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Induction of Allograft Tolerance by Monoclonal CD3 Antibodies: A Matter of Timing
Induction of Allograft Tolerance by Monoclonal CD3 Antibodies: A Matter of Timing
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Induction of Allograft Tolerance by Monoclonal CD3 Antibodies: A Matter of Timing

You, S. ; Zuber, J. ; Kuhn, C. ; Baas, M. ; Valette, F. ; Sauvaget, V. ; Sarnacki, S. ; Sawitzki, B. ; Bach, J.‐F. ; Volk, H.‐D. ; Chatenoud, L.

American journal of transplantation, 2012-11, Vol.12 (11), p.2909-2919 [Periódico revisado por pares]

Malden, USA: Blackwell Publishing Inc

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2
TOLERANCE TO ISLET AUTOANTIGENS IN TYPE 1 DIABETES
TOLERANCE TO ISLET AUTOANTIGENS IN TYPE 1 DIABETES
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TOLERANCE TO ISLET AUTOANTIGENS IN TYPE 1 DIABETES

Bach, Jean-François ; Chatenoud, Lucienne

Annual review of immunology, 2001-01, Vol.19 (1), p.131-161 [Periódico revisado por pares]

Palo Alto, CA 94303-0139: Annual Reviews

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3
99th Dahlem Conference on Infection, Inflammation and Chronic Inflammatory Disorders: Immune therapies of type 1 diabetes: new opportunities based on the hygiene hypothesis
99th Dahlem Conference on Infection, Inflammation and Chronic Inflammatory Disorders: Immune therapies of type 1 diabetes: new opportunities based on the hygiene hypothesis
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99th Dahlem Conference on Infection, Inflammation and Chronic Inflammatory Disorders: Immune therapies of type 1 diabetes: new opportunities based on the hygiene hypothesis

Chatenoud, L ; You, S ; Okada, H ; Kuhn, C ; Michaud, B ; Bach, J.-F

Clinical and experimental immunology, 2010-04, Vol.160 (1), p.106-112 [Periódico revisado por pares]

Oxford, UK: Oxford, UK : Blackwell Publishing Ltd

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4
CD3 antibody-induced dominant self tolerance in overtly diabetic NOD mice
CD3 antibody-induced dominant self tolerance in overtly diabetic NOD mice
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CD3 antibody-induced dominant self tolerance in overtly diabetic NOD mice

Chatenoud, L ; Primo, J ; Bach, JF

The Journal of immunology (1950), 1997-03, Vol.158 (6), p.2947-2954 [Periódico revisado por pares]

United States: Am Assoc Immnol

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5
Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia
Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia
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Notch3 mutations in CADASIL, a hereditary adult-onset condition causing stroke and dementia

Joutel, Anne ; Corpechot, Christophe ; Ducros, Anne ; Vahedi, Katayoun ; Chabriat, Hugues ; Mouton, Philippe ; Alamowitch, Sonia ; Domenga, Valérie ; Cécillion, Michaelle ; Maréchal, Emmanuelle ; Maciazek, Jacqueline ; Vayssière, Céline ; Cruaud, Corinne ; Cabanis, Emmanuel-Alain ; Ruchoux, Marie Madeleine ; Weissenbach, Jean ; Bach, Jean François ; Bousser, Marie Germaine ; Tournier-Lasserve, Elisabeth

Nature (London), 1996-10, Vol.383 (6602), p.707-710 [Periódico revisado por pares]

London: Nature Publishing

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6
TGF-β-dependent mechanisms mediate restoration of self-tolerance induced by antibodies to CD3 in overt autoimmune diabetes
TGF-β-dependent mechanisms mediate restoration of self-tolerance induced by antibodies to CD3 in overt autoimmune diabetes
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TGF-β-dependent mechanisms mediate restoration of self-tolerance induced by antibodies to CD3 in overt autoimmune diabetes

Chatenoud, Lucienne ; Belghith, Mériam ; Bluestone, Jeffrey A ; Barriot, Samia ; Mégret, Jérôme ; Bach, Jean-François

Nature medicine, 2003-09, Vol.9 (9), p.1202-1208 [Periódico revisado por pares]

United States

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7
Anti-CD3 Antibody Induces Long-Term Remission of Overt Autoimmunity in Nonobese Diabetic Mice
Anti-CD3 Antibody Induces Long-Term Remission of Overt Autoimmunity in Nonobese Diabetic Mice
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Anti-CD3 Antibody Induces Long-Term Remission of Overt Autoimmunity in Nonobese Diabetic Mice

Chatenoud, Lucienne ; Thervet, Eric ; Primo, Jacqueline ; Bach, Jean-Francois

Proceedings of the National Academy of Sciences - PNAS, 1994-01, Vol.91 (1), p.123-127 [Periódico revisado por pares]

Washington, DC: National Academy of Sciences of the United States of America

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8
Activation of natural killer T cells by α-galactosylceramide treatment prevents the onset and recurrence of autoimmune Type 1 diabetes
Activation of natural killer T cells by α-galactosylceramide treatment prevents the onset and recurrence of autoimmune Type 1 diabetes
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Activation of natural killer T cells by α-galactosylceramide treatment prevents the onset and recurrence of autoimmune Type 1 diabetes

Delovitch, Terry L ; Bach, Jean-François ; Sharif, Shayan ; Arreaza, Guillermo A ; Zucker, Peter ; Mi, Qing-Sheng ; Sondhi, Jitin ; Naidenko, Olga V ; Kronenberg, Mitchell ; Koezuka, Yasuhiko ; Gombert, Jean-Marc ; Leite-de-Moraes, Maria ; Gouarin, Christine ; Zhu, Ren ; Hameg, Agathe ; Nakayama, Toshinori ; Taniguchi, Masaru ; Lepault, Françoise ; Lehuen, Agnès ; Herbelin, André

Nature Medicine, 2001-09, Vol.7 (9), p.1057-1062 [Periódico revisado por pares]

United States: Nature Publishing Group

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9
Naturally occurring autoantibodies interfere with β-amyloid metabolism and improve cognition in a transgenic mouse model of Alzheimer's disease 24 h after single treatment
Naturally occurring autoantibodies interfere with β-amyloid metabolism and improve cognition in a transgenic mouse model of Alzheimer's disease 24 h after single treatment
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Naturally occurring autoantibodies interfere with β-amyloid metabolism and improve cognition in a transgenic mouse model of Alzheimer's disease 24 h after single treatment

Mengel, D ; Röskam, S ; Neff, F ; Balakrishnan, K ; Deuster, O ; Gold, M ; Oertel, W H ; Bacher, M ; Bach, J P ; Dodel, R

Translational psychiatry, 2013-03, Vol.3 (3), p.e236-e236 [Periódico revisado por pares]

United States: Nature Publishing Group

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10
Brassica juncea 3-hydroxy-3-methylglutaryl (HMG)-CoA synthase 1: expression and characterization of recombinant wild-type and mutant enzymes
Brassica juncea 3-hydroxy-3-methylglutaryl (HMG)-CoA synthase 1: expression and characterization of recombinant wild-type and mutant enzymes
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Brassica juncea 3-hydroxy-3-methylglutaryl (HMG)-CoA synthase 1: expression and characterization of recombinant wild-type and mutant enzymes

Nagegowda, Dinesh A ; Bach, Thomas J ; Chye, Mee-Len

Biochemical journal, 2004-11, Vol.383 (Pt. 3), p.517-527 [Periódico revisado por pares]

England: Portland Press Ltd

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