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1
Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons
Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons
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Endogenous Nmnat2 is an essential survival factor for maintenance of healthy axons

Gilley, Jonathan ; Coleman, Michael P Barres, Ben A.

PLoS biology, 2010-01, Vol.8 (1), p.e1000300-e1000300 [Periódico revisado por pares]

United States: Public Library of Science

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2
Traumatic Axonal Injury: Mechanisms and Translational Opportunities
Traumatic Axonal Injury: Mechanisms and Translational Opportunities
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Traumatic Axonal Injury: Mechanisms and Translational Opportunities

Hill, Ciaran S ; Coleman, Michael P ; Menon, David K

Trends in neurosciences (Regular ed.), 2016-05, Vol.39 (5), p.311-324 [Periódico revisado por pares]

England: Elsevier Ltd

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3
Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons
Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons
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Absence of SARM1 Rescues Development and Survival of NMNAT2-Deficient Axons

Gilley, Jonathan ; Orsomando, Giuseppe ; Nascimento-Ferreira, Isabel ; Coleman, Michael P.

Cell reports (Cambridge), 2015-03, Vol.10 (12), p.1974-1981 [Periódico revisado por pares]

United States: Elsevier Inc

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4
Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta
Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta
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Lipopolysaccharide-induced neuroinflammation induces presynaptic disruption through a direct action on brain tissue involving microglia-derived interleukin 1 beta

Sheppard, Olivia ; Coleman, Michael P ; Durrant, Claire S

Journal of neuroinflammation, 2019-05, Vol.16 (1), p.106-106, Article 106 [Periódico revisado por pares]

England: BioMed Central Ltd

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5
Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2
Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2
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Subcellular localization determines the stability and axon protective capacity of axon survival factor Nmnat2

Milde, Stefan ; Gilley, Jonathan ; Coleman, Michael P Barres, Ben A.

PLoS biology, 2013-04, Vol.11 (4), p.e1001539-e1001539 [Periódico revisado por pares]

United States: Public Library of Science

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6
NAD + cleavage activity by animal and plant TIR domains in cell death pathways
NAD + cleavage activity by animal and plant TIR domains in cell death pathways
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NAD + cleavage activity by animal and plant TIR domains in cell death pathways

Horsefield, Shane ; Burdett, Hayden ; Zhang, Xiaoxiao ; Manik, Mohammad K ; Shi, Yun ; Chen, Jian ; Qi, Tiancong ; Gilley, Jonathan ; Lai, Jhih-Siang ; Rank, Maxwell X ; Casey, Lachlan W ; Gu, Weixi ; Ericsson, Daniel J ; Foley, Gabriel ; Hughes, Robert O ; Bosanac, Todd ; von Itzstein, Mark ; Rathjen, John P ; Nanson, Jeffrey D ; Boden, Mikael ; Dry, Ian B ; Williams, Simon J ; Staskawicz, Brian J ; Coleman, Michael P ; Ve, Thomas ; Dodds, Peter N ; Kobe, Bostjan

Science (American Association for the Advancement of Science), 2019-08, Vol.365 (6455), p.793-799 [Periódico revisado por pares]

United States: The American Association for the Advancement of Science

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7
Low levels of NMNAT2 compromise axon development and survival
Low levels of NMNAT2 compromise axon development and survival
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Low levels of NMNAT2 compromise axon development and survival

Gilley, Jonathan ; Mayer, Paul R ; Yu, Gang ; Coleman, Michael P

Human molecular genetics, 2019-02, Vol.28 (3), p.448-458 [Periódico revisado por pares]

England: Oxford University Press

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8
Mitochondrial impairment activates the Wallerian pathway through depletion of NMNAT2 leading to SARM1-dependent axon degeneration
Mitochondrial impairment activates the Wallerian pathway through depletion of NMNAT2 leading to SARM1-dependent axon degeneration
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Mitochondrial impairment activates the Wallerian pathway through depletion of NMNAT2 leading to SARM1-dependent axon degeneration

Loreto, Andrea ; Hill, Ciaran S. ; Hewitt, Victoria L. ; Orsomando, Giuseppe ; Angeletti, Carlo ; Gilley, Jonathan ; Lucci, Cristiano ; Sanchez-Martinez, Alvaro ; Whitworth, Alexander J. ; Conforti, Laura ; Dajas-Bailador, Federico ; Coleman, Michael P.

Neurobiology of disease, 2020-02, Vol.134, p.104678-104678, Article 104678 [Periódico revisado por pares]

United States: Elsevier Inc

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9
Rescue of peripheral and CNS axon defects in mice lacking NMNAT2
Rescue of peripheral and CNS axon defects in mice lacking NMNAT2
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Rescue of peripheral and CNS axon defects in mice lacking NMNAT2

Gilley, Jonathan ; Adalbert, Robert ; Yu, Gang ; Coleman, Michael P

The Journal of neuroscience, 2013-08, Vol.33 (33), p.13410-13424 [Periódico revisado por pares]

United States: Society for Neuroscience

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10
TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD
TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD
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TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD

White, Matthew A ; Kim, Eosu ; Duffy, Amanda ; Adalbert, Robert ; Phillips, Benjamin U ; Peters, Owen M ; Stephenson, Jodie ; Yang, Sujeong ; Massenzio, Francesca ; Lin, Ziqiang ; Andrews, Simon ; Segonds-Pichon, Anne ; Metterville, Jake ; Saksida, Lisa M ; Mead, Richard ; Ribchester, Richard R ; Barhomi, Youssef ; Serre, Thomas ; Coleman, Michael P ; Fallon, Justin R ; Bussey, Timothy J ; Brown, Jr, Robert H ; Sreedharan, Jemeen

Nature neuroscience, 2018-04, Vol.21 (4), p.552-563 [Periódico revisado por pares]

United States: Nature Publishing Group

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