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Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis
Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis
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Sabutoclax (BI97C1) and BI112D1, Putative Inhibitors of MCL-1, Induce Mitochondrial Fragmentation Either Upstream of or Independent of Apoptosis

Varadarajan, Shankar ; Butterworth, Michael ; Wei, Jun ; Pellecchia, Maurizio ; Dinsdale, David ; Cohen, Gerald M

Neoplasia (New York, N.Y.), 2013-05, Vol.15 (5), p.568-IN22 [Periódico revisado por pares]

United States: Elsevier Inc

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2
Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome
Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome
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Heat-shock protein 70 inhibits apoptosis by preventing recruitment of procaspase-9 to the Apaf-1 apoptosome

Beere, Helen M ; Wolf, Beni B ; Cain, Kelvin ; Mosser, Dick D ; Mahboubi, Artin ; Kuwana, Tomomi ; Tailor, Pankaj ; Morimoto, Richard I ; Cohen, Gerald M ; Green, Douglas R

Nature cell biology, 2000-08, Vol.2 (8), p.469-475 [Periódico revisado por pares]

England: Nature Publishing Group

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3
Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis
Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis
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Endoplasmic reticulum membrane reorganization is regulated by ionic homeostasis

Varadarajan, Shankar ; Tanaka, Kayoko ; Smalley, Joshua L ; Bampton, Edward T W ; Pellecchia, Maurizio ; Dinsdale, David ; Willars, Gary B ; Cohen, Gerald M Holowka, David

PloS one, 2013-02, Vol.8 (2), p.e56603 [Periódico revisado por pares]

United States: Public Library of Science

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4
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L
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BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-X L

Greaves, Georgia ; Milani, Mateus ; Butterworth, Michael ; Carter, Rachel J ; Byrne, Dominic P ; Eyers, Patrick A ; Luo, Xu ; Cohen, Gerald M ; Varadarajan, Shankar

Cell death and differentiation, 2019-06, Vol.26 (6), p.1037 [Periódico revisado por pares]

England

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5
BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cells
BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cells
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BH3 profiling and a toolkit of BH3-mimetic drugs predict anti-apoptotic dependence of cancer cells

Butterworth, Michael ; Pettitt, Andrew ; Varadarajan, Shankar ; Cohen, Gerald M

British journal of cancer, 2016-03, Vol.114 (6), p.638-641 [Periódico revisado por pares]

England: Nature Publishing Group

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6
Conformational change and mitochondrial translocation of Bax accompany proteasome inhibitor-induced apoptosis of chronic lymphocytic leukemic cells
Conformational change and mitochondrial translocation of Bax accompany proteasome inhibitor-induced apoptosis of chronic lymphocytic leukemic cells
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Conformational change and mitochondrial translocation of Bax accompany proteasome inhibitor-induced apoptosis of chronic lymphocytic leukemic cells

DEWSON, Grant ; SNOWDEN, Roger T ; ALMOND, Jason B ; DYER, Martin J. S ; COHEN, Gerald M

Oncogene, 2003-05, Vol.22 (17), p.2643-2654 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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7
Mechanisms of resistance to TRAIL-induced apoptosis in primary B cell chronic lymphocytic leukaemia
Mechanisms of resistance to TRAIL-induced apoptosis in primary B cell chronic lymphocytic leukaemia
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Mechanisms of resistance to TRAIL-induced apoptosis in primary B cell chronic lymphocytic leukaemia

MACFARLANE, Marion ; HARPER, Nicholas ; SNOWDEN, Roger T ; DYER, Martin J. S ; BARNETT, Georgina A ; PRINGLE, J. Howard ; COHEN, Gerald M

Oncogene, 2002-10, Vol.21 (44), p.6809-6818 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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8
Apoptosis, in human monocytic THP.1 cells, results in the release of cytochrome c from mitochondria prior to their ultracondensation, formation of outer membrane discontinuities and reduction in inner membrane potential
Apoptosis, in human monocytic THP.1 cells, results in the release of cytochrome c from mitochondria prior to their ultracondensation, formation of outer membrane discontinuities and reduction in inner membrane potential
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Apoptosis, in human monocytic THP.1 cells, results in the release of cytochrome c from mitochondria prior to their ultracondensation, formation of outer membrane discontinuities and reduction in inner membrane potential

Zhuang, J ; Dinsdale, D ; Cohen, G M

Cell death and differentiation, 1998-11, Vol.5 (11), p.953-962 [Periódico revisado por pares]

England

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9
Apoptosis in hematopoietic cells (FL5.12) caused by interleukin-3 withdrawal: relationship to caspase activity and the loss of glutathione
Apoptosis in hematopoietic cells (FL5.12) caused by interleukin-3 withdrawal: relationship to caspase activity and the loss of glutathione
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Apoptosis in hematopoietic cells (FL5.12) caused by interleukin-3 withdrawal: relationship to caspase activity and the loss of glutathione

Bojes, H K ; Feng, X ; Kehrer, J P ; Cohen, G M

Cell death and differentiation, 1999-01, Vol.6 (1), p.61-70 [Periódico revisado por pares]

England

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10
The adenomatous polyposis coli protein and retinoblastoma protein are cleaved early in apoptosis and are potential substrates for caspases
The adenomatous polyposis coli protein and retinoblastoma protein are cleaved early in apoptosis and are potential substrates for caspases
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The adenomatous polyposis coli protein and retinoblastoma protein are cleaved early in apoptosis and are potential substrates for caspases

Browne, S J ; MacFarlane, M ; Cohen, G M ; Paraskeva, C

Cell death and differentiation, 1998-03, Vol.5 (3), p.206-213 [Periódico revisado por pares]

England

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