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1
Mice lacking sister chromatid cohesion protein PDS5B exhibit developmental abnormalities reminiscent of Cornelia de Lange syndrome
Mice lacking sister chromatid cohesion protein PDS5B exhibit developmental abnormalities reminiscent of Cornelia de Lange syndrome
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Mice lacking sister chromatid cohesion protein PDS5B exhibit developmental abnormalities reminiscent of Cornelia de Lange syndrome

Zhang, Bin ; Jain, Sanjay ; Song, Haengseok ; Fu, Ming ; Heuckeroth, Robert O ; Erlich, Jonathan M ; Jay, Patrick Y ; Milbrandt, Jeffrey

Development (Cambridge), 2007-09, Vol.134 (17), p.3191-3201 [Periódico revisado por pares]

England: The Company of Biologists Limited

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2
RET signaling is essential for migration, axonal growth and axon guidance of developing sympathetic neurons
RET signaling is essential for migration, axonal growth and axon guidance of developing sympathetic neurons
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RET signaling is essential for migration, axonal growth and axon guidance of developing sympathetic neurons

Enomoto, H ; Crawford, P A ; Gorodinsky, A ; Heuckeroth, R O ; Johnson, Jr, E M ; Milbrandt, J

Development (Cambridge), 2001-10, Vol.128 (20), p.3963-3974 [Periódico revisado por pares]

England: The Company of Biologists Limited

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3
Mice expressing a dominant-negative Ret mutation phenocopy human Hirschsprung disease and delineate a direct role of Ret in spermatogenesis
Mice expressing a dominant-negative Ret mutation phenocopy human Hirschsprung disease and delineate a direct role of Ret in spermatogenesis
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Mice expressing a dominant-negative Ret mutation phenocopy human Hirschsprung disease and delineate a direct role of Ret in spermatogenesis

Jain, Sanjay ; Naughton, Cathy K ; Yang, Mao ; Strickland, Amy ; Vij, Kiran ; Encinas, Mario ; Golden, Judy ; Gupta, Akshay ; Heuckeroth, Robert ; Johnson, Jr, Eugene M ; Milbrandt, Jeffrey

Development (Cambridge), 2004-11, Vol.131 (21), p.5503-5513 [Periódico revisado por pares]

England: The Company of Biologists Limited

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4
Development of cranial parasympathetic ganglia requires sequential actions of GDNF and neurturin
Development of cranial parasympathetic ganglia requires sequential actions of GDNF and neurturin
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Development of cranial parasympathetic ganglia requires sequential actions of GDNF and neurturin

Enomoto, H ; Heuckeroth, R O ; Golden, J P ; Johnson, E M ; Milbrandt, J

Development (Cambridge), 2000-11, Vol.127 (22), p.4877-4889 [Periódico revisado por pares]

England: The Company of Biologists Limited

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5
Conditional ablation of GFR alpha 1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
Conditional ablation of GFR alpha 1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
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Conditional ablation of GFR alpha 1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype

Uesaka, Toshihiro ; Jain, Sanjay ; Yonemura, Shigenobu ; Uchiyama, Yasuo ; Milbrandt, Jeffrey ; Enomoto, Hideki

Development (Cambridge), 2007-06, Vol.134 (11), p.2171-2181 [Periódico revisado por pares]

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6
GDNF and neurturin are target-derived factors essential for cranial parasympathetic neuron development
GDNF and neurturin are target-derived factors essential for cranial parasympathetic neuron development
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GDNF and neurturin are target-derived factors essential for cranial parasympathetic neuron development

Hashino, E ; Shero, M ; Junghans, D ; Rohrer, H ; Milbrandt, J ; Johnson, Jr, E M

Development (Cambridge), 2001-10, Vol.128 (19), p.3773-3782 [Periódico revisado por pares]

England: The Company of Biologists Limited

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7
Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
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Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype

Uesaka, Toshihiro ; Jain, Sanjay ; Yonemura, Shigenobu ; Uchiyama, Yasuo ; Milbrandt, Jeffrey ; Enomoto, Hideki

Development (Cambridge), 2007-06, Vol.134 (11), p.2171-2181 [Periódico revisado por pares]

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8
Expression of FGF receptor gene in rat development
Expression of FGF receptor gene in rat development
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Expression of FGF receptor gene in rat development

WANAKA, A ; MILBRANDT, J ; JOHNSON, E. M

Development (Cambridge), 1991-02, Vol.111 (2), p.455-468 [Periódico revisado por pares]

Cambridge: The Company of Biologists Limited

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9
Expression of the nerve growth factor-regulated NGFI-A and NGFI-B genes in the developing rat
Expression of the nerve growth factor-regulated NGFI-A and NGFI-B genes in the developing rat
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Expression of the nerve growth factor-regulated NGFI-A and NGFI-B genes in the developing rat

Watson, M A ; Milbrandt, J

Development (Cambridge), 1990-09, Vol.110 (1), p.173-183 [Periódico revisado por pares]

England: The Company of Biologists Limited

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10
Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype
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Conditional ablation of GFRα1 in postmigratory enteric neurons triggers unconventional neuronal death in the colon and causes a Hirschsprung's disease phenotype

Toshihiro Uesaka ; Sanjay Jain ; Shigenobu Yonemura ; Yasuo Uchiyama ; Jeffrey Milbrandt ; Hideki Enomoto

Development (Cambridge), 2007-06, Vol.134 (11), p.2171 [Periódico revisado por pares]

The Company of Biologists Limited

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