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Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF'alfa' in the cutaneous microvasculature
Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF'alfa' in the cutaneous microvasculature
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Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF'alfa' in the cutaneous microvasculature

Soraia Katia Pereira Costa Lidia M Yshii; Robin N Poston; Marcelo Nicolás Muscará; Susan D Brain

Journal of Neuroimmunology Amsterdam v. 171, n. 1/2, p. 99-109, 2006

Amsterdam 2006

Item não circula. Consulte sua biblioteca.(Acessar)

2
Pivotal role of endogenous tachykinins and the NK sub(1) receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF alpha in the cutaneous microvasculature
Pivotal role of endogenous tachykinins and the NK sub(1) receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF alpha in the cutaneous microvasculature
Material Type:
Artigo 		Adicionar ao Meu Espaço

Pivotal role of endogenous tachykinins and the NK sub(1) receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNF alpha in the cutaneous microvasculature

Costa, SKP ; Yshii, L M ; Poston, R N ; Muscara, M N ; Brain, S D

Journal of neuroimmunology, 2006-02, Vol.171 (1-2), p.99-109 [Periódico revisado por pares]

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3
Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature
Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature
Material Type:
Artigo 		Adicionar ao Meu Espaço

Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature

Costa, Soraia K.P. ; Yshii, Lidia M. ; Poston, Robin N. ; Muscará, Marcelo N. ; Brain, Susan D.

Journal of neuroimmunology, 2006-02, Vol.171 (1-2), p.99-109 [Periódico revisado por pares]

Texto completo disponível

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4
Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFalpha in the cutaneous microvasculature
Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFalpha in the cutaneous microvasculature
Material Type:
Artigo 		Adicionar ao Meu Espaço

Pivotal role of endogenous tachykinins and the NK1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFalpha in the cutaneous microvasculature

Costa, Soraia K P ; Yshii, Lidia M ; Poston, Robin N ; Muscará, Marcelo N ; Brain, Susan D

Journal of neuroimmunology, 2006-02, Vol.171 (1-2), p.99-109 [Periódico revisado por pares]

Netherlands

Texto completo disponível

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5
Pivotal role of endogenous tachykinins and the NK 1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature
Pivotal role of endogenous tachykinins and the NK 1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature
Material Type:
Artigo 		Adicionar ao Meu Espaço

Pivotal role of endogenous tachykinins and the NK 1 receptor in mediating leukocyte accumulation, in the absence of oedema formation, in response to TNFα in the cutaneous microvasculature

Costa, Soraia K.P. ; Yshii, Lidia M. ; Poston, Robin N. ; Muscará, Marcelo N. ; Brain, Susan D.

Journal of neuroimmunology, 2006, Vol.171 (1), p.99-109 [Periódico revisado por pares]

Elsevier B.V

Texto completo disponível

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