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Bruton tyrosine kinase represents a promising therapeutic target for treatment of chronic lymphocytic leukemia and is effectively targeted by PCI-32765
Bruton tyrosine kinase represents a promising therapeutic target for treatment of chronic lymphocytic leukemia and is effectively targeted by PCI-32765
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Bruton tyrosine kinase represents a promising therapeutic target for treatment of chronic lymphocytic leukemia and is effectively targeted by PCI-32765

Herman, Sarah E.M. ; Gordon, Amber L. ; Hertlein, Erin ; Ramanunni, Asha ; Zhang, Xiaoli ; Jaglowski, Samantha ; Flynn, Joseph ; Jones, Jeffrey ; Blum, Kristie A. ; Buggy, Joseph J. ; Hamdy, Ahmed ; Johnson, Amy J. ; Byrd, John C.

Blood, 2011-06, Vol.117 (23), p.6287-6296 [Periódico revisado por pares]

Washington, DC: Elsevier Inc

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2
Ascorbic acid-induced TET activation mitigates adverse hydroxymethylcytosine loss in renal cell carcinoma
Ascorbic acid-induced TET activation mitigates adverse hydroxymethylcytosine loss in renal cell carcinoma
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Ascorbic acid-induced TET activation mitigates adverse hydroxymethylcytosine loss in renal cell carcinoma

Shenoy, Niraj ; Bhagat, Tushar D ; Cheville, John ; Lohse, Christine ; Bhattacharyya, Sanchari ; Tischer, Alexander ; Machha, Venkata ; Gordon-Mitchell, Shanisha ; Choudhary, Gaurav ; Wong, Li-Fan ; Gross, LouAnn ; Ressigue, Emily ; Leibovich, Bradley ; Boorjian, Stephen A ; Steidl, Ulrich ; Wu, Xiaosheng ; Pradhan, Kith ; Gartrell, Benjamin ; Agarwal, Beamon ; Pagliaro, Lance ; Suzuki, Masako ; Greally, John M ; Rakheja, Dinesh ; Thompson, R Houston ; Susztak, Katalin ; Witzig, Thomas ; Zou, Yiyu ; Verma, Amit

The Journal of clinical investigation, 2019-04, Vol.129 (4), p.1612-1625 [Periódico revisado por pares]

United States: American Society for Clinical Investigation

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3
Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities
Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities
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Patterns of transcription factor programs and immune pathway activation define four major subtypes of SCLC with distinct therapeutic vulnerabilities

Gay, Carl M. ; Stewart, C. Allison ; Park, Elizabeth M. ; Diao, Lixia ; Groves, Sarah M. ; Heeke, Simon ; Nabet, Barzin Y. ; Fujimoto, Junya ; Solis, Luisa M. ; Lu, Wei ; Xi, Yuanxin ; Cardnell, Robert J. ; Wang, Qi ; Fabbri, Giulia ; Cargill, Kasey R. ; Vokes, Natalie I. ; Ramkumar, Kavya ; Zhang, Bingnan ; Della Corte, Carminia M. ; Robson, Paul ; Swisher, Stephen G. ; Roth, Jack A. ; Glisson, Bonnie S. ; Shames, David S. ; Wistuba, Ignacio I. ; Wang, Jing ; Quaranta, Vito ; Minna, John ; Heymach, John V. ; Byers, Lauren Averett

Cancer cell, 2021-03, Vol.39 (3), p.346-360.e7 [Periódico revisado por pares]

United States: Elsevier Inc

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4
DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity
DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity
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DNA exonuclease Trex1 regulates radiotherapy-induced tumour immunogenicity

Vanpouille-Box, Claire ; Alard, Amandine ; Aryankalayil, Molykutty J ; Sarfraz, Yasmeen ; Diamond, Julie M ; Schneider, Robert J ; Inghirami, Giorgio ; Coleman, C Norman ; Formenti, Silvia C ; Demaria, Sandra

Nature communications, 2017-06, Vol.8 (1), p.15618-15618, Article 15618 [Periódico revisado por pares]

England: Nature Publishing Group

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5
An Evolutionarily Conserved Function of Polycomb Silences the MHC Class I Antigen Presentation Pathway and Enables Immune Evasion in Cancer
An Evolutionarily Conserved Function of Polycomb Silences the MHC Class I Antigen Presentation Pathway and Enables Immune Evasion in Cancer
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An Evolutionarily Conserved Function of Polycomb Silences the MHC Class I Antigen Presentation Pathway and Enables Immune Evasion in Cancer

Burr, Marian L. ; Sparbier, Christina E. ; Chan, Kah Lok ; Chan, Yih-Chih ; Kersbergen, Ariena ; Lam, Enid Y.N. ; Azidis-Yates, Elizabeth ; Vassiliadis, Dane ; Bell, Charles C. ; Gilan, Omer ; Jackson, Susan ; Tan, Lavinia ; Wong, Stephen Q. ; Hollizeck, Sebastian ; Michalak, Ewa M. ; Siddle, Hannah V. ; McCabe, Michael T. ; Prinjha, Rab K. ; Guerra, Glen R. ; Solomon, Benjamin J. ; Sandhu, Shahneen ; Dawson, Sarah-Jane ; Beavis, Paul A. ; Tothill, Richard W. ; Cullinane, Carleen ; Lehner, Paul J. ; Sutherland, Kate D. ; Dawson, Mark A.

Cancer cell, 2019-10, Vol.36 (4), p.385-401.e8 [Periódico revisado por pares]

United States: Elsevier Inc

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6
Exosomal miR-221/222 enhances tamoxifen resistance in recipient ER-positive breast cancer cells
Exosomal miR-221/222 enhances tamoxifen resistance in recipient ER-positive breast cancer cells
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Exosomal miR-221/222 enhances tamoxifen resistance in recipient ER-positive breast cancer cells

Wei, Yifang ; Lai, Xiaofeng ; Yu, Shentong ; Chen, Suning ; Ma, Yongzheng ; Zhang, Yuan ; Li, Huichen ; Zhu, Xingmei ; Yao, Libo ; Zhang, Jian

Breast cancer research and treatment, 2014-09, Vol.147 (2), p.423-431 [Periódico revisado por pares]

Boston: Springer US

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7
HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells
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HIF-1 regulates CD47 expression in breast cancer cells to promote evasion of phagocytosis and maintenance of cancer stem cells

Zhang, Huimin ; Lu, Haiquan ; Xiang, Lisha ; Bullen, John W. ; Zhang, Chuanzhao ; Samanta, Debangshu ; Gilkes, Daniele M. ; He, Jianjun ; Semenza, Gregg L.

Proceedings of the National Academy of Sciences - PNAS, 2015-11, Vol.112 (45), p.E6215-E6223 [Periódico revisado por pares]

United States: National Academy of Sciences

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8
DNA methyltransferase inhibition upregulates MHC-I to potentiate cytotoxic T lymphocyte responses in breast cancer
DNA methyltransferase inhibition upregulates MHC-I to potentiate cytotoxic T lymphocyte responses in breast cancer
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DNA methyltransferase inhibition upregulates MHC-I to potentiate cytotoxic T lymphocyte responses in breast cancer

Luo, Na ; Nixon, Mellissa J ; Gonzalez-Ericsson, Paula I ; Sanchez, Violeta ; Opalenik, Susan R ; Li, Huili ; Zahnow, Cynthia A ; Nickels, Michael L ; Liu, Fei ; Tantawy, Mohammed N ; Sanders, Melinda E ; Manning, H Charles ; Balko, Justin M

Nature communications, 2018-01, Vol.9 (1), p.248-11, Article 248 [Periódico revisado por pares]

England: Nature Publishing Group

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9
Low-dose decitabine priming endows CAR T cells with enhanced and persistent antitumour potential via epigenetic reprogramming
Low-dose decitabine priming endows CAR T cells with enhanced and persistent antitumour potential via epigenetic reprogramming
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Low-dose decitabine priming endows CAR T cells with enhanced and persistent antitumour potential via epigenetic reprogramming

Wang, Yao ; Tong, Chuan ; Dai, Hanren ; Wu, Zhiqiang ; Han, Xiao ; Guo, Yelei ; Chen, Deyun ; Wei, Jianshu ; Ti, Dongdong ; Liu, Zongzhi ; Mei, Qian ; Li, Xiang ; Dong, Liang ; Nie, Jing ; Zhang, Yajing ; Han, Weidong

Nature communications, 2021-01, Vol.12 (1), p.409-18, Article 409 [Periódico revisado por pares]

England: Nature Publishing Group

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10
PKM2 Phosphorylates Histone H3 and Promotes Gene Transcription and Tumorigenesis
PKM2 Phosphorylates Histone H3 and Promotes Gene Transcription and Tumorigenesis
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PKM2 Phosphorylates Histone H3 and Promotes Gene Transcription and Tumorigenesis

Yang, Weiwei ; Xia, Yan ; Hawke, David ; Li, Xinjian ; Liang, Ji ; Xing, Dongming ; Aldape, Kenneth ; Hunter, Tony ; Alfred Yung, W.K. ; Lu, Zhimin

Cell, 2012-08, Vol.150 (4), p.685-696 [Periódico revisado por pares]

United States: Elsevier Inc

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