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Refinado por: assunto: Biochemistry & Molecular Biology remover
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1
Molecular Pathogenesis in Huntington’s Disease
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Molecular Pathogenesis in Huntington’s Disease

Illarioshkin, S. N. ; Klyushnikov, S. A. ; Vigont, V. A. ; Seliverstov, Yu. A. ; Kaznacheyeva, E. V.

Biochemistry (Moscow), 2018-09, Vol.83 (9), p.1030-1039 [Periódico revisado por pares]

Moscow: Pleiades Publishing

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2
Coupling endoplasmic reticulum stress to the cell death program
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Coupling endoplasmic reticulum stress to the cell death program

Rao, R V ; Ellerby, H M ; Bredesen, D E

Cell death and differentiation, 2004-04, Vol.11 (4), p.372-380 [Periódico revisado por pares]

England: Nature Publishing Group

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3
Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's disease
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Specific caspase interactions and amplification are involved in selective neuronal vulnerability in Huntington's disease

Hermel, E ; Gafni, J ; Propp, S S ; Leavitt, B R ; Wellington, C L ; Young, J E ; Hackam, A S ; Logvinova, A V ; Peel, A L ; Chen, S F ; Hook, V ; Singaraja, R ; Krajewski, S ; Goldsmith, P C ; Ellerby, H M ; Hayden, M R ; Bredesen, D E ; Ellerby, L M

Cell death and differentiation, 2004-04, Vol.11 (4), p.424-438 [Periódico revisado por pares]

England: Nature Publishing Group

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4
Parkin Facilitates the Elimination of Expanded Polyglutamine Proteins and Leads to Preservation of Proteasome Function
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Parkin Facilitates the Elimination of Expanded Polyglutamine Proteins and Leads to Preservation of Proteasome Function

Tsai, Yien Che ; Fishman, Paul S. ; Thakor, Nitish V. ; Oyler, George A.

The Journal of biological chemistry, 2003-06, Vol.278 (24), p.22044-22055 [Periódico revisado por pares]

United States: Elsevier Inc

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5
Study on TRPV1-mediated mechanism for the hypersecretion of mucus in respiratory inflammation
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Study on TRPV1-mediated mechanism for the hypersecretion of mucus in respiratory inflammation

Yang, J. ; Yu, H.M. ; Zhou, X.D. ; Kolosov, V.P. ; Perelman, J.M.

Molecular immunology, 2013-01, Vol.53 (1-2), p.161-171 [Periódico revisado por pares]

England: Elsevier Ltd

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6
The mitochondrial permeability transition in neurologic disease
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The mitochondrial permeability transition in neurologic disease

Norenberg, M.D. ; Rao, K.V. Rama

Neurochemistry international, 2007-06, Vol.50 (7), p.983-997 [Periódico revisado por pares]

England: Elsevier Ltd

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7
Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice
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Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice

Schilling, Gabriele ; Savonenko, Alena V. ; Klevytska, Alexandra ; Morton, Johanna L. ; Tucker, Stina M. ; Poirier, Michelle ; Gale, Alexa ; Chan, Ning ; Gonzales, Vicky ; Slunt, Hilda H. ; Coonfield, Michael L. ; Jenkins, Nancy A. ; Copeland, Neal G. ; Ross, Christopher A. ; Borchelt, David R.

Human molecular genetics, 2004-08, Vol.13 (15), p.1599-1610 [Periódico revisado por pares]

Oxford: Oxford University Press

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8
Cholesterol biosynthesis pathway is disturbed in YAC128 mice and is modulated by huntingtin mutation
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Cholesterol biosynthesis pathway is disturbed in YAC128 mice and is modulated by huntingtin mutation

Valenza, Marta ; Carroll, Jeffrey B. ; Leoni, Valerio ; Bertram, Lisa N. ; Björkhem, Ingeman ; Singaraja, Roshni R. ; Di Donato, Stefano ; Lutjohann, Dieter ; Hayden, Michael R. ; Cattaneo, Elena

Human molecular genetics, 2007-09, Vol.16 (18), p.2187-2198 [Periódico revisado por pares]

Oxford: Oxford University Press

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9
Striatal cells from mutant huntingtin knock-in mice are selectively vulnerable to mitochondrial complex II inhibitor-induced cell death through a non-apoptotic pathway
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Striatal cells from mutant huntingtin knock-in mice are selectively vulnerable to mitochondrial complex II inhibitor-induced cell death through a non-apoptotic pathway

Ruan, Qingmin ; Lesort, Mathieu ; MacDonald, Marcy E. ; Johnson, Gail V.W.

Human molecular genetics, 2004-04, Vol.13 (7), p.669-681 [Periódico revisado por pares]

Oxford: Oxford University Press

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10
Double-stranded RNA-dependent protein kinase, PKR, binds preferentially to Huntington's disease (HD) transcripts and is activated in HD tissue
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Double-stranded RNA-dependent protein kinase, PKR, binds preferentially to Huntington's disease (HD) transcripts and is activated in HD tissue

PEEL, Alyson L ; RAO, Ram V ; COTTRELL, Barbara A ; HAYDEN, Michael R ; ELLERBY, Lisa M ; BREDESEN, Dale E

Human molecular genetics, 2001-07, Vol.10 (15), p.1531-1538 [Periódico revisado por pares]

Oxford: Oxford University Press

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