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1
Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liver
Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liver
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Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liver

Volkmann, Xandra ; Fischer, Ute ; Bahr, Matthias J. ; Ott, Michael ; Lehner, Frank ; MacFarlane, Marion ; Cohen, Gerald M. ; Manns, Michael P. ; Schulze‐Osthoff, Klaus ; Bantel, Heike

Hepatology (Baltimore, Md.), 2007-11, Vol.46 (5), p.1498-1508 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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2
ABT‐199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets
ABT‐199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets
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ABT‐199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets

Vogler, Meike ; Dinsdale, David ; Dyer, Martin J. S. ; Cohen, Gerald M.

British journal of haematology, 2013-10, Vol.163 (1), p.139-142 [Periódico revisado por pares]

Oxford: Blackwell

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3
TRAIL signals to apoptosis in chronic lymphocytic leukaemia cells primarily through TRAIL‐R1 whereas cross‐linked agonistic TRAIL‐R2 antibodies facilitate signalling via TRAIL‐R2
TRAIL signals to apoptosis in chronic lymphocytic leukaemia cells primarily through TRAIL‐R1 whereas cross‐linked agonistic TRAIL‐R2 antibodies facilitate signalling via TRAIL‐R2
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TRAIL signals to apoptosis in chronic lymphocytic leukaemia cells primarily through TRAIL‐R1 whereas cross‐linked agonistic TRAIL‐R2 antibodies facilitate signalling via TRAIL‐R2

Natoni, Alessandro ; MacFarlane, Marion ; Inoue, Satoshi ; Walewska, Renata ; Majid, Aneela ; Knee, Deborah ; Stover, David R. ; Dyer, Martin J. S. ; Cohen, Gerald M.

British journal of haematology, 2007-11, Vol.139 (4), p.568-577 [Periódico revisado por pares]

Oxford, UK: Blackwell Publishing Ltd

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4
ABT ‐199 selectively inhibits BCL 2 but not BCL 2 L 1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets
ABT ‐199 selectively inhibits BCL 2 but not BCL 2 L 1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets
Material Type:
Artigo 		Adicionar ao Meu Espaço

ABT ‐199 selectively inhibits BCL 2 but not BCL 2 L 1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not platelets

Vogler, Meike ; Dinsdale, David ; Dyer, Martin J. S. ; Cohen, Gerald M.

British journal of haematology, 2013-10, Vol.163 (1), p.139-142 [Periódico revisado por pares]

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5
Processing/activation of CPP32‐like proteases is involved in transforming growth factor β1–induced apoptosis in rat hepatocytes
Processing/activation of CPP32‐like proteases is involved in transforming growth factor β1–induced apoptosis in rat hepatocytes
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Processing/activation of CPP32‐like proteases is involved in transforming growth factor β1–induced apoptosis in rat hepatocytes

Inayat‐Hussain, S H ; Couet, C ; Cohen, G M ; Cain, K

Hepatology (Baltimore, Md.), 1997-06, Vol.25 (6), p.1516-1526 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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6
"First-pass" metabolism of paracetamol in rat liver
"First-pass" metabolism of paracetamol in rat liver
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"First-pass" metabolism of paracetamol in rat liver

COHEN, GERALD M. ; BAKKE, OLAV M. ; DAVIES, DONALD S.

Journal of pharmacy and pharmacology, 1974-05, Vol.26 (5), p.348-351 [Periódico revisado por pares]

Oxford, UK: Blackwell Publishing Ltd

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7
Large interindividual variations in metabolism of benzo(α)pyrene by peripheral lung tissue from lung cancer patients
Large interindividual variations in metabolism of benzo(α)pyrene by peripheral lung tissue from lung cancer patients
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Large interindividual variations in metabolism of benzo(α)pyrene by peripheral lung tissue from lung cancer patients

Cohen, Gerald M. ; Mehta, Rekha ; Meredith‐Brown, M.

International journal of cancer, 1979-08, Vol.24 (2), p.129-133 [Periódico revisado por pares]

New York: Wiley Subscription Services, Inc., A Wiley Company

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8
Defining the disulphide stress response in Streptomyces coelicolor A3(2): identification of the sigmaR regulon
Defining the disulphide stress response in Streptomyces coelicolor A3(2): identification of the sigmaR regulon
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Defining the disulphide stress response in Streptomyces coelicolor A3(2): identification of the sigmaR regulon

Paget, Mark ; Molle, Virginie ; Cohen, Gerald M ; Aharonowitz, Yair ; Buttner, Mark

Molecular microbiology, 2001-11, Vol.42 (4), p.1007-1020 [Periódico revisado por pares]

Wiley

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9
Mechanisms of toxicity of 2- and 5-hydroxy-1,4-naphthoquinone; absence of a role for redox cycling in the toxicity of 2-hydroxy-1,4-naphthoquinone to isolated hepatocytes
Mechanisms of toxicity of 2- and 5-hydroxy-1,4-naphthoquinone; absence of a role for redox cycling in the toxicity of 2-hydroxy-1,4-naphthoquinone to isolated hepatocytes
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Mechanisms of toxicity of 2- and 5-hydroxy-1,4-naphthoquinone; absence of a role for redox cycling in the toxicity of 2-hydroxy-1,4-naphthoquinone to isolated hepatocytes

Doherty, Mary D'Arcy ; Rodgers, Adrian ; Cohen, Gerald M.

Journal of applied toxicology, 1987-04, Vol.7 (2), p.123-129 [Periódico revisado por pares]

Chichester: John Wiley & Sons, Ltd

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10
In vivo formation of benzo (a) pyrene diol epoxide‐deoxyadenosine adducts in the skin of mice susceptible to benzo (a) pyrene‐induced carcinogenesis
In vivo formation of benzo (a) pyrene diol epoxide‐deoxyadenosine adducts in the skin of mice susceptible to benzo (a) pyrene‐induced carcinogenesis
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In vivo formation of benzo (a) pyrene diol epoxide‐deoxyadenosine adducts in the skin of mice susceptible to benzo (a) pyrene‐induced carcinogenesis

Ashurst, Stephen W. ; Cohen, Gerald M.

International journal of cancer, 1981-03, Vol.27 (3), p.357-364 [Periódico revisado por pares]

New York: Wiley Subscription Services, Inc., A Wiley Company

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