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Refinado por: assunto: Neurochemistry remover assunto: Neurology remover
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1
1-(7-Chloroquinolin-4-yl)-N-(4-Methoxybenzyl)-5-Methyl-1H-1,2, 3-Triazole-4- carboxamide Reduces Aβ Formation and Tau Phosphorylation in Cellular Models of Alzheimer’s Disease
1-(7-Chloroquinolin-4-yl)-N-(4-Methoxybenzyl)-5-Methyl-1H-1,2, 3-Triazole-4- carboxamide Reduces Aβ Formation and Tau Phosphorylation in Cellular Models of Alzheimer’s Disease
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1-(7-Chloroquinolin-4-yl)-N-(4-Methoxybenzyl)-5-Methyl-1H-1,2, 3-Triazole-4- carboxamide Reduces Aβ Formation and Tau Phosphorylation in Cellular Models of Alzheimer’s Disease

Fronza, Mariana G. ; Sacramento, Manoela ; Alves, Diego ; Praticò, Domenico ; Savegnago, Lucielli

Neurochemical research, 2022-04, Vol.47 (4), p.1110-1122 [Periódico revisado por pares]

New York: Springer US

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2
1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat
1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat
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1-Benzyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neurotoxic Compound, Disturbs the Behavioral and Biochemical Effects of l-DOPA: In Vivo and Ex Vivo Studies in the Rat

Wąsik, Agnieszka ; Romańska, Irena ; Michaluk, Jerzy ; Kajta, Małgorzata ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2014-10, Vol.26 (3), p.240-254 [Periódico revisado por pares]

Boston: Springer US

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3
1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies
1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies
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1-Benzyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Parkinsonism-Inducing Toxin, Strongly Potentiates MAO-Dependent Dopamine Oxidation and Impairs Dopamine Release: Ex vivo and In vivo Neurochemical Studies

Wąsik, Agnieszka ; Romańska, Irena ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2009, Vol.15 (1), p.15-23 [Periódico revisado por pares]

New York: Springer-Verlag

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4
1-Methyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Amine with Unexpected Mechanism of Action: New Vistas of Therapeutic Application
1-Methyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Amine with Unexpected Mechanism of Action: New Vistas of Therapeutic Application
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1-Methyl-1,2,3,4-Tetrahydroisoquinoline, an Endogenous Amine with Unexpected Mechanism of Action: New Vistas of Therapeutic Application

Antkiewicz-Michaluk, Lucyna ; Wąsik, Agnieszka ; Michaluk, Jerzy

Neurotoxicity research, 2014-01, Vol.25 (1), p.1-12 [Periódico revisado por pares]

Boston: Springer US

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5
1-Methyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neuroprotectant and MAO Inhibitor with Antidepressant-Like Properties in the Rat
1-Methyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neuroprotectant and MAO Inhibitor with Antidepressant-Like Properties in the Rat
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1-Methyl-1,2,3,4-tetrahydroisoquinoline, an Endogenous Neuroprotectant and MAO Inhibitor with Antidepressant-Like Properties in the Rat

Wąsik, Agnieszka ; Możdżeń, Edyta ; Michaluk, Jerzy ; Romańska, Irena ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2014-05, Vol.25 (4), p.323-334 [Periódico revisado por pares]

Boston: Springer US

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6
1-Methyl-1,2,3,4-tetrahydroisoquinoline Antagonizes a Rise in Brain Dopamine Metabolism, Glutamate Release in Frontal Cortex and Locomotor Hyperactivity Produced by MK-801 but not the Disruptions of Prepulse Inhibition, and Impairment of Working Memory in Rat
1-Methyl-1,2,3,4-tetrahydroisoquinoline Antagonizes a Rise in Brain Dopamine Metabolism, Glutamate Release in Frontal Cortex and Locomotor Hyperactivity Produced by MK-801 but not the Disruptions of Prepulse Inhibition, and Impairment of Working Memory in Rat
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1-Methyl-1,2,3,4-tetrahydroisoquinoline Antagonizes