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Traumatic scratch injury in astrocytes triggers calcium influx to activate the JNK/c-Jun/AP-1 pathway and switch on GFAP expression
Traumatic scratch injury in astrocytes triggers calcium influx to activate the JNK/c-Jun/AP-1 pathway and switch on GFAP expression
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Traumatic scratch injury in astrocytes triggers calcium influx to activate the JNK/c-Jun/AP-1 pathway and switch on GFAP expression

Gao, Kai ; Wang, Chen Ran ; Jiang, Feng ; Wong, Ann Yuen Kwan ; Su, Na ; Jiang, Jiao Hua ; Chai, Rui Chao ; Vatcher, Greg ; Teng, Junlin ; Chen, Jianguo ; Jiang, Yu-Wu ; Yu, Albert Cheung Hoi

Glia, 2013-12, Vol.61 (12), p.2063-2077 [Periódico revisado por pares]

United States: Blackwell Publishing Ltd

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2
Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation
Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation
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Induction of dsRNA-activated protein kinase links mitochondrial unfolded protein response to the pathogenesis of intestinal inflammation

Rath, Eva ; Berger, Emanuel ; Messlik, Anja ; Nunes, Tiago ; Liu, Bo ; Kim, Sandy C ; Hoogenraad, Nick ; Sans, Miquel ; Sartor, R Balfour ; Haller, Dirk

Gut, 2012-09, Vol.61 (9), p.1269-1278 [Periódico revisado por pares]

London: BMJ Publishing Group Ltd and British Society of Gastroenterology

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3
Together JUN and DDIT3 (CHOP) control retinal ganglion cell death after axonal injury
Together JUN and DDIT3 (CHOP) control retinal ganglion cell death after axonal injury
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Together JUN and DDIT3 (CHOP) control retinal ganglion cell death after axonal injury

Syc-Mazurek, Stephanie B ; Fernandes, Kimberly A ; Wilson, Michael P ; Shrager, Peter ; Libby, Richard T

Molecular neurodegeneration, 2017-10, Vol.12 (1), p.71-71, Article 71 [Periódico revisado por pares]

England: BioMed Central Ltd

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4
Nonuniform molecular features of myelinating Schwann cells in models for CMT1: Distinct disease patterns are associated with NCAM and c-Jun upregulation
Nonuniform molecular features of myelinating Schwann cells in models for CMT1: Distinct disease patterns are associated with NCAM and c-Jun upregulation
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Nonuniform molecular features of myelinating Schwann cells in models for CMT1: Distinct disease patterns are associated with NCAM and c-Jun upregulation

Klein, Dennis ; Groh, Janos ; Wettmarshausen, Jennifer ; Martini, Rudolf

Glia, 2014-05, Vol.62 (5), p.736-750 [Periódico revisado por pares]

United States: Blackwell Publishing Ltd

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5
Coupling Computational and Intracellular Screening and Selection Toward Co-compatible cJun and cFos Antagonists
Coupling Computational and Intracellular Screening and Selection Toward Co-compatible cJun and cFos Antagonists
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Coupling Computational and Intracellular Screening and Selection Toward Co-compatible cJun and cFos Antagonists

Lathbridge, Alexander ; Michalowska, Anna S ; Mason, Jody M

Biochemistry (Easton), 2020-02, Vol.59 (4), p.530-540 [Periódico revisado por pares]

United States: American Chemical Society

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6
Transcriptome of the inner circular smooth muscle of the developing mouse intestine: Evidence for regulation of visceral smooth muscle genes by the hedgehog target gene, cJun
Transcriptome of the inner circular smooth muscle of the developing mouse intestine: Evidence for regulation of visceral smooth muscle genes by the hedgehog target gene, cJun
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Transcriptome of the inner circular smooth muscle of the developing mouse intestine: Evidence for regulation of visceral smooth muscle genes by the hedgehog target gene, cJun

Gurdziel, Katherine ; Vogt, Kyle R. ; Walton, Katherine D. ; Schneider, Gary K. ; Gumucio, Deborah L.

Developmental dynamics, 2016-05, Vol.245 (5), p.614-626 [Periódico revisado por pares]

United States: Wiley Subscription Services, Inc

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7
Upregulation of thioredoxin system via Nrf2-antioxidant responsive element pathway in adaptive-retinal neuroprotection in vivo and in vitro
Upregulation of thioredoxin system via Nrf2-antioxidant responsive element pathway in adaptive-retinal neuroprotection in vivo and in vitro
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Upregulation of thioredoxin system via Nrf2-antioxidant responsive element pathway in adaptive-retinal neuroprotection in vivo and in vitro

Tanito, Masaki ; Agbaga, Martin-Paul ; Anderson, Robert E.

Free radical biology & medicine, 2007-06, Vol.42 (12), p.1838-1850 [Periódico revisado por pares]

United States: Elsevier Inc

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8
Roles of Binding Elements, FOXL2 Domains, and Interactions With cJUN and SMADs in Regulation of FSHβ
Roles of Binding Elements, FOXL2 Domains, and Interactions With cJUN and SMADs in Regulation of FSHβ
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Roles of Binding Elements, FOXL2 Domains, and Interactions With cJUN and SMADs in Regulation of FSHβ

Roybal, Lacey L ; Hambarchyan, Arpi ; Meadows, Jason D ; Barakat, Nermeen H ; Pepa, Patricia A ; Breen, Kellie M ; Mellon, Pamela L ; Coss, Djurdjica

Molecular endocrinology (Baltimore, Md.), 2014-10, Vol.28 (10), p.1640-1655 [Periódico revisado por pares]

United States: Endocrine Society

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9
c-Jun Is Essential for Organization of the Epidermal Leading Edge
c-Jun Is Essential for Organization of the Epidermal Leading Edge
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c-Jun Is Essential for Organization of the Epidermal Leading Edge

Li, Guochun ; Gustafson-Brown, Cindy ; Hanks, Steven K ; Nason, Katie ; Arbeit, Jeffrey M ; Pogliano, Kit ; Wisdom, Ronald M ; Johnson, Randall S

Developmental cell, 2003-06, Vol.4 (6), p.865-877 [Periódico revisado por pares]

United States: Elsevier Inc

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10
Intrathecal application of short interfering RNA knocks down c-jun expression and augments spinal motoneuron death after root avulsion in adult rats
Intrathecal application of short interfering RNA knocks down c-jun expression and augments spinal motoneuron death after root avulsion in adult rats
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Intrathecal application of short interfering RNA knocks down c-jun expression and augments spinal motoneuron death after root avulsion in adult rats

Cheng, X ; Fu, R ; Gao, M ; Liu, S ; Li, Y.-Q ; Song, F.-H ; Bruce, I.C ; Zhou, L.-H ; Wu, W

Neuroscience, 2013-06, Vol.241, p.268-279 [Periódico revisado por pares]

Amsterdam: Elsevier Ltd

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