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Refinado por: Nome da Publicação: Oncogene remover assunto: Oncology remover
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1
SPOP regulates prostate epithelial cell proliferation and promotes ubiquitination and turnover of c-MYC oncoprotein
SPOP regulates prostate epithelial cell proliferation and promotes ubiquitination and turnover of c-MYC oncoprotein
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SPOP regulates prostate epithelial cell proliferation and promotes ubiquitination and turnover of c-MYC oncoprotein

Geng, C ; Kaochar, S ; Li, M ; Rajapakshe, K ; Fiskus, W ; Dong, J ; Foley, C ; Dong, B ; Zhang, L ; Kwon, O-J ; Shah, S S ; Bolaki, M ; Xin, L ; Ittmann, M ; O'Malley, B W ; Coarfa, C ; Mitsiades, N

Oncogene, 2017-08, Vol.36 (33), p.4767-4777 [Periódico revisado por pares]

England: Nature Publishing Group

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2
Loss of ATF3 promotes hormone-induced prostate carcinogenesis and the emergence of CK5(+)CK8(+) epithelial cells
Loss of ATF3 promotes hormone-induced prostate carcinogenesis and the emergence of CK5(+)CK8(+) epithelial cells
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Loss of ATF3 promotes hormone-induced prostate carcinogenesis and the emergence of CK5(+)CK8(+) epithelial cells

Wang, Z ; Kim, J ; Teng, Y ; Ding, H-F ; Zhang, J ; Hai, T ; Cowell, J K ; Yan, C

Oncogene, 2016-07, Vol.35 (27), p.3555-3564 [Periódico revisado por pares]

England: Nature Publishing Group

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3
Evaluation of potential mechanisms underlying genotype-phenotype correlations in multiple endocrine neoplasia type 2
Evaluation of potential mechanisms underlying genotype-phenotype correlations in multiple endocrine neoplasia type 2
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Evaluation of potential mechanisms underlying genotype-phenotype correlations in multiple endocrine neoplasia type 2

MISE, N ; DROSTEN, M ; RACEK, T ; TANNAPFEL, A ; PÜTZER, B. M

Oncogene, 2006-10, Vol.25 (50), p.6637-6647 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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4
Sin3a acts through a multi-gene module to regulate invasion in Drosophila and human tumors
Sin3a acts through a multi-gene module to regulate invasion in Drosophila and human tumors
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Sin3a acts through a multi-gene module to regulate invasion in Drosophila and human tumors

Das, T K ; Sangodkar, J ; Negre, N ; Narla, G ; Cagan, R L

Oncogene, 2013-06, Vol.32 (26), p.3184-3197 [Periódico revisado por pares]

England: Nature Publishing Group

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5
The DACH1 gene is frequently deleted in prostate cancer, restrains prostatic intraepithelial neoplasia, decreases DNA damage repair, and predicts therapy responses
The DACH1 gene is frequently deleted in prostate cancer, restrains prostatic intraepithelial neoplasia, decreases DNA damage repair, and predicts therapy responses
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The DACH1 gene is frequently deleted in prostate cancer, restrains prostatic intraepithelial neoplasia, decreases DNA damage repair, and predicts therapy responses

Li, Zhiping ; Jiao, Xuanmao ; Robertson, A Gordon ; Di Sante, Gabriele ; Ashton, Anthony W ; DiRocco, Agnese ; Wang, Min ; Zhao, Jun ; Addya, Sankar ; Wang, Chenguang ; McCue, Peter A ; South, Andrew P ; Cordon-Cardo, Carlos ; Liu, Runzhi ; Patel, Kishan ; Hamid, Rasha ; Parmar, Jorim ; DuHadaway, James B ; Jones, Steven J M ; Casimiro, Mathew C ; Schultz, Nikolaus ; Kossenkov, Andrew ; Phoon, Lai Yee ; Chen, Hao ; Lan, Li ; Sun, Yunguang ; Iczkowski, Kenneth A ; Rui, Hallgeir ; Pestell, Richard G

Oncogene, 2023-06, Vol.42 (22), p.1857-1873 [Periódico revisado por pares]

England: Nature Publishing Group

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6
Androgen signaling is a confounding factor for β-catenin-mediated prostate tumorigenesis
Androgen signaling is a confounding factor for β-catenin-mediated prostate tumorigenesis
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Androgen signaling is a confounding factor for β-catenin-mediated prostate tumorigenesis

Lee, S H ; Luong, R ; Johnson, D T ; Cunha, G R ; Rivina, L ; Gonzalgo, M L ; Sun, Z

Oncogene, 2016-02, Vol.35 (6), p.702-714 [Periódico revisado por pares]

England: Nature Publishing Group

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7
Complex patterns of ETS gene alteration arise during cancer development in the human prostate
Complex patterns of ETS gene alteration arise during cancer development in the human prostate
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Complex patterns of ETS gene alteration arise during cancer development in the human prostate

CLARK, J ; ATTARD, G ; PARKER, M. D ; FOSTER, C. S ; BERNEY, D ; KOVACS, G ; COOPER, C. S ; JHAVAR, S ; FLOHR, P ; REID, A ; DE-BONO, J ; EELES, R ; SCARDINO, P ; CUZICK, J ; FISHER, G

Oncogene, 2008-03, Vol.27 (14), p.1993-2003 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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8
A mouse model of heterogeneous, c-MYC-initiated prostate cancer with loss of Pten and p53
A mouse model of heterogeneous, c-MYC-initiated prostate cancer with loss of Pten and p53
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A mouse model of heterogeneous, c-MYC-initiated prostate cancer with loss of Pten and p53

Kim, J ; Roh, M ; Doubinskaia, I ; Algarroba, G N ; Eltoum, I-E A ; Abdulkadir, S A

Oncogene, 2012-01, Vol.31 (3), p.322-332 [Periódico revisado por pares]

England: Nature Publishing Group

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9
Dedifferentiation into blastomere-like cancer stem cells via formation of polyploid giant cancer cells
Dedifferentiation into blastomere-like cancer stem cells via formation of polyploid giant cancer cells
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Dedifferentiation into blastomere-like cancer stem cells via formation of polyploid giant cancer cells

Niu, N ; Mercado-Uribe, I ; Liu, J

Oncogene, 2017-08, Vol.36 (34), p.4887-4900 [Periódico revisado por pares]

England: Nature Publishing Group

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10
Integration of the HPV16 genome does not invariably result in high levels of viral oncogene transcripts
Integration of the HPV16 genome does not invariably result in high levels of viral oncogene transcripts
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Integration of the HPV16 genome does not invariably result in high levels of viral oncogene transcripts

HÄFNER, N ; DRIESCH, C ; GAJDA, M ; JANSEN, L ; KIRCHMAYR, R ; RUNNEBAUM, I. B ; DÜRST, M

Oncogene, 2008-03, Vol.27 (11), p.1610-1617 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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