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Mitochondria and oxidative stress in neurodegenerative disorders

Gary E Gibson; Rajiv R Ratan; M. Flint Beal

Boston, Mass Published by Blackwell Publishing on behalf of the New York Academy of Sciences 2008

Acceso en línea. La biblioteca tiene también copias físicas.

  • Título:
    Mitochondria and oxidative stress in neurodegenerative disorders
  • Autor: Gary E Gibson; Rajiv R Ratan; M. Flint Beal
  • Materias: Nervous system -- Degeneration -- Pathophysiology; Mitochondrial pathology; Oxidative stress -- Pathophysiology; Neurodegenerative Diseases -- physiopathology; Mitochondria -- physiology; Oxidative Stress -- physiology; MITOCÔNDRIAS (PATOLOGIA FISIOLOGIA); ESTRESSE OXIDATIVO (FISIOPATOLOGIA FISIOLOGIA); DOENÇAS NEURODEGENERATIVAS (FISIOPATOLOGIA)
  • Notas: Includes bibliographical references and index
  • Descripción: Mitochondrial approaches for neuroprotection / Small molecule activation of adaptive gene expression: Tilorone or its analogs are novel potent activators of hypoxia inducible factor-1 that provide prophylaxis against stroke and spinal cord injury / Mitochondria and huntington's disease pathogenesis: Insight from genetic and chemical models / Screening for the metabolic basis of neurodegeneration: Developing a focused proteomic approach / Regulation of forkhead transcription factor foxo3a contributes to calorie restriction-induced prevention of alzheimer's disease-type amyloid neuropathology and spatial memory deterioration / Nuclear control of respiratory chain expression by nuclear respiratory factors and pgc-1-related coactivator / Mitochondria and hypoxia-induced gene expression mediated by hypoxia-inducible factors / Dysfunction of mitochondria and sarcoplasmic reticulum in the pathogenesis of collagen vi muscular dystrophies / The disulfide relay of the intermembrane space of mitochondria: An oxygen-sensing system? / Impairing the mitochondrial fission and fusion balance: A new mechanism of neurodegeneration / Preventing nad(+) depletion protects neurons against excitotoxicity: Bioenergetic effects of mild mitochondrial uncoupling and caloric restriction / The mitochondrial antioxidants mitoe(2) and mitoq(10) increase mitochondrial ca(2+) load upon cell stimulation by inhibiting ca(2+) efflux from the organelle / Developmental restructuring of the creatine kinase system integrates mitochondrial energetics with stem cell cardiogenesis / Ro
  • Títulos relacionados: Serie:Annals of the New York Academy of Sciences v. 1147
  • Editor: Boston, Mass Published by Blackwell Publishing on behalf of the New York Academy of Sciences
  • Fecha de creación: 2008
  • Formato: xii, 414 p ill. (some col.) 26 cm.
  • Idioma: Inglés
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