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1
The crucial roles of apolipoproteins E and C-III in apoB lipoprotein metabolism in normolipidemia and hypertriglyceridemia
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Artigo
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The crucial roles of apolipoproteins E and C-III in apoB lipoprotein metabolism in normolipidemia and hypertriglyceridemia

Sacks, Frank M

Current opinion in lipidology, 2015-02, Vol.26 (1), p.56-63 [Periódico revisado por pares]

England

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2
Lipoprotein lipase: From gene to atherosclerosis
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Artigo
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Lipoprotein lipase: From gene to atherosclerosis

Li, Yuan ; He, Ping-Ping ; Zhang, Da-Wei ; Zheng, Xi-Long ; Cayabyab, Fracisco S ; Yin, Wei-Dong ; Tang, Chao-Ke

Atherosclerosis, 2014-12, Vol.237 (2), p.597-608 [Periódico revisado por pares]

Ireland: Elsevier Ireland Ltd

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3
Apolipoprotein C-III and the Metabolic Basis for Hypertriglyceridemia and the Dense Low-Density Lipoprotein Phenotype
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Artigo
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Apolipoprotein C-III and the Metabolic Basis for Hypertriglyceridemia and the Dense Low-Density Lipoprotein Phenotype

CHUNYU ZHENG ; KHOO, Christina ; FURTADO, Jeremy ; SACKS, Frank M

Circulation (New York, N.Y.), 2010-04, Vol.121 (15), p.1722-1734 [Periódico revisado por pares]

Hagerstown, MD: Lippincott Williams & Wilkins

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4
The effect of PPAR-α agonism on apolipoprotein metabolism in humans
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Artigo
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The effect of PPAR-α agonism on apolipoprotein metabolism in humans

Shah, Ashish ; Rader, Daniel J ; Millar, John S

Atherosclerosis, 2010-05, Vol.210 (1), p.35-40 [Periódico revisado por pares]

Amsterdam: Elsevier Ireland Ltd

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5
Neutrophil Elastase Inhibition by Sivelestat (ONO-5046) Attenuates AngII-Induced Abdominal Aortic Aneurysms in Apolipoprotein E-Deficient Mice
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Artigo
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Neutrophil Elastase Inhibition by Sivelestat (ONO-5046) Attenuates AngII-Induced Abdominal Aortic Aneurysms in Apolipoprotein E-Deficient Mice

Hada, Yoshiko ; Uchida, Haruhito A ; Okamoto, Shugo ; Otaka, Nozomu ; Katayama, Katsuyoshi ; Subramanian, Venkateswaran ; Daugherty, Alan ; Wada, Jun

American journal of hypertension, 2024-04, Vol.37 (5), p.349-357 [Periódico revisado por pares]

United States

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6
Atherogenic Lipoprotein(a) Increases Vascular Glycolysis, Thereby Facilitating Inflammation and Leukocyte Extravasation
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Atherogenic Lipoprotein(a) Increases Vascular Glycolysis, Thereby Facilitating Inflammation and Leukocyte Extravasation

Schnitzler, Johan G ; Hoogeveen, Renate M ; Ali, Lubna ; Prange, Koen H.M ; Waissi, Farahnaz ; van Weeghel, Michel ; Bachmann, Julian C ; Versloot, Miranda ; Borrelli, Matthew J ; Yeang, Calvin ; De Kleijn, Dominique P.V ; Houtkooper, Riekelt H ; Koschinsky, Marlys L ; de Winther, Menno P.J ; Groen, Albert K ; Witztum, Joseph L ; Tsimikas, Sotirios ; Stroes, Erik S.G ; Kroon, Jeffrey

Circulation research, 2020-05, Vol.126 (10), p.1346-1359 [Periódico revisado por pares]

United States: American Heart Association, Inc

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7
Serum Amyloid A Is an Exchangeable Apolipoprotein
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Serum Amyloid A Is an Exchangeable Apolipoprotein

Wilson, Patricia G ; Thompson, Joel C ; Shridas, Preetha ; McNamara, Patrick J ; de Beer, Maria C ; de Beer, Frederick C ; Webb, Nancy R ; Tannock, Lisa R

Arteriosclerosis, thrombosis, and vascular biology, 2018-08, Vol.38 (8), p.1890-1900 [Periódico revisado por pares]

United States: American Heart Association, Inc

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8
TRIF adaptor signaling is important in abdominal aortic aneurysm formation
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Artigo
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TRIF adaptor signaling is important in abdominal aortic aneurysm formation

Vorkapic, Emina ; Lundberg, Anna M ; Mäyränpää, Mikko I ; Eriksson, Per ; Wågsäter, Dick

Atherosclerosis, 2015-08, Vol.241 (2), p.561-568 [Periódico revisado por pares]

Ireland: Elsevier Ireland Ltd

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9
Apo(a) and ApoB Interact Noncovalently Within Hepatocytes: Implications for Regulation of Lp(a) Levels by Modulation of ApoB Secretion
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Apo(a) and ApoB Interact Noncovalently Within Hepatocytes: Implications for Regulation of Lp(a) Levels by Modulation of ApoB Secretion

Youssef, Amer ; Clark, Justin R ; Marcovina, Santica M ; Boffa, Michael B ; Koschinsky, Marlys L

Arteriosclerosis, thrombosis, and vascular biology, 2022-03, Vol.42 (3), p.289-304 [Periódico revisado por pares]

United States

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10
High-density lipoproteins (HDL) are present in stenotic aortic valves and may interfere with the mechanisms of valvular calcification
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High-density lipoproteins (HDL) are present in stenotic aortic valves and may interfere with the mechanisms of valvular calcification

Lommi, Jaakko I ; Kovanen, Petri T ; Jauhiainen, Matti ; Lee-Rueckert, Miriam ; Kupari, Markku ; Helske, Satu

Atherosclerosis, 2011-12, Vol.219 (2), p.538-544 [Periódico revisado por pares]

Amsterdam: Elsevier Ireland Ltd

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