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1
1-Methyl-D-tryptophan potentiates TGF-β-induced epithelial-mesenchymal transition in T24 human bladder cancer cells
1-Methyl-D-tryptophan potentiates TGF-β-induced epithelial-mesenchymal transition in T24 human bladder cancer cells
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1-Methyl-D-tryptophan potentiates TGF-β-induced epithelial-mesenchymal transition in T24 human bladder cancer cells

Brito, Rodrigo Barbosa Oliveira ; Malta, Camila Soares ; Souza, Diego Mota ; Matheus, Luiz Henrique Gomes ; Matos, Yves Silva Teles ; Silva, Chrisna Souza ; Ferreira, Janaína Mendes ; Nunes, Valeria Sutti ; França, Cristiane Miranda ; Dellê, Humberto Platten, Michael

PloS one, 2015-08, Vol.10 (8), p.e0134858-e0134858 [Periódico revisado por pares]

United States: Public Library of Science

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2
1-Methylnicotinamide and nicotinamide: two related anti-inflammatory agents that differentially affect the functions of activated macrophages
1-Methylnicotinamide and nicotinamide: two related anti-inflammatory agents that differentially affect the functions of activated macrophages
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1-Methylnicotinamide and nicotinamide: two related anti-inflammatory agents that differentially affect the functions of activated macrophages

Biedroń, Rafał ; Ciszek, Marta ; Tokarczyk, Marianna ; Bobek, Małgorzata ; Kurnyta, Maria ; Słominska, Ewa M ; Smoleński, Ryszard T ; Marcinkiewicz, Janusz

Archivum Immunologiae et Therapiae Experimentalis, 2008-04, Vol.56 (2), p.127-134 [Periódico revisado por pares]

Basel: Basel : Birkhäuser-Verlag

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3
1,3-β-glucanases as candidates in latex-pollen-vegetable food cross-reactivity
1,3-β-glucanases as candidates in latex-pollen-vegetable food cross-reactivity
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1,3-β-glucanases as candidates in latex-pollen-vegetable food cross-reactivity

Palomares, O. ; Villalba, M. ; Quiralte, J. ; Polo, F. ; Rodríguez, R.

Clinical and experimental allergy, 2005-03, Vol.35 (3), p.345-351 [Periódico revisado por pares]

Oxford, UK: Blackwell Science Ltd

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4
1,4-Dihydroxy-2-naphthoic acid from Propionibacterium freudenreichii reduces inflammation in interleukin-10-deficient mice with colitis by suppressing macrophage-derived proinflammatory cytokines
1,4-Dihydroxy-2-naphthoic acid from Propionibacterium freudenreichii reduces inflammation in interleukin-10-deficient mice with colitis by suppressing macrophage-derived proinflammatory cytokines
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1,4-Dihydroxy-2-naphthoic acid from Propionibacterium freudenreichii reduces inflammation in interleukin-10-deficient mice with colitis by suppressing macrophage-derived proinflammatory cytokines

Okada, Yoshikiyo ; Tsuzuki, Yoshikazu ; Narimatsu, Kazuyuki ; Sato, Hirokazu ; Ueda, Toshihide ; Hozumi, Hideaki ; Sato, Shingo ; Hokari, Ryota ; Kurihara, Chie ; Komoto, Shunsuke ; Watanabe, Chikako ; Tomita, Kengo ; Kawaguchi, Atsushi ; Nagao, Shigeaki ; Miura, Soichiro

Journal of leukocyte biology, 2013-09, Vol.94 (3), p.473-480 [Periódico revisado por pares]

United States: Society for Leukocyte Biology

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5
IL-1-induced inflammation promotes development of leishmaniasis in susceptible BALB/c mice
IL-1-induced inflammation promotes development of leishmaniasis in susceptible BALB/c mice
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IL-1-induced inflammation promotes development of leishmaniasis in susceptible BALB/c mice

Voronov, Elena ; Dotan, Shahar ; Gayvoronsky, Lubov ; White, Rosalyn M. ; Cohen, Idan ; Krelin, Yakov ; Benchetrit, Fabrice ; Elkabets, Moshe ; Huszar, Monika ; El-On, Joseph ; Apte, Ron N.

International immunology, 2010-04, Vol.22 (4), p.245-257 [Periódico revisado por pares]

England: Oxford University Press

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6
IL‐1‐like production in adriamycin‐induced nephrotic syndrome in the rat
IL‐1‐like production in adriamycin‐induced nephrotic syndrome in the rat
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IL‐1‐like production in adriamycin‐induced nephrotic syndrome in the rat

BRICIO, T. ; MOLINA, A. ; EGIDO, J. ; GONZALEZ, E. ; MAMPASO, F.

Clinical and experimental immunology, 1992-01, Vol.87 (1), p.117-121 [Periódico revisado por pares]

Oxford, UK: Blackwell Publishing Ltd

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7
IL-1 signal affects both protection and pathogenesis of virus-induced chronic CNS demyelinating disease
IL-1 signal affects both protection and pathogenesis of virus-induced chronic CNS demyelinating disease
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IL-1 signal affects both protection and pathogenesis of virus-induced chronic CNS demyelinating disease

Kim, Byung S ; Jin, Young-Hee ; Meng, Liping ; Hou, Wanqiu ; Kang, Hyun Seok ; Park, Hey Suk ; Koh, Chang-Sung

Journal of neuroinflammation, 2012-09, Vol.9 (1), p.217-217, Article 693 [Periódico revisado por pares]

England: BioMed Central Ltd

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8
IL-10 administration attenuates pulmonary neutrophil infiltration and alters pulmonary iNOS activation following hemorrhagic shock
IL-10 administration attenuates pulmonary neutrophil infiltration and alters pulmonary iNOS activation following hemorrhagic shock
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IL-10 administration attenuates pulmonary neutrophil infiltration and alters pulmonary iNOS activation following hemorrhagic shock

Kobbe, P. ; Schmidt, J. ; Stoffels, B. ; Chanthaphavong, R. S. ; Bauer, A. J. ; Pape, H.-C.

Inflammation research, 2009-03, Vol.58 (3), p.170-174 [Periódico revisado por pares]

Basel: Birkhäuser-Verlag

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9
IL-10 alters DC function via modulation of cell surface molecules resulting in impaired T-cell responses
IL-10 alters DC function via modulation of cell surface molecules resulting in impaired T-cell responses
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IL-10 alters DC function via modulation of cell surface molecules resulting in impaired T-cell responses

McBride, Jacqueline M ; Jung, Thomas ; de Vries, Jan E ; Aversa, Gregorio

Cellular immunology, 2002-02, Vol.215 (2), p.162-172 [Periódico revisado por pares]

Netherlands: Elsevier Inc

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10
IL-10 and IL-27 producing dendritic cells capable of enhancing IL-10 production of T cells are induced in oral tolerance
IL-10 and IL-27 producing dendritic cells capable of enhancing IL-10 production of T cells are induced in oral tolerance
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IL-10 and IL-27 producing dendritic cells capable of enhancing IL-10 production of T cells are induced in oral tolerance

Shiokawa, Aya ; Tanabe, Kosuke ; Tsuji, Noriko M ; Sato, Ryuichiro ; Hachimura, Satoshi

Immunology letters, 2009-06, Vol.125 (1), p.7-14 [Periódico revisado por pares]

Netherlands: Elsevier B.V

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