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Refinado por: autor: Carvalho, M remover assunto: Ratos remover
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1
The heart gone wrong stabilization of cardiac function the b1-kinin receptor antagonism reduces the hypertrophic effects of angiotensin II in the cardiovascular system of rats
The heart gone wrong stabilization of cardiac function the b1-kinin receptor antagonism reduces the hypertrophic effects of angiotensin II in the cardiovascular system of rats
Material Type:
Artigo de Congresso 		Adicionar ao Meu Espaço

The heart gone wrong stabilization of cardiac function the b1-kinin receptor antagonism reduces the hypertrophic effects of angiotensin II in the cardiovascular system of rats

Graziela S. Ceravolo T. J Costa; Eliana Hiromi Akamine; Rita de Cassia Aleixo Tostes; Maria Helena Catelli de Carvalho; Zuleica Bruno Fortes; World Congress of Basic and Clinical Pharmacology (16. 2010 Copenhagen)

Basic and Clinical Pharmacology and Toxicology Oxford v. 107, suppl. 1, p. 126, res. 2953, 2010

Oxford 2010

Item não circula. Consulte sua biblioteca.(Acessar)

2
Endothelium in health and disease role of protein disulfide isomerase in angiotensin II induced contraction in vascular smooth muscle cells from spontaneously hypertensive rats
Endothelium in health and disease role of protein disulfide isomerase in angiotensin II induced contraction in vascular smooth muscle cells from spontaneously hypertensive rats
Material Type:
Artigo de Congresso 		Adicionar ao Meu Espaço

Endothelium in health and disease role of protein disulfide isomerase in angiotensin II induced contraction in vascular smooth muscle cells from spontaneously hypertensive rats

Livia de Lucca Camargo A. C. D Androwiki; G. S Ceravolo; A. Z Chignalia; Alexandre Denadai-Souza; Marcelo Nicolás Muscará; Zuleica Bruno Fortes; Maria Helena Catelli de Carvalho; M Janizewski; Lucia Rossetti Lopes; World Congress of Basic and Clinical Pharmacology (16. 2010 Copenhagen)

Basic and Clinical Pharmacology and Toxicology Oxford v. 107, suppl. 1, p. 253, res. 1482, 2010

Oxford 2010

Item não circula. Consulte sua biblioteca.(Acessar)

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