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1
Signaling mediated by the NF-κB sub-units NF-κB1, NF-κB2 and c-Rel differentially regulate Helicobacter felis-induced gastric carcinogenesis in C57BL/6 mice
Signaling mediated by the NF-κB sub-units NF-κB1, NF-κB2 and c-Rel differentially regulate Helicobacter felis-induced gastric carcinogenesis in C57BL/6 mice
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Signaling mediated by the NF-κB sub-units NF-κB1, NF-κB2 and c-Rel differentially regulate Helicobacter felis-induced gastric carcinogenesis in C57BL/6 mice

Burkitt, M D ; Williams, J M ; Duckworth, C A ; O'Hara, A ; Hanedi, A ; Varro, A ; Caamaño, J H ; Pritchard, D M

Oncogene, 2013-12, Vol.32 (50), p.5563-5573 [Periódico revisado por pares]

England: Nature Publishing Group

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2
Autoantibodies against type I IFNs in humans with alternative NF-κB pathway deficiency
Autoantibodies against type I IFNs in humans with alternative NF-κB pathway deficiency
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Autoantibodies against type I IFNs in humans with alternative NF-κB pathway deficiency

Le Voyer, Tom ; Gervais, Adrian ; Rosain, Jérémie ; Rinchai, Darawan ; Hancioglu, Gonca ; Ghillani-Dalbin, Pascale ; Charuel, Jean-Luc ; Philippot, Quentin ; Gueye, Mame Sokhna ; Maglorius Renkilaraj, Majistor Raj Luxman ; Ogishi, Masato ; Soudée, Camille ; Migaud, Mélanie ; Rozenberg, Flore ; Momenilandi, Mana ; Delmonte, Ottavia M ; Müller, Gabriele ; Keller, Baerbel ; Franco Gallego, William Alexander ; Rubin, Tamar ; Emiroglu, Melike ; Parvaneh, Nima ; Aranda-Guillen, Maribel ; Berrios, David I ; Vong, Linda ; Katelaris, Constance H ; Mustillo, Peter ; Raedler, Johannes ; Bohlen, Jonathan ; Winter, Sarah ; McLean, Catriona ; Guffroy, Aurélien ; DeRisi, Joseph L ; Yu, David ; Miller, Corey ; Feng, Yi ; Béziat, Vivien ; Bustamante, Jacinta ; Pan-Hammarström, Qiang ; Holland, Steve M ; Bosticardo, Marita ; Kenney, Heather ; Slade, Charlotte A ; Boztuğ, Kaan ; Mahlaoui, Nizar ; Abraham, Roshini S ; Lougaris, Vassilios ; Hauck, Fabian ; Sediva, Anna ; Atschekzei, Faranaz ; Poli, M Cecilia ; Slatter, Mary A ; Palterer, Boaz ; Suan, Daniel ; Wong, Melanie ; Kane, Alisa ; Ma, Cindy ; Grombiříková, Hana ; Dalal, Ilan ; Aladjidi, Nathalie ; Hie, Miguel ; Lazaro, Estibaliz ; Franco, Jose ; Keles, Sevgi ; Malphettes, Marion ; Pasquet, Marlene ; Maccari, Maria Elena ; Meinhardt, Andrea ; Shahrooei, Mohammad ; Goodnow, Christopher C ; Gray, Paul E ; Belot, Alexandre ; Kuehn, Hye Sun ; Rosenzweig, Sergio D ; Miyara, Makoto ; Licciardi, Francesco ; Servettaz, Amélie ; Le Guenno, Guillaume ; Herrmann, Vera-Maria ; Sarrot-Reynauld, Françoise ; Schuetz, Catharina ; Cunningham-Rundles, Charlotte ; Tangye, Stuart G ; Sobacchi, Cristina ; Grimbacher, Bodo ; Fieschi, Claire ; Berteloot, Laureline ; Trouillet Assant, Sophie ; Neven, Benedicte ; Abel, Laurent ; Cobat, Aurélie ; Jouanguy, Emmanuelle ; Kampe, Olle ; Bastard, Paul ; Roifman, Chaim M ; Landegren, Nils ; Notarangelo, Luigi D ; Anderson, Mark S ; Casanova, Jean-Laurent ; Puel, Anne

Nature (London), 2023-11, Vol.623 (7988), p.803-813 [Periódico revisado por pares]

England: Nature Publishing Group

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3
Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency
Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency
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Biochemically deleterious human NFKB1 variants underlie an autosomal dominant form of common variable immunodeficiency

Li, Juan ; Lei, Wei-Te ; Zhang, Peng ; Rapaport, Franck ; Seeleuthner, Yoann ; Lyu, Bingnan ; Asano, Takaki ; Rosain, Jérémie ; Hammadi, Boualem ; Zhang, Yu ; Pelham, Simon J ; Spaan, András N ; Migaud, Mélanie ; Hum, David ; Bigio, Benedetta ; Chrabieh, Maya ; Béziat, Vivien ; Bustamante, Jacinta ; Zhang, Shen-Ying ; Jouanguy, Emmanuelle ; Boisson-Dupuis, Stephanie ; El Baghdadi, Jamila ; Aimanianda, Vishukumar ; Thoma, Katharina ; Fliegauf, Manfred ; Grimbacher, Bodo ; Korganow, Anne-Sophie ; Saunders, Carol ; Rao, V Koneti ; Uzel, Gulbu ; Freeman, Alexandra F ; Holland, Steven M ; Su, Helen C ; Cunningham-Rundles, Charlotte ; Fieschi, Claire ; Abel, Laurent ; Puel, Anne ; Cobat, Aurélie ; Casanova, Jean-Laurent ; Zhang, Qian ; Boisson, Bertrand

