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In vitro activity of Bcr-Abl inhibitors AMN107 and BMS-354825 against clinically relevant imatinib-resistant Abl kinase domain mutants
In vitro activity of Bcr-Abl inhibitors AMN107 and BMS-354825 against clinically relevant imatinib-resistant Abl kinase domain mutants
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In vitro activity of Bcr-Abl inhibitors AMN107 and BMS-354825 against clinically relevant imatinib-resistant Abl kinase domain mutants

O'HARE, Thomas ; WALTERS, Denise K ; DRUKER, Brian J ; STOFFREGEN, Eric P ; TAIPING JIA ; MANLEY, Paul W ; MESTAN, Jürgen ; COWAN-JACOB, Sandra W ; LEE, Francis Y ; HEINRICH, Michael C ; DEININGER, Michael W. N

Cancer research (Chicago, Ill.), 2005-06, Vol.65 (11), p.4500-4505 [Periódico revisado por pares]

Philadelphia, PA: American Association for Cancer Research

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2
Novel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environment
Novel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environment
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Novel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environment

VanderVen, Brian C ; Fahey, Ruth J ; Lee, Wonsik ; Liu, Yancheng ; Abramovitch, Robert B ; Memmott, Christine ; Crowe, Adam M ; Eltis, Lindsay D ; Perola, Emanuele ; Deininger, David D ; Wang, Tiansheng ; Locher, Christopher P ; Russell, David G Boshoff, Helena Ingrid

PLoS pathogens, 2015-02, Vol.11 (2), p.e1004679-e1004679 [Periódico revisado por pares]

United States: Public Library of Science

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3
CYT387, a novel JAK2 inhibitor, induces hematologic responses and normalizes inflammatory cytokines in murine myeloproliferative neoplasms
CYT387, a novel JAK2 inhibitor, induces hematologic responses and normalizes inflammatory cytokines in murine myeloproliferative neoplasms
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CYT387, a novel JAK2 inhibitor, induces hematologic responses and normalizes inflammatory cytokines in murine myeloproliferative neoplasms

Tyner, Jeffrey W. ; Bumm, Thomas G. ; Deininger, Jutta ; Wood, Lisa ; Aichberger, Karl J. ; Loriaux, Marc M. ; Druker, Brian J. ; Burns, Christopher J. ; Fantino, Emmanuelle ; Deininger, Michael W.

Blood, 2010-06, Vol.115 (25), p.5232-5240 [Periódico revisado por pares]

Washington, DC: Elsevier Inc

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4
Dasatinib (BMS-354825), a dual SRC/ABL kinase inhibitor, inhibits the kinase activity of wild-type, juxtamembrane, and activation loop mutant KIT isoforms associated with human malignancies
Dasatinib (BMS-354825), a dual SRC/ABL kinase inhibitor, inhibits the kinase activity of wild-type, juxtamembrane, and activation loop mutant KIT isoforms associated with human malignancies
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Dasatinib (BMS-354825), a dual SRC/ABL kinase inhibitor, inhibits the kinase activity of wild-type, juxtamembrane, and activation loop mutant KIT isoforms associated with human malignancies

SCHITTENHELM, Marcus M ; SHIRAGA, Sharon ; SCHROEDER, Arin ; CORBIN, Amie S ; GRIFFITH, Diana ; LEE, Francis Y ; BOKEMEYER, Carsten ; DEININGER, Michael W. N ; DRUKER, Brian J ; HEINRICH, Michael C

Cancer research (Chicago, Ill.), 2006, Vol.66 (1), p.473-481 [Periódico revisado por pares]

Philadelphia, PA: American Association for Cancer Research

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5
The molecular biology of chronic myeloid leukemia
The molecular biology of chronic myeloid leukemia
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The molecular biology of chronic myeloid leukemia

Deininger, Michael W.N. ; Goldman, John M. ; Melo, Junia V.

Blood, 2000-11, Vol.96 (10), p.3343-3356 [Periódico revisado por pares]

Washington, DC: Elsevier Inc

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6
Specific Targeted Therapy of Chronic Myelogenous Leukemia with Imatinib
Specific Targeted Therapy of Chronic Myelogenous Leukemia with Imatinib
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Specific Targeted Therapy of Chronic Myelogenous Leukemia with Imatinib

Deininger, Michael W N ; Druker, Brian J

Pharmacological reviews, 2003-09, Vol.55 (3), p.401-423 [Periódico revisado por pares]

United States: American Society for Pharmacology and Experimental Therapeutics

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7
Oncogenic CSF3R Mutations in Chronic Neutrophilic Leukemia and Atypical CML
Oncogenic CSF3R Mutations in Chronic Neutrophilic Leukemia and Atypical CML
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Oncogenic CSF3R Mutations in Chronic Neutrophilic Leukemia and Atypical CML

Maxson, Julia E ; Gotlib, Jason ; Pollyea, Daniel A ; Fleischman, Angela G ; Agarwal, Anupriya ; Eide, Christopher A ; Bottomly, Daniel ; Wilmot, Beth ; McWeeney, Shannon K ; Tognon, Cristina E ; Pond, J. Blake ; Collins, Robert H ; Goueli, Basem ; Oh, Stephen T ; Deininger, Michael W ; Chang, Bill H ; Loriaux, Marc M ; Druker, Brian J ; Tyner, Jeffrey W

The New England journal of medicine, 2013-05, Vol.368 (19), p.1781-1790 [Periódico revisado por pares]

Waltham, MA: Massachusetts Medical Society

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8
The contribution of alu elements to mutagenic DNA double-strand break repair
The contribution of alu elements to mutagenic DNA double-strand break repair
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The contribution of alu elements to mutagenic DNA double-strand break repair

Morales, Maria E ; White, Travis B ; Streva, Vincent A ; DeFreece, Cecily B ; Hedges, Dale J ; Deininger, Prescott L Jinks-Robertson, Sue

PLoS genetics, 2015-03, Vol.11 (3), p.e1005016 [Periódico revisado por pares]

United States: Public Library of Science

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9
The development of imatinib as a therapeutic agent for chronic myeloid leukemia
The development of imatinib as a therapeutic agent for chronic myeloid leukemia
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The development of imatinib as a therapeutic agent for chronic myeloid leukemia

Deininger, Michael ; Buchdunger, Elisabeth ; Druker, Brian J.

Blood, 2005-04, Vol.105 (7), p.2640-2653 [Periódico revisado por pares]

Washington, DC: Elsevier Inc

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10
Inviting instability: Transposable elements, double-strand breaks, and the maintenance of genome integrity
Inviting instability: Transposable elements, double-strand breaks, and the maintenance of genome integrity
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Inviting instability: Transposable elements, double-strand breaks, and the maintenance of genome integrity

Hedges, D J ; Deininger, P L

Mutation Research-Fundamental and Molecular Mechanisms of Mutagenesis, 2007-03, Vol.616 (1-2), p.46-59 [Periódico revisado por pares]

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