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Regulation of closely juxtaposed proto-oncogene c-fms and HMGXB3 gene expression by mRNA 3' end polymorphism in breast cancer cells
Regulation of closely juxtaposed proto-oncogene c-fms and HMGXB3 gene expression by mRNA 3' end polymorphism in breast cancer cells
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Regulation of closely juxtaposed proto-oncogene c-fms and HMGXB3 gene expression by mRNA 3' end polymorphism in breast cancer cells

Woo, Ho-Hyung ; Chambers, Setsuko K

RNA (Cambridge), 2021-09, Vol.27 (9), p.1068-1081 [Periódico revisado por pares]

United States: Cold Spring Harbor Laboratory Press

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2
Deletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populations
Deletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populations
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Deletion of a Csf1r enhancer selectively impacts CSF1R expression and development of tissue macrophage populations

Rojo, Rocío ; Raper, Anna ; Ozdemir, Derya D ; Lefevre, Lucas ; Grabert, Kathleen ; Wollscheid-Lengeling, Evi ; Bradford, Barry ; Caruso, Melanie ; Gazova, Iveta ; Sánchez, Alejandra ; Lisowski, Zofia M ; Alves, Joana ; Molina-Gonzalez, Irene ; Davtyan, Hayk ; Lodge, Rebecca J ; Glover, James D ; Wallace, Robert ; Munro, David A D ; David, Eyal ; Amit, Ido ; Miron, Véronique E ; Priller, Josef ; Jenkins, Stephen J ; Hardingham, Giles E ; Blurton-Jones, Mathew ; Mabbott, Neil A ; Summers, Kim M ; Hohenstein, Peter ; Hume, David A ; Pridans, Clare

Nature communications, 2019-07, Vol.10 (1), p.3215-17, Article 3215 [Periódico revisado por pares]

England: Nature Publishing Group

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3
E2a Pbx1 oncogene inhibits terminal differentiation but not myeloid potential of pro-T cells
E2a Pbx1 oncogene inhibits terminal differentiation but not myeloid potential of pro-T cells
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E2a Pbx1 oncogene inhibits terminal differentiation but not myeloid potential of pro-T cells

BOURETTE, R. P ; GRASSET, M.-F ; MOUCHIROUD, G

Oncogene, 2007-01, Vol.26 (2), p.234-247 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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4
Microglia derived from aging mice exhibit an altered inflammatory profile
Microglia derived from aging mice exhibit an altered inflammatory profile
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Microglia derived from aging mice exhibit an altered inflammatory profile

Sierra, Amanda ; Gottfried-Blackmore, Andres C. ; McEwen, Bruce S. ; Bulloch, Karen

Glia, 2007-03, Vol.55 (4), p.412-424 [Periódico revisado por pares]

Hoboken: Wiley Subscription Services, Inc., A Wiley Company

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5
A highly conserved c-fms gene intronic element controls macrophage-specific and regulated expression
A highly conserved c-fms gene intronic element controls macrophage-specific and regulated expression
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A highly conserved c-fms gene intronic element controls macrophage-specific and regulated expression

Himes, S. Roy ; Tagoh, Hiromi ; Goonetilleke, Nilukshi ; Sasmono, Tedjo ; Oceandy, Delvac ; Clark, Richard ; Bonifer, Constanze ; Hume, David A.

Journal of leukocyte biology, 2001-11, Vol.70 (5), p.812-820 [Periódico revisado por pares]

United States: Society for Leukocyte Biology

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6
M2 differentiation of MonoMac‐1 cell line induced by M‐CSF and glucocorticoid pathways
M2 differentiation of MonoMac‐1 cell line induced by M‐CSF and glucocorticoid pathways
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M2 differentiation of MonoMac‐1 cell line induced by M‐CSF and glucocorticoid pathways

Vakili, Sarah ; Fischer, Tracy ; Rappaport, Jay

Journal of cellular physiology, 2020-10, Vol.235 (10), p.7383-7391 [Periódico revisado por pares]

United States: Wiley Subscription Services, Inc

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7
Inhibition of IL-34 Unveils Tissue-Selectivity and Is Sufficient to Reduce Microglial Proliferation in a Model of Chronic Neurodegeneration
Inhibition of IL-34 Unveils Tissue-Selectivity and Is Sufficient to Reduce Microglial Proliferation in a Model of Chronic Neurodegeneration
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Inhibition of IL-34 Unveils Tissue-Selectivity and Is Sufficient to Reduce Microglial Proliferation in a Model of Chronic Neurodegeneration

Obst, Juliane ; Simon, Emilie ; Martin-Estebane, Maria ; Pipi, Elena ; Barkwill, Liana M ; Gonzalez-Rivera, Ivette ; Buchanan, Fergus ; Prescott, Alan R ; Faust, Dorte ; Fox, Simon ; Brownlees, Janet ; Taylor, Debra ; Perry, V Hugh ; Nuthall, Hugh ; Atkinson, Peter J ; Karran, Eric ; Routledge, Carol ; Gomez-Nicola, Diego

Frontiers in immunology, 2020-10, Vol.11, p.579000-579000 [Periódico revisado por pares]

Switzerland: Frontiers Research Foundation

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8
Tumor Necrosis Factor-α Increases Circulating Osteoclast Precursor Numbers by Promoting Their Proliferation and Differentiation in the Bone Marrow through Up-regulation of c-Fms Expression
Tumor Necrosis Factor-α Increases Circulating Osteoclast Precursor Numbers by Promoting Their Proliferation and Differentiation in the Bone Marrow through Up-regulation of c-Fms Expression
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Tumor Necrosis Factor-α Increases Circulating Osteoclast Precursor Numbers by Promoting Their Proliferation and Differentiation in the Bone Marrow through Up-regulation of c-Fms Expression

Yao, Zhenqiang ; Li, Ping ; Zhang, Qian ; Schwarz, Edward M. ; Keng, Peter ; Arbini, Arnaldo ; Boyce, Brendan F. ; Xing, Lianping

The Journal of biological chemistry, 2006-04, Vol.281 (17), p.11846-11855 [Periódico revisado por pares]

United States: Elsevier Inc

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9
FMS Kinase Inhibitors: Current Status and Future Prospects
FMS Kinase Inhibitors: Current Status and Future Prospects
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FMS Kinase Inhibitors: Current Status and Future Prospects

El-Gamal, Mohammed I. ; Anbar, Hanan S. ; Yoo, Kyung Ho ; Oh, Chang-Hyun

Medicinal research reviews, 2013-05, Vol.33 (3), p.599-636 [Periódico revisado por pares]

United States: Blackwell Publishing Ltd

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10
NF-IL6 and HSF1 Have Mutually Antagonistic Effects on Transcription in Monocytic Cells
NF-IL6 and HSF1 Have Mutually Antagonistic Effects on Transcription in Monocytic Cells
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NF-IL6 and HSF1 Have Mutually Antagonistic Effects on Transcription in Monocytic Cells

Xie, Yue ; Chen, Changmin ; Stevenson, Mary Ann ; Hume, David A. ; Auron, Philip E. ; Calderwood, Stuart K.

Biochemical and biophysical research communications, 2002-03, Vol.291 (4), p.1071-1080 [Periódico revisado por pares]

United States: Elsevier Inc

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