The Journal of experimental medicine, 2021-11, Vol.218 (11) [Periódico revisado por pares]

United States: Rockefeller University Press

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4
Non-canonical NFκB mutations reinforce pro-survival TNF response in multiple myeloma through an autoregulatory RelB:p50 NFκB pathway
Non-canonical NFκB mutations reinforce pro-survival TNF response in multiple myeloma through an autoregulatory RelB:p50 NFκB pathway
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Non-canonical NFκB mutations reinforce pro-survival TNF response in multiple myeloma through an autoregulatory RelB:p50 NFκB pathway

Roy, P ; Mukherjee, T ; Chatterjee, B ; Vijayaragavan, B ; Banoth, B ; Basak, S

Oncogene, 2017-03, Vol.36 (10), p.1417-1429 [Periódico revisado por pares]

England: Nature Publishing Group

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5
NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB
NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB
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NOTCH inhibits osteoblast formation in inflammatory arthritis via noncanonical NF-κB

Zhang, Hengwei ; Hilton, Matthew J ; Anolik, Jennifer H ; Welle, Stephen L ; Zhao, Chen ; Yao, Zhenqiang ; Li, Xing ; Wang, Zhiyu ; Boyce, Brendan F ; Xing, Lianping

The Journal of clinical investigation, 2014-07, Vol.124 (7), p.3200-3214 [Periódico revisado por pares]

United States: American Society for Clinical Investigation

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6
Guanine nucleotide exchange factor RABGEF1 regulates keratinocyte-intrinsic signaling to maintain skin homeostasis
Guanine nucleotide exchange factor RABGEF1 regulates keratinocyte-intrinsic signaling to maintain skin homeostasis
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Guanine nucleotide exchange factor RABGEF1 regulates keratinocyte-intrinsic signaling to maintain skin homeostasis

Marichal, Thomas ; Gaudenzio, Nicolas ; El Abbas, Sophie ; Sibilano, Riccardo ; Zurek, Oliwia ; Starkl, Philipp ; Reber, Laurent L ; Pirottin, Dimitri ; Kim, Jinah ; Chambon, Pierre ; Roers, Axel ; Antoine, Nadine ; Kawakami, Yuko ; Kawakami, Toshiaki ; Bureau, Fabrice ; Tam, See-Ying ; Tsai, Mindy ; Galli, Stephen J

The Journal of clinical investigation, 2016-12, Vol.126 (12), p.4497-4515 [Periódico revisado por pares]

United States: American Society for Clinical Investigation

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7
Characterization of a high TNF-α phenotype in children with moderate-to-severe asthma
Characterization of a high TNF-α phenotype in children with moderate-to-severe asthma
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Characterization of a high TNF-α phenotype in children with moderate-to-severe asthma

Brown, Sheena D., PhD ; Brown, Lou Ann, PhD ; Stephenson, Susan, PhD ; Dodds, Jennifer C., PhD ; Douglas, Shaneka L., BS ; Qu, Hongyan, MS ; Fitzpatrick, Anne M., PhD

Journal of allergy and clinical immunology, 2015-06, Vol.135 (6), p.1651-1654 [Periódico revisado por pares]

United States: Elsevier Inc

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8
miR-15b reduces amyloid-β accumulation in SH-SY5Y cell line through targetting NF-κB signaling and BACE1
miR-15b reduces amyloid-β accumulation in SH-SY5Y cell line through targetting NF-κB signaling and BACE1
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miR-15b reduces amyloid-β accumulation in SH-SY5Y cell line through targetting NF-κB signaling and BACE1

Li, Juan ; Wang, Haitao

Bioscience reports, 2018-12, Vol.38 (6) [Periódico revisado por pares]

England: Portland Press Ltd

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9
Inhibitory role of Gas6 in intestinal tumorigenesis
Inhibitory role of Gas6 in intestinal tumorigenesis
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Inhibitory role of Gas6 in intestinal tumorigenesis

Akitake-Kawano, Reiko ; Seno, Hiroshi ; Nakatsuji, Masato ; Kimura, Yuto ; Nakanishi, Yuki ; Yoshioka, Takuto ; Kanda, Keitaro ; Kawada, Mayumi ; Kawada, Kenji ; Sakai, Yoshiharu ; Chiba, Tsutomu

Carcinogenesis (New York), 2013-07, Vol.34 (7), p.1567-1574 [Periódico revisado por pares]

England

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10
NF-κB is a critical mediator of BRCA1-induced chemoresistance
NF-κB is a critical mediator of BRCA1-induced chemoresistance
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NF-κB is a critical mediator of BRCA1-induced chemoresistance

Harte, Mary T ; Gorski, Julia J ; Savage, Kienan I ; Purcell, James W ; Barros, Eliana M ; Burn, Philip M ; McFarlane, Cheryl ; Mullan, Paul B ; Kennedy, Richard D ; Perkins, Neil D ; Harkin, D Paul

Oncogene, 2014-02, Vol.33 (6), p.713-723 [Periódico revisado por pares]

England: Nature Publishing Group

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