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Refinado por: Base de dados/Biblioteca: ClinicalKey remover Nome da Publicação: Neurobiology Of Disease remover
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11
Mic60/mitofilin overexpression alters mitochondrial dynamics and attenuates vulnerability of dopaminergic cells to dopamine and rotenone
Mic60/mitofilin overexpression alters mitochondrial dynamics and attenuates vulnerability of dopaminergic cells to dopamine and rotenone
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Mic60/mitofilin overexpression alters mitochondrial dynamics and attenuates vulnerability of dopaminergic cells to dopamine and rotenone

Van Laar, Victor S ; Berman, Sarah B ; Hastings, Teresa G

Neurobiology of disease, 2016-07, Vol.91, p.247-261 [Periódico revisado por pares]

United States: Elsevier Inc

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12
Alteration of autophagosomal proteins (LC3, GABARAP and GATE-16) in Lewy body disease
Alteration of autophagosomal proteins (LC3, GABARAP and GATE-16) in Lewy body disease
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Alteration of autophagosomal proteins (LC3, GABARAP and GATE-16) in Lewy body disease

Tanji, Kunikazu ; Mori, Fumiaki ; Kakita, Akiyoshi ; Takahashi, Hitoshi ; Wakabayashi, Koichi

Neurobiology of disease, 2011-09, Vol.43 (3), p.690-697 [Periódico revisado por pares]

United States: Elsevier Inc

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13
A cell-penetrating ester of the neural metabolite lanthionine ketimine stimulates autophagy through the mTORC1 pathway: Evidence for a mechanism of action with pharmacological implications for neurodegenerative pathologies
A cell-penetrating ester of the neural metabolite lanthionine ketimine stimulates autophagy through the mTORC1 pathway: Evidence for a mechanism of action with pharmacological implications for neurodegenerative pathologies
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A cell-penetrating ester of the neural metabolite lanthionine ketimine stimulates autophagy through the mTORC1 pathway: Evidence for a mechanism of action with pharmacological implications for neurodegenerative pathologies

Harris-White, Marni E ; Ferbas, Kathie G ; Johnson, Ming F ; Eslami, Pirooz ; Poteshkina, Aleksandra ; Venkova, Kalina ; Christov, Alexandar ; Hensley, Kenneth

Neurobiology of disease, 2015-12, Vol.84, p.60-68 [Periódico revisado por pares]

United States: Elsevier Inc

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14
LDB3 splicing abnormalities are specific to skeletal muscles of patients with myotonic dystrophy type 1 and alter its PKC binding affinity
LDB3 splicing abnormalities are specific to skeletal muscles of patients with myotonic dystrophy type 1 and alter its PKC binding affinity
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LDB3 splicing abnormalities are specific to skeletal muscles of patients with myotonic dystrophy type 1 and alter its PKC binding affinity

Yamashita, Yoshihiro ; Matsuura, Tohru ; Kurosaki, Tatsuaki ; Amakusa, Yoshinobu ; Kinoshita, Masanobu ; Ibi, Tohru ; Sahashi, Ko ; Ohno, Kinji

Neurobiology of disease, 2014-09, Vol.69, p.200-205 [Periódico revisado por pares]

United States: Elsevier Inc

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