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1
Cannabinoids inhibit energetic metabolism and induce AMPK-dependent autophagy in pancreatic cancer cells
Cannabinoids inhibit energetic metabolism and induce AMPK-dependent autophagy in pancreatic cancer cells
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Cannabinoids inhibit energetic metabolism and induce AMPK-dependent autophagy in pancreatic cancer cells

Dando, I ; Donadelli, M ; Costanzo, C ; Dalla Pozza, E ; D'Alessandro, A ; Zolla, L ; Palmieri, M

Cell death & disease, 2013-06, Vol.4 (6), p.e664-e664 [Periódico revisado por pares]

England: Springer Nature B.V

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2
Gemcitabine cannabinoid combination triggers autophagy in pancreatic cancer cells through a ROS-mediated mechanism
Gemcitabine cannabinoid combination triggers autophagy in pancreatic cancer cells through a ROS-mediated mechanism
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Gemcitabine cannabinoid combination triggers autophagy in pancreatic cancer cells through a ROS-mediated mechanism

Donadelli, M ; Dando, I ; Zaniboni, T ; Costanzo, C ; Dalla Pozza, E ; Scupoli, M T ; Scarpa, A ; Zappavigna, S ; Marra, M ; Abbruzzese, A ; Bifulco, M ; Caraglia, M ; Palmieri, M

Cell death & disease, 2011-04, Vol.2 (4), p.e152-e152 [Periódico revisado por pares]

England: Springer Nature B.V

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3
Growth delay of human pancreatic cancer cells by methylase inhibitor 5-aza-2′-deoxycytidine treatment is associated with activation of the interferon signalling pathway
Growth delay of human pancreatic cancer cells by methylase inhibitor 5-aza-2′-deoxycytidine treatment is associated with activation of the interferon signalling pathway
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Growth delay of human pancreatic cancer cells by methylase inhibitor 5-aza-2′-deoxycytidine treatment is associated with activation of the interferon signalling pathway

MISSIAGLIA, Edoardo ; DONADELLI, Massimo ; PALMIERI, Marta ; CRNOGORAC-JURCEVIC, Tatjana ; SCARPA, Aldo ; LEMOINE, Nicholas R

Oncogene, 2005-01, Vol.24 (1), p.199-211 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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4
B-cell receptor signaling activity identifies patients with mantle cell lymphoma at higher risk of progression
B-cell receptor signaling activity identifies patients with mantle cell lymphoma at higher risk of progression
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B-cell receptor signaling activity identifies patients with mantle cell lymphoma at higher risk of progression

Gambino, Simona ; Quaglia, Francesca Maria ; Galasso, Marilisa ; Cavallini, Chiara ; Chignola, Roberto ; Lovato, Ornella ; Giacobazzi, Luca ; Caligola, Simone ; Adamo, Annalisa ; Putta, Santosh ; Aparo, Antonino ; Ferrarini, Isacco ; Ugel, Stefano ; Giugno, Rosalba ; Donadelli, Massimo ; Dando, Ilaria ; Krampera, Mauro ; Visco, Carlo ; Scupoli, Maria Teresa

Scientific reports, 2024-03, Vol.14 (1), p.6595-6595, Article 6595 [Periódico revisado por pares]

England: Nature Publishing Group

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5
To metabolomics and beyond: a technological portfolio to investigate cancer metabolism
To metabolomics and beyond: a technological portfolio to investigate cancer metabolism
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To metabolomics and beyond: a technological portfolio to investigate cancer metabolism

Danzi, Federica ; Pacchiana, Raffaella ; Mafficini, Andrea ; Scupoli, Maria T ; Scarpa, Aldo ; Donadelli, Massimo ; Fiore, Alessandra

Signal transduction and targeted therapy, 2023-03, Vol.8 (1), p.137-137, Article 137 [Periódico revisado por pares]

