; Moser, Sarah E. ; Yang, Yi ; Gao, Hongyu ; Scully, Kathleen ; Itkin‐Ansari, Pamela ; Liu, Yunlong ; Konieczny, Stephen F.

Molecular oncology, 2018-06, Vol.12 (7), p.1104-1124 [Periódico revisado por pares]

Hoboken: John Wiley & Sons, Inc

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