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Refinado por: assunto: Medical Sciences remover assunto: Rats remover
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1
Pivotal roles of p53 transcription-dependent and -independent pathways in manganese-induced mitochondrial dysfunction and neuronal apoptosis
Pivotal roles of p53 transcription-dependent and -independent pathways in manganese-induced mitochondrial dysfunction and neuronal apoptosis
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Pivotal roles of p53 transcription-dependent and -independent pathways in manganese-induced mitochondrial dysfunction and neuronal apoptosis

Wan, Chunhua ; Ma, Xa ; Shi, Shangshi ; Zhao, Jianya ; Nie, Xiaoke ; Han, Jingling ; Xiao, Jing ; Wang, Xiaoke ; Jiang, Shengyang ; Jiang, Junkang

Toxicology and applied pharmacology, 2014-12, Vol.281 (3), p.294-302 [Periódico revisado por pares]

Amsterdam: Elsevier Inc

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2
Curcumin and hesperidin improve cognition by suppressing mitochondrial dysfunction and apoptosis induced by D-galactose in rat brain
Curcumin and hesperidin improve cognition by suppressing mitochondrial dysfunction and apoptosis induced by D-galactose in rat brain
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Curcumin and hesperidin improve cognition by suppressing mitochondrial dysfunction and apoptosis induced by D-galactose in rat brain

Banji, Otilia J.F. ; Banji, David ; Ch, Kalpana

Food and chemical toxicology, 2014-12, Vol.74, p.51-59 [Periódico revisado por pares]

Oxford: Elsevier Ltd

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3
2,3,4,7,8-Pentachlorodibenzofuran is far less potent than 2,3,7,8-tetrachlorodibenzo-p-dioxin in disrupting the pituitary–gonad axis of the rat fetus
2,3,4,7,8-Pentachlorodibenzofuran is far less potent than 2,3,7,8-tetrachlorodibenzo-p-dioxin in disrupting the pituitary–gonad axis of the rat fetus
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2,3,4,7,8-Pentachlorodibenzofuran is far less potent than 2,3,7,8-tetrachlorodibenzo-p-dioxin in disrupting the pituitary–gonad axis of the rat fetus

Taura, Junki ; Takeda, Tomoki ; Fujii, Misaki ; Hattori, Yukiko ; Ishii, Yuji ; Kuroki, Hiroaki ; Tsukimori, Kiyomi ; Uchi, Hiroshi ; Furue, Masutaka ; Yamada, Hideyuki

Toxicology and applied pharmacology, 2014-11, Vol.281 (1), p.48-57 [Periódico revisado por pares]

Amsterdam: Elsevier Inc

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4
Ototoxicity of paclitaxel in rat cochlear organotypic cultures
Ototoxicity of paclitaxel in rat cochlear organotypic cultures
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Ototoxicity of paclitaxel in rat cochlear organotypic cultures

Dong, Yang ; Ding, Dalian ; Jiang, Haiyan ; Shi, Jian-rong ; Salvi, Richard ; Roth, Jerome A.

Toxicology and applied pharmacology, 2014-11, Vol.280 (3), p.526-533 [Periódico revisado por pares]

Amsterdam: Elsevier Inc

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5
Inhibition of neutral sphingomyelinase decreases elevated levels of inducible nitric oxide synthase and apoptotic cell death in ocular hypertensive rats
Inhibition of neutral sphingomyelinase decreases elevated levels of inducible nitric oxide synthase and apoptotic cell death in ocular hypertensive rats
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Inhibition of neutral sphingomyelinase decreases elevated levels of inducible nitric oxide synthase and apoptotic cell death in ocular hypertensive rats

Aslan, Mutay ; Basaranlar, Goksun ; Unal, Mustafa ; Ciftcioglu, Akif ; Derin, Narin ; Mutus, Bulent

Toxicology and applied pharmacology, 2014-11, Vol.280 (3), p.389-398 [Periódico revisado por pares]

Amsterdam: Elsevier Inc

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6
Increased CD147 (EMMPRIN) expression in the rat brain following traumatic brain injury
Increased CD147 (EMMPRIN) expression in the rat brain following traumatic brain injury
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Increased CD147 (EMMPRIN) expression in the rat brain following traumatic brain injury

Wei, Ming ; Li, Hong ; Shang, Yanguo ; Zhou, Ziwei ; Zhang, Jianning

Brain research, 2014-10, Vol.1585, p.150-158 [Periódico revisado por pares]

Amsterdam: Elsevier B.V

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7
Frequency matters: beta-band subthalamic nucleus deep-brain stimulation induces Parkinsonian-like blink abnormalities in normal rats
Frequency matters: beta-band subthalamic nucleus deep-brain stimulation induces Parkinsonian-like blink abnormalities in normal rats
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Frequency matters: beta-band subthalamic nucleus deep-brain stimulation induces Parkinsonian-like blink abnormalities in normal rats

Kaminer, Jaime ; Thakur, Pratibha ; Evinger, Craig

The European journal of neuroscience, 2014-10, Vol.40 (8), p.3237-3242 [Periódico revisado por pares]

Oxford: Blackwell Publishing Ltd

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8
A Platform for Controlled Dual-Drug Delivery to the Retina: Protective Effects against Light-Induced Retinal Damage in Rats
A Platform for Controlled Dual-Drug Delivery to the Retina: Protective Effects against Light-Induced Retinal Damage in Rats
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A Platform for Controlled Dual-Drug Delivery to the Retina: Protective Effects against Light-Induced Retinal Damage in Rats

Nagai, Nobuhiro ; Kaji, Hirokazu ; Onami, Hideyuki ; Katsukura, Yuki ; Ishikawa, Yumi ; Nezhad, Zhaleh Kashkouli ; Sampei, Kaori ; Iwata, Satoru ; Ito, Shuntaro ; Nishizawa, Matsuhiko ; Nakazawa, Toru ; Osumi, Noriko ; Mashima, Yukihiko ; Abe, Toshiaki

Advanced healthcare materials, 2014-10, Vol.3 (10), p.1555-1560 [Periódico revisado por pares]

Weinheim: Blackwell Publishing Ltd

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9
Enhanced visual responses in the superior colliculus in an animal model of attention-deficit hyperactivity disorder and their suppression by d -amphetamine
Enhanced visual responses in the superior colliculus in an animal model of attention-deficit hyperactivity disorder and their suppression by d -amphetamine
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Enhanced visual responses in the superior colliculus in an animal model of attention-deficit hyperactivity disorder and their suppression by d -amphetamine

Clements, K.M ; Devonshire, I.M ; Reynolds, J.N.J ; Overton, P.G

Neuroscience, 2014-08, Vol.274, p.289-298 [Periódico revisado por pares]

Amsterdam: Elsevier Ltd

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10
Prevention of NKCC1 phosphorylation avoids downregulation of KCC2 in central sensory pathways and reduces neuropathic pain after peripheral nerve injury
Prevention of NKCC1 phosphorylation avoids downregulation of KCC2 in central sensory pathways and reduces neuropathic pain after peripheral nerve injury
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Prevention of NKCC1 phosphorylation avoids downregulation of KCC2 in central sensory pathways and reduces neuropathic pain after peripheral nerve injury

Mòdol, Laura ; Cobianchi, Stefano ; Navarro, Xavier

Pain (Amsterdam), 2014-08, Vol.155 (8), p.1577-1590 [Periódico revisado por pares]

Philadelphia, PA: Elsevier B.V

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