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Refinado por: Base de dados/Biblioteca: Open Access: PubMed Central remover Nome da Publicação: The American Journal Of Pathology remover
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1
Neutrophil-Epithelial Interactions: A Double-Edged Sword
Neutrophil-Epithelial Interactions: A Double-Edged Sword
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Neutrophil-Epithelial Interactions: A Double-Edged Sword

Parkos, Charles A

The American journal of pathology, 2016-06, Vol.186 (6), p.1404-1416 [Periódico revisado por pares]

United States: American Society for Investigative Pathology

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2
Wound Macrophages as Key Regulators of Repair: Origin, Phenotype, and Function
Wound Macrophages as Key Regulators of Repair: Origin, Phenotype, and Function
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Wound Macrophages as Key Regulators of Repair: Origin, Phenotype, and Function

BRANCATO, Samielle K ; ALBINA, Jorge E

The American journal of pathology, 2011, Vol.178 (1), p.19-25 [Periódico revisado por pares]

Bethesda, MD: American Society for Investigative Pathology

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3
Targeted Recycling of the Lateral Border Recycling Compartment Precedes Adherens Junction Dissociation during Transendothelial Migration
Targeted Recycling of the Lateral Border Recycling Compartment Precedes Adherens Junction Dissociation during Transendothelial Migration
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Targeted Recycling of the Lateral Border Recycling Compartment Precedes Adherens Junction Dissociation during Transendothelial Migration

Gonzalez, Annette M ; Cyrus, Bita F ; Muller, William A

The American journal of pathology, 2016-05, Vol.186 (5), p.1387-1402 [Periódico revisado por pares]

United States: Elsevier Inc

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4
Carbon Monoxide–Releasing Molecule-401 Suppresses Polymorphonuclear Leukocyte Migratory Potential by Modulating F-Actin Dynamics
Carbon Monoxide–Releasing Molecule-401 Suppresses Polymorphonuclear Leukocyte Migratory Potential by Modulating F-Actin Dynamics
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Carbon Monoxide–Releasing Molecule-401 Suppresses Polymorphonuclear Leukocyte Migratory Potential by Modulating F-Actin Dynamics

Inoue, Ken ; Patterson, Eric K ; Capretta, Alfredo ; Lawendy, Abdel R ; Fraser, Douglas D ; Cepinskas, Gediminas

The American journal of pathology, 2017-05, Vol.187 (5), p.1121-1133 [Periódico revisado por pares]

United States: Elsevier Inc

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5
Nontypeable Haemophilus influenzae –Promoted Proliferation of KRas-Induced Early Adenomatous Lesions Is Completely Dependent on Toll-Like Receptor Signaling
Nontypeable Haemophilus influenzae –Promoted Proliferation of KRas-Induced Early Adenomatous Lesions Is Completely Dependent on Toll-Like Receptor Signaling
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Nontypeable Haemophilus influenzae –Promoted Proliferation of KRas-Induced Early Adenomatous Lesions Is Completely Dependent on Toll-Like Receptor Signaling

Jungnickel, Christopher ; Schnabel, Philipp A ; Bohle, Rainer ; Wiewrodt, Rainer ; Herr, Christian ; Bals, Robert ; Beisswenger, Christoph

The American journal of pathology, 2017-05, Vol.187 (5), p.973-979 [Periódico revisado por pares]

United States: Elsevier Inc

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6
Biomaterials Selectively Modulate Interactions between Human Blood-Derived Polymorphonuclear Leukocytes and Monocytes
Biomaterials Selectively Modulate Interactions between Human Blood-Derived Polymorphonuclear Leukocytes and Monocytes
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Biomaterials Selectively Modulate Interactions between Human Blood-Derived Polymorphonuclear Leukocytes and Monocytes

Cohen, Hannah C ; Joyce, Evan J ; Kao, W. John

The American journal of pathology, 2013-06, Vol.182 (6), p.2180-2190 [Periódico revisado por pares]

United States: Elsevier Inc

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7
Role of Leukocyte Elastase in Preventing Cellular Re-Colonization of the Mural Thrombus
Role of Leukocyte Elastase in Preventing Cellular Re-Colonization of the Mural Thrombus
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Role of Leukocyte Elastase in Preventing Cellular Re-Colonization of the Mural Thrombus

Fontaine, Vincent ; Touat, Ziad ; Mtairag, El Mostafa ; Vranckx, Roger ; Louedec, Liliane ; Houard, Xavier ; Andreassian, Bernard ; Sebbag, Uriel ; Palombi, Tonino ; Jacob, Marie-Paule ; Meilhac, Olivier ; Michel, Jean-Baptiste

The American journal of pathology, 2004-06, Vol.164 (6), p.2077-2087 [Periódico revisado por pares]

Bethesda, MD: Elsevier Inc

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8
LPS-Induced Murine Systemic Inflammation Is Driven by Parenchymal Cell Activation and Exclusively Predicted by Early MCP-1 Plasma Levels
LPS-Induced Murine Systemic Inflammation Is Driven by Parenchymal Cell Activation and Exclusively Predicted by Early MCP-1 Plasma Levels
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LPS-Induced Murine Systemic Inflammation Is Driven by Parenchymal Cell Activation and Exclusively Predicted by Early MCP-1 Plasma Levels

Juskewitch, Justin E ; Knudsen, Bruce E ; Platt, Jeffrey L ; Nath, Karl A ; Knutson, Keith L ; Brunn, Gregory J ; Grande, Joseph P

The American journal of pathology, 2012, Vol.180 (1), p.32-40 [Periódico revisado por pares]

Bethesda, MD: Elsevier Inc

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9
Tumor-Derived Granulocyte-Macrophage Colony-Stimulating Factor and Granulocyte Colony-Stimulating Factor Prolong the Survival of Neutrophils Infiltrating Bronchoalveolar Subtype Pulmonary Adenocarcinoma
Tumor-Derived Granulocyte-Macrophage Colony-Stimulating Factor and Granulocyte Colony-Stimulating Factor Prolong the Survival of Neutrophils Infiltrating Bronchoalveolar Subtype Pulmonary Adenocarcinoma
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Tumor-Derived Granulocyte-Macrophage Colony-Stimulating Factor and Granulocyte Colony-Stimulating Factor Prolong the Survival of Neutrophils Infiltrating Bronchoalveolar Subtype Pulmonary Adenocarcinoma

Wislez, Marie ; Fleury-Feith, Jocelyne ; Rabbe, Nathalie ; Moreau, Joelle ; Cesari, Danielle ; Milleron, Bernard ; Mayaud, Charles ; Antoine, Martine ; Soler, Paul ; Cadranel, Jacques

The American journal of pathology, 2001-10, Vol.159 (4), p.1423-1433 [Periódico revisado por pares]

Bethesda, MD: Elsevier Inc

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10
Resolvin D1 Receptor Stereoselectivity and Regulation of Inflammation and Proresolving MicroRNAs
Resolvin D1 Receptor Stereoselectivity and Regulation of Inflammation and Proresolving MicroRNAs
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Resolvin D1 Receptor Stereoselectivity and Regulation of Inflammation and Proresolving MicroRNAs

Krishnamoorthy, Sriram ; Recchiuti, Antonio ; Chiang, Nan ; Fredman, Gabrielle ; Serhan, Charles N

The American journal of pathology, 2012-05, Vol.180 (5), p.2018-2027 [Periódico revisado por pares]

Bethesda, MD: Elsevier Inc

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