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1 |
Material Type: Artigo
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Concurrent up-regulation of BCL-XL and BCL2A1 induces approximately 1000-fold resistance to ABT-737 in chronic lymphocytic leukemiaVogler, Meike ; Butterworth, Michael ; Majid, Aneela ; Walewska, Renata J. ; Sun, Xiao-Ming ; Dyer, Martin J.S. ; Cohen, Gerald M.Blood, 2009-04, Vol.113 (18), p.4403-4413 [Periódico revisado por pares]Washington, DC: Elsevier IncTexto completo disponível |
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Material Type: Artigo
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BCL2/BCL-XL inhibition induces apoptosis, disrupts cellular calcium homeostasis, and prevents platelet activationVogler, Meike ; Hamali, Hassan A. ; Sun, Xiao-Ming ; Bampton, Edward T.W. ; Dinsdale, David ; Snowden, Roger T. ; Dyer, Martin J.S. ; Goodall, Alison H. ; Cohen, Gerald M.Blood, 2011-06, Vol.117 (26), p.7145-7154 [Periódico revisado por pares]Washington, DC: Elsevier IncTexto completo disponível |
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Material Type: Artigo
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Increased hepatotoxicity of tumor necrosis factor–related apoptosis‐inducing ligand in diseased human liverVolkmann, Xandra ; Fischer, Ute ; Bahr, Matthias J. ; Ott, Michael ; Lehner, Frank ; MacFarlane, Marion ; Cohen, Gerald M. ; Manns, Michael P. ; Schulze‐Osthoff, Klaus ; Bantel, HeikeHepatology (Baltimore, Md.), 2007-11, Vol.46 (5), p.1498-1508 [Periódico revisado por pares]Hoboken: Wiley Subscription Services, Inc., A Wiley CompanyTexto completo disponível |
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Material Type: Artigo
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Role of NOXA and its ubiquitination in proteasome inhibitor-induced apoptosis in chronic lymphocytic leukemia cellsBAOU, Maria ; KOHLHAAS, Susan L ; BUTTERWORTH, Michael ; VOGLER, Meike ; DINSDALE, David ; WALEWSKA, Renata ; MAJID, Aneela ; ELDERING, Eric ; DYER, Martin J. S ; COHEN, Gerald MHaematologica (Roma), 2010-09, Vol.95 (9), p.1510-1518 [Periódico revisado por pares]Pavia: Ferrata Storti FoundationTexto completo disponível |
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Material Type: Artigo
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TRAIL receptor-selective mutants signal to apoptosis via TRAIL-R1 in primary lymphoid malignanciesMACFARLANE, Marion ; KOHLHAAS, Susan L ; SUTCLIFFE, Michael J ; DYER, Martin J. S ; COHEN, Gerald MCancer research (Chicago, Ill.), 2005-12, Vol.65 (24), p.11265-11270 [Periódico revisado por pares]Philadelphia, PA: American Association for Cancer ResearchTexto completo disponível |
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Material Type: Artigo
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ABT‐199 selectively inhibits BCL2 but not BCL2L1 and efficiently induces apoptosis of chronic lymphocytic leukaemic cells but not plateletsVogler, Meike ; Dinsdale, David ; Dyer, Martin J. S. ; Cohen, Gerald M.British journal of haematology, 2013-10, Vol.163 (1), p.139-142 [Periódico revisado por pares]Oxford: BlackwellTexto completo disponível |
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Material Type: Artigo
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Conformational change and mitochondrial translocation of Bax accompany proteasome inhibitor-induced apoptosis of chronic lymphocytic leukemic cellsDEWSON, Grant ; SNOWDEN, Roger T ; ALMOND, Jason B ; DYER, Martin J. S ; COHEN, Gerald MOncogene, 2003-05, Vol.22 (17), p.2643-2654 [Periódico revisado por pares]Basingstoke: Nature PublishingTexto completo disponível |
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Material Type: Artigo
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Interleukin-5 inhibits translocation of Bax to the mitochondria, cytochrome c release, and activation of caspases in human eosinophilsDewson, Grant ; Cohen, Gerald M. ; Wardlaw, Andrew J.Blood, 2001-10, Vol.98 (7), p.2239-2247 [Periódico revisado por pares]Washington, DC: Elsevier IncTexto completo disponível |
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Material Type: Artigo
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Diminished Sensitivity of Chronic Lymphocytic Leukemia Cells to ABT-737 and ABT-263 Due to Albumin Binding in BloodVOGLER, Meike ; FURDAS, Silviya D ; JUNG, Manfred ; KUWANA, Tomomi ; DYER, Martin J. S ; COHEN, Gerald MClinical cancer research, 2010-08, Vol.16 (16), p.4217-4225 [Periódico revisado por pares]Philadelphia, PA: American Association for Cancer ResearchTexto completo disponível |
10 |
Material Type: Artigo
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Mechanisms of resistance to TRAIL-induced apoptosis in primary B cell chronic lymphocytic leukaemiaMACFARLANE, Marion ; HARPER, Nicholas ; SNOWDEN, Roger T ; DYER, Martin J. S ; BARNETT, Georgina A ; PRINGLE, J. Howard ; COHEN, Gerald MOncogene, 2002-10, Vol.21 (44), p.6809-6818 [Periódico revisado por pares]Basingstoke: Nature PublishingTexto completo disponível |