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Refinado por: Base de dados/Biblioteca: ScienceDirect® remover Base de dados/Biblioteca: Wiley-Blackwell Full Collection 2013 remover
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1
HypERlnc attenuates angiotensin II‐induced cardiomyocyte hypertrophy via promoting SIRT1 SUMOylation‐mediated activation of PGC‐1α/PPARα pathway in AC16 cells
HypERlnc attenuates angiotensin II‐induced cardiomyocyte hypertrophy via promoting SIRT1 SUMOylation‐mediated activation of PGC‐1α/PPARα pathway in AC16 cells
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HypERlnc attenuates angiotensin II‐induced cardiomyocyte hypertrophy via promoting SIRT1 SUMOylation‐mediated activation of PGC‐1α/PPARα pathway in AC16 cells

Yue, Lijuan ; Sheng, Siqi ; Yuan, Meng ; Lu, Jing ; Li, Tianyu ; Shi, Yuanqi ; Dong, Zengxiang

Cell biology international, 2023-06, Vol.47 (6), p.1068-1080 [Periódico revisado por pares]

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2
Dynamics of metabolism and regulation of epigenetics during cardiomyocytes maturation
Dynamics of metabolism and regulation of epigenetics during cardiomyocytes maturation
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Dynamics of metabolism and regulation of epigenetics during cardiomyocytes maturation

Hayat, Rabia

Cell biology international, 2023-01, Vol.47 (1), p.30-40 [Periódico revisado por pares]

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3
Knockdown circ_0040414 inhibits inflammation, apoptosis and promotes the proliferation of cardiomyocytes via miR‐186‐5p/PTEN/AKT axis in chronic heart failure
Knockdown circ_0040414 inhibits inflammation, apoptosis and promotes the proliferation of cardiomyocytes via miR‐186‐5p/PTEN/AKT axis in chronic heart failure
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Knockdown circ_0040414 inhibits inflammation, apoptosis and promotes the proliferation of cardiomyocytes via miR‐186‐5p/PTEN/AKT axis in chronic heart failure

Feng, Yanling ; Yan, Biao ; Cheng, Hongji ; Wu, Jinlei ; Chen, Qinxiu ; Duan, Yuexing ; Zhang, Peng ; Zheng, Dong ; Lin, Guixiong ; Zhuo, Yufeng

Cell biology international, 2021-11, Vol.45 (11), p.2304-2315 [Periódico revisado por pares]

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4
Soluble factors released by dedifferentiated fat cells reduce the functional activity of iPS cell‐derived cardiomyocytes
Soluble factors released by dedifferentiated fat cells reduce the functional activity of iPS cell‐derived cardiomyocytes
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Soluble factors released by dedifferentiated fat cells reduce the functional activity of iPS cell‐derived cardiomyocytes

Watanabe, Hirofumi ; Kanemaru, Kazunori ; Hagikura, Kazuhiro ; Matsumoto, Taro ; Ayusawa, Mamoru ; Morioka, Ichiro

Cell biology international, 2021-02, Vol.45 (2), p.295-304 [Periódico revisado por pares]

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5
LncRNA SNHG1 exerts a protective role in cardiomyocytes hypertrophy via targeting miR‐15a‐5p/HMGA1 axis
LncRNA SNHG1 exerts a protective role in cardiomyocytes hypertrophy via targeting miR‐15a‐5p/HMGA1 axis
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LncRNA SNHG1 exerts a protective role in cardiomyocytes hypertrophy via targeting miR‐15a‐5p/HMGA1 axis

Yan, Si‐Min ; Li, Hu ; Shu, Qing ; Wu, Wei‐Jun ; Luo, Xue‐Mei ; Lu, Lei

Cell biology international, 2020-04, Vol.44 (4), p.1009-1019 [Periódico revisado por pares]

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6
Knockdown of HIF1A‐AS2 suppresses TRIM44 to protect cardiomyocytes against hypoxia‐induced injury
Knockdown of HIF1A‐AS2 suppresses TRIM44 to protect cardiomyocytes against hypoxia‐induced injury
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Knockdown of HIF1A‐AS2 suppresses TRIM44 to protect cardiomyocytes against hypoxia‐induced injury

Luo, Fang ; Wu, Yitian ; Zhu, Liying ; Zhang, Jun ; Liu, Yixin ; Jia, Weiguo

Cell biology international, 2020-07, Vol.44 (7), p.1523-1534 [Periódico revisado por pares]

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7
Inhibition of intermittent calcium‐activated potassium channel (SK4) attenuates Ang II‐induced hypertrophy of human‐induced stem cell‐derived cardiomyocytes via targeting Ras–Raf–MEK1/2–ERK1/2 and CN–NFAT signaling pathways
Inhibition of intermittent calcium‐activated potassium channel (SK4) attenuates Ang II‐induced hypertrophy of human‐induced stem cell‐derived cardiomyocytes via targeting Ras–Raf–MEK1/2–ERK1/2 and CN–NFAT signaling pathways
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Inhibition of intermittent calcium‐activated potassium channel (SK4) attenuates Ang II‐induced hypertrophy of human‐induced stem cell‐derived cardiomyocytes via targeting Ras–Raf–MEK1/2–ERK1/2 and CN–NFAT signaling pathways

Zhao, Hongyi ; Wang, Xi ; Tang, Yanhong ; Zhao, Qingyan ; Huang, Congxin

Cell biology international, 2023-02, Vol.47 (2), p.480-491 [Periódico revisado por pares]

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8
FNDC5/irisin reduces ferroptosis and improves mitochondrial dysfunction in hypoxic cardiomyocytes by Nrf2/HO‐1 axis
FNDC5/irisin reduces ferroptosis and improves mitochondrial dysfunction in hypoxic cardiomyocytes by Nrf2/HO‐1 axis
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FNDC5/irisin reduces ferroptosis and improves mitochondrial dysfunction in hypoxic cardiomyocytes by Nrf2/HO‐1 axis

Cao, Guangying ; Yang, Chao ; Jin, Zhitao ; Wei, Hanwen ; Xin, Chao ; Zheng, Chengrong ; Xu, Jibing ; Huang, Qing ; Zhang, Zheng ; Hu, Taohong

Cell biology international, 2022-05, Vol.46 (5), p.723-736 [Periódico revisado por pares]

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9
miR‐16 exhibits protective function in LPS‐treated cardiomyocytes by targeting DOCK2 to repress cell apoptosis and exert anti‐inflammatory effect
miR‐16 exhibits protective function in LPS‐treated cardiomyocytes by targeting DOCK2 to repress cell apoptosis and exert anti‐inflammatory effect
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miR‐16 exhibits protective function in LPS‐treated cardiomyocytes by targeting DOCK2 to repress cell apoptosis and exert anti‐inflammatory effect

Wang, Lei ; Zhang, Yangyang ; Zhu, Guangfu ; Ma, Yuncong ; Zuo, Huan ; Tian, Xia

Cell biology international, 2020-08, Vol.44 (8), p.1760-1768 [Periódico revisado por pares]

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10
Penehyclidine hydrochloride protects against anoxia/reoxygenation injury in cardiomyocytes through ATP‐sensitive potassium channels, and the Akt/GSK‐3β and Akt/mTOR signaling pathways
Penehyclidine hydrochloride protects against anoxia/reoxygenation injury in cardiomyocytes through ATP‐sensitive potassium channels, and the Akt/GSK‐3β and Akt/mTOR signaling pathways
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Penehyclidine hydrochloride protects against anoxia/reoxygenation injury in cardiomyocytes through ATP‐sensitive potassium channels, and the Akt/GSK‐3β and Akt/mTOR signaling pathways

Zi, Congna ; Zhang, Chunlei ; Yang, Yanli ; Ma, Jun

Cell biology international, 2020-06, Vol.44 (6), p.1353-1362 [Periódico revisado por pares]

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