England: Nature Publishing Group

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6
Mutant p53 blocks SESN1/AMPK/PGC-1α/UCP2 axis increasing mitochondrial O2ˉ· production in cancer cells
Mutant p53 blocks SESN1/AMPK/PGC-1α/UCP2 axis increasing mitochondrial O2ˉ· production in cancer cells
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Mutant p53 blocks SESN1/AMPK/PGC-1α/UCP2 axis increasing mitochondrial O2ˉ· production in cancer cells

Cordani, Marco ; Butera, Giovanna ; Dando, Ilaria ; Torrens-Mas, Margalida ; Butturini, Elena ; Pacchiana, Raffaella ; Oppici, Elisa ; Cavallini, Chiara ; Gasperini, Sara ; Tamassia, Nicola ; Nadal-Serrano, Mercedes ; Coan, Michela ; Rossi, Davide ; Gaidano, Gianluca ; Caraglia, Michele ; Mariotto, Sofia ; Spizzo, Riccardo ; Roca, Pilar ; Oliver, Jordi ; Scupoli, Maria Teresa ; Donadelli, Massimo

British journal of cancer, 2018-10, Vol.119 (8), p.994-1008 [Periódico revisado por pares]

London: Nature Publishing Group

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7
Autophagy and chemotherapy resistance: a promising therapeutic target for cancer treatment
Autophagy and chemotherapy resistance: a promising therapeutic target for cancer treatment
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Autophagy and chemotherapy resistance: a promising therapeutic target for cancer treatment

Sui, X ; Chen, R ; Wang, Z ; Huang, Z ; Kong, N ; Zhang, M ; Han, W ; Lou, F ; Yang, J ; Zhang, Q ; Wang, X ; He, C ; Pan, H

Cell death & disease, 2013-10, Vol.4 (10), p.e838-e838 [Periódico revisado por pares]

England: Springer Nature B.V

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8
Integrated stress response is critical for gemcitabine resistance in pancreatic ductal adenocarcinoma
Integrated stress response is critical for gemcitabine resistance in pancreatic ductal adenocarcinoma
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Integrated stress response is critical for gemcitabine resistance in pancreatic ductal adenocarcinoma

Palam, L R ; Gore, J ; Craven, K E ; Wilson, J L ; Korc, M

Cell death & disease, 2015-10, Vol.6 (10), p.e1913-e1913 [Periódico revisado por pares]

England: Springer Nature B.V

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9
Defects in the mitochondrial-tRNA modification enzymes MTO1 and GTPBP3 promote different metabolic reprogramming through a HIF-PPARγ-UCP2-AMPK axis
Defects in the mitochondrial-tRNA modification enzymes MTO1 and GTPBP3 promote different metabolic reprogramming through a HIF-PPARγ-UCP2-AMPK axis
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Defects in the mitochondrial-tRNA modification enzymes MTO1 and GTPBP3 promote different metabolic reprogramming through a HIF-PPARγ-UCP2-AMPK axis

Boutoual, Rachid ; Meseguer, Salvador ; Villarroya, Magda ; Martín-Hernández, Elena ; Errami, Mohammed ; Martín, Miguel A ; Casado, Marta ; Armengod, M-Eugenia

Scientific reports, 2018-01, Vol.8 (1), p.1163-18, Article 1163 [Periódico revisado por pares]

England: Nature Publishing Group

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10
Albumin-induced apoptosis of tubular cells is modulated by BASP1
Albumin-induced apoptosis of tubular cells is modulated by BASP1
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Albumin-induced apoptosis of tubular cells is modulated by BASP1

Sanchez-Niño, M D ; Fernandez-Fernandez, B ; Perez-Gomez, M V ; Poveda, J ; Sanz, A B ; Cannata-Ortiz, P ; Ruiz-Ortega, M ; Egido, J ; Selgas, R ; Ortiz, A

Cell death & disease, 2015-02, Vol.6 (2), p.e1644-e1644 [Periódico revisado por pares]

England: Springer Nature B.V

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