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Proximal Tubulopathy: Prime Mover and Key Therapeutic Target in Diabetic Kidney Disease
Proximal Tubulopathy: Prime Mover and Key Therapeutic Target in Diabetic Kidney Disease
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Proximal Tubulopathy: Prime Mover and Key Therapeutic Target in Diabetic Kidney Disease

Gilbert, Richard E

Diabetes (New York, N.Y.), 2017-04, Vol.66 (4), p.791-800 [Periódico revisado por pares]

United States: American Diabetes Association

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2
Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion
Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion
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Na+-D-glucose Cotransporter SGLT1 is Pivotal for Intestinal Glucose Absorption and Glucose-Dependent Incretin Secretion

GORBOULEV, Valentin ; SCHÜRMANN, Annette ; VEYHL-WICHMANN, Maike ; SRINIVASAN, Aruna ; BALEN, Daniela ; BRELJAK, Davorka ; REXHEPAJ, Rexhep ; PARKER, Helen E ; GRIBBLE, Fiona M ; REIMANN, Frank ; LANG, Florian ; WIESE, Stefan ; VALLON, Volker ; SABOLIC, Ivan ; SENDTNER, Michael ; KOEPSELL, Hermann ; KIPP, Helmut ; JASCHKE, Alexander ; KLESSEN, Dirk ; FRIEDRICH, Alexandra ; SCHERNECK, Stephan ; RIEG, Timo ; CUNARD, Robyn

Diabetes (New York, N.Y.), 2012-01, Vol.61 (1), p.187-196 [Periódico revisado por pares]

Alexandria, VA: American Diabetes Association

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3
Exocytosis-Mediated Urinary Full-Length Megalin Excretion Is Linked With the Pathogenesis of Diabetic Nephropathy
Exocytosis-Mediated Urinary Full-Length Megalin Excretion Is Linked With the Pathogenesis of Diabetic Nephropathy
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Exocytosis-Mediated Urinary Full-Length Megalin Excretion Is Linked With the Pathogenesis of Diabetic Nephropathy

De, Shankhajit ; Kuwahara, Shoji ; Hosojima, Michihiro ; Ishikawa, Tomomi ; Kaseda, Ryohei ; Sarkar, Piyali ; Yoshioka, Yusuke ; Kabasawa, Hideyuki ; Iida, Tomomichi ; Goto, Sawako ; Toba, Koji ; Higuchi, Yuki ; Suzuki, Yoshiki ; Hara, Masanori ; Kurosawa, Hiroyuki ; Narita, Ichiei ; Hirayama, Yoshiaki ; Ochiya, Takahiro ; Saito, Akihiko

Diabetes (New York, N.Y.), 2017-05, Vol.66 (5), p.1391-1404 [Periódico revisado por pares]

United States: American Diabetes Association

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4
Proximal Tubular Cell-Specific Ablation of Carnitine Acetyltransferase Causes Tubular Disease and Secondary Glomerulosclerosis
Proximal Tubular Cell-Specific Ablation of Carnitine Acetyltransferase Causes Tubular Disease and Secondary Glomerulosclerosis
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Proximal Tubular Cell-Specific Ablation of Carnitine Acetyltransferase Causes Tubular Disease and Secondary Glomerulosclerosis

Kruger, Claudia ; Nguyen, Trang-Tiffany ; Breaux, Chelsea ; Guillory, Alana ; Mangelli, Margaret ; Fridianto, Kevin T ; Kovalik, Jean-Paul ; Burk, David H ; Noland, Robert C ; Mynatt, Randall ; Stadler, Krisztian

Diabetes (New York, N.Y.), 2019-04, Vol.68 (4), p.819-831 [Periódico revisado por pares]

United States: American Diabetes Association

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5
Albumin uptake and processing by the proximal tubule: physiological, pathological, and therapeutic implications
Albumin uptake and processing by the proximal tubule: physiological, pathological, and therapeutic implications
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Albumin uptake and processing by the proximal tubule: physiological, pathological, and therapeutic implications

Molitoris, Bruce A ; Sandoval, Ruben M ; Yadav, Shiv Pratap S ; Wagner, Mark C

Physiological reviews, 2022-10, Vol.102 (4), p.1625-1667 [Periódico revisado por pares]

United States: American Physiological Society

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6
Megalin, cubilin, and Dab2 drive endocytic flux in kidney proximal tubule cells
Megalin, cubilin, and Dab2 drive endocytic flux in kidney proximal tubule cells
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Megalin, cubilin, and Dab2 drive endocytic flux in kidney proximal tubule cells

Rbaibi, Youssef ; Long, Kimberly R ; Shipman, Katherine E ; Ren, Qidong ; Baty, Catherine J ; Kashlan, Ossama B ; Weisz, Ora A Parton, Robert

Molecular biology of the cell, 2023-06, Vol.34 (7), p.ar74-ar74 [Periódico revisado por pares]

United States: The American Society for Cell Biology

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7
Oxidation of Fatty Acids Is the Source of Increased Mitochondrial Reactive Oxygen Species Production in Kidney Cortical Tubules in Early Diabetes
Oxidation of Fatty Acids Is the Source of Increased Mitochondrial Reactive Oxygen Species Production in Kidney Cortical Tubules in Early Diabetes
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Oxidation of Fatty Acids Is the Source of Increased Mitochondrial Reactive Oxygen Species Production in Kidney Cortical Tubules in Early Diabetes

ROSCA, Mariana G ; VAZQUEZ, Edwin J ; QUN CHEN ; KERNER, Janos ; KERN, Timothy S ; HOPPEL, Charles L

Diabetes (New York, N.Y.), 2012-08, Vol.61 (8), p.2074-2083 [Periódico revisado por pares]

Alexandria, VA: American Diabetes Association

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8
Interaction of PPARα With the Canonic Wnt Pathway in the Regulation of Renal Fibrosis
Interaction of PPARα With the Canonic Wnt Pathway in the Regulation of Renal Fibrosis
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Interaction of PPARα With the Canonic Wnt Pathway in the Regulation of Renal Fibrosis

Cheng, Rui ; Ding, Lexi ; He, Xuemin ; Takahashi, Yusuke ; Ma, Jian-Xing

Diabetes (New York, N.Y.), 2016-12, Vol.65 (12), p.3730-3743 [Periódico revisado por pares]

United States: American Diabetes Association

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9
Glucose Transporters in Human Renal Proximal Tubular Cells Isolated From the Urine of Patients With Non–Insulin-Dependent Diabetes
Glucose Transporters in Human Renal Proximal Tubular Cells Isolated From the Urine of Patients With Non–Insulin-Dependent Diabetes
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Glucose Transporters in Human Renal Proximal Tubular Cells Isolated From the Urine of Patients With Non–Insulin-Dependent Diabetes

Rahmoune, Hassan ; Thompson, Paul W ; Ward, Joanna M ; Smith, Chari D ; Hong, Guizhu ; Brown, John

Diabetes (New York, N.Y.), 2005-12, Vol.54 (12), p.3427-3434 [Periódico revisado por pares]

Alexandria, VA: American Diabetes Association

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10
Molecular and Cellular Physiology of Renal Organic Cation and Anion Transport
Molecular and Cellular Physiology of Renal Organic Cation and Anion Transport
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Molecular and Cellular Physiology of Renal Organic Cation and Anion Transport

Wright, Stephen H ; Dantzler, William H

Physiological reviews, 2004-07, Vol.84 (3), p.987-1049 [Periódico revisado por pares]

United States: Am Physiological Soc

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