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Cellular and molecular mechanisms of mitochondrial function
Cellular and molecular mechanisms of mitochondrial function
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Cellular and molecular mechanisms of mitochondrial function

Osellame, Laura D., B.Sc (Hons), Ph.D ; Blacker, Thomas S., MPhys, MRes ; Duchen, Michael R., BA (Oxon), MBBS, MRCP, PhD, MAE, FSB

Best Practice & Research Clinical Endocrinology & Metabolism, 2012-12, Vol.26 (6), p.711-723 [Periódico revisado por pares]

Netherlands: Elsevier Ltd

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Reversal of Mitochondrial Transhydrogenase Causes Oxidative Stress in Heart Failure
Reversal of Mitochondrial Transhydrogenase Causes Oxidative Stress in Heart Failure
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Reversal of Mitochondrial Transhydrogenase Causes Oxidative Stress in Heart Failure

Nickel, Alexander G. ; von Hardenberg, Albrecht ; Hohl, Mathias ; Löffler, Joachim R. ; Kohlhaas, Michael ; Becker, Janne ; Reil, Jan-Christian ; Kazakov, Andrey ; Bonnekoh, Julia ; Stadelmaier, Moritz ; Puhl, Sarah-Lena ; Wagner, Michael ; Bogeski, Ivan ; Cortassa, Sonia ; Kappl, Reinhard ; Pasieka, Bastian ; Lafontaine, Michael ; Lancaster, C. Roy D. ; Blacker, Thomas S. ; Hall, Andrew R. ; Duchen, Michael R. ; Kästner, Lars ; Lipp, Peter ; Zeller, Tanja ; Müller, Christian ; Knopp, Andreas ; Laufs, Ulrich ; Böhm, Michael ; Hoth, Markus ; Maack, Christoph

Cell metabolism, 2015-09, Vol.22 (3), p.472-484 [Periódico revisado por pares]

United States: Elsevier Inc

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3
Separating NADH and NADPH fluorescence in live cells and tissues using FLIM
Separating NADH and NADPH fluorescence in live cells and tissues using FLIM
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Separating NADH and NADPH fluorescence in live cells and tissues using FLIM

Blacker, Thomas S ; Mann, Zoe F ; Gale, Jonathan E ; Ziegler, Mathias ; Bain, Angus J ; Szabadkai, Gyorgy ; Duchen, Michael R

Nature communications, 2014-05, Vol.5 (1), p.3936-3936, Article 3936 [Periódico revisado por pares]

England: Nature Publishing Group

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4
Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells
Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells
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Diabetes causes marked inhibition of mitochondrial metabolism in pancreatic β-cells

Haythorne, Elizabeth ; Rohm, Maria ; van de Bunt, Martijn ; Brereton, Melissa F ; Tarasov, Andrei I ; Blacker, Thomas S ; Sachse, Gregor ; Silva Dos Santos, Mariana ; Terron Exposito, Raul ; Davis, Simon ; Baba, Otto ; Fischer, Roman ; Duchen, Michael R ; Rorsman, Patrik ; MacRae, James I ; Ashcroft, Frances M

Nature communications, 2019-06, Vol.10 (1), p.2474-2474, Article 2474 [Periódico revisado por pares]

England: Nature Publishing Group

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5
Nrf2 regulates glucose uptake and metabolism in neurons and astrocytes
Nrf2 regulates glucose uptake and metabolism in neurons and astrocytes
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Nrf2 regulates glucose uptake and metabolism in neurons and astrocytes

Esteras, Noemí ; Blacker, Thomas S. ; Zherebtsov, Evgeny A. ; Stelmashuk, Olga A. ; Zhang, Ying ; Wigley, W. Christian ; Duchen, Michael R. ; Dinkova-Kostova, Albena T. ; Abramov, Andrey Y.

Redox biology, 2023-06, Vol.62, p.102672-102672, Article 102672 [Periódico revisado por pares]

Netherlands: Elsevier B.V

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6
Gradients of glucose metabolism regulate morphogen signalling required for specifying tonotopic organization in the chicken cochlea
Gradients of glucose metabolism regulate morphogen signalling required for specifying tonotopic organization in the chicken cochlea
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Gradients of glucose metabolism regulate morphogen signalling required for specifying tonotopic organization in the chicken cochlea

O'Sullivan, James D B ; Blacker, Thomas S ; Scott, Claire ; Chang, Weise ; Ahmed, Mohi ; Yianni, Val ; Mann, Zoe F

eLife, 2023-08, Vol.12 [Periódico revisado por pares]

England: eLife Science Publications, Ltd

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7
Photoactivated cell-killing involving a low molecular weight, donor-acceptor diphenylacetylene
Photoactivated cell-killing involving a low molecular weight, donor-acceptor diphenylacetylene
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Photoactivated cell-killing involving a low molecular weight, donor-acceptor diphenylacetylene

Chisholm, David R ; Lamb, Rebecca ; Pallett, Tommy ; Affleck, Valerie ; Holden, Claire ; Marrison, Joanne ; O'Toole, Peter ; Ashton, Peter D ; Newling, Katherine ; Steffen, Andreas ; Nelson, Amanda K ; Mahler, Christoph ; Valentine, Roy ; Blacker, Thomas S ; Bain, Angus J ; Girkin, John ; Marder, Todd B ; Whiting, Andrew ; Ambler, Carrie A

Chemical science (Cambridge), 2019-05, Vol.1 (17), p.4673-4683 [Periódico revisado por pares]

England: Royal Society of Chemistry

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8
CHCHD4 regulates tumour proliferation and EMT-related phenotypes, through respiratory chain-mediated metabolism
CHCHD4 regulates tumour proliferation and EMT-related phenotypes, through respiratory chain-mediated metabolism
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CHCHD4 regulates tumour proliferation and EMT-related phenotypes, through respiratory chain-mediated metabolism

Thomas, Luke W ; Esposito, Cinzia ; Stephen, Jenna M ; Costa, Ana S H ; Frezza, Christian ; Blacker, Thomas S ; Szabadkai, Gyorgy ; Ashcroft, Margaret

Cancer & metabolism, 2019-07, Vol.7 (1), p.7-7, Article 7 [Periódico revisado por pares]

England: BioMed Central Ltd

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9
NAD(P)H binding configurations revealed by time-resolved fluorescence and two-photon absorption
NAD(P)H binding configurations revealed by time-resolved fluorescence and two-photon absorption
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NAD(P)H binding configurations revealed by time-resolved fluorescence and two-photon absorption

Blacker, Thomas S. ; Duchen, Michael R. ; Bain, Angus J.

Biophysical journal, 2023-04, Vol.122 (7), p.1240-1253 [Periódico revisado por pares]

United States: Elsevier Inc

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10
The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α
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The mitochondrial calcium uniporter regulates breast cancer progression via HIF‐1α

Tosatto, Anna ; Sommaggio, Roberta ; Kummerow, Carsten ; Bentham, Robert B ; Blacker, Thomas S ; Berecz, Tunde ; Duchen, Michael R ; Rosato, Antonio ; Bogeski, Ivan ; Szabadkai, Gyorgy ; Rizzuto, Rosario ; Mammucari, Cristina

EMBO molecular medicine, 2016-05, Vol.8 (5), p.569-585 [Periódico revisado por pares]

Germany: John Wiley & Sons, Inc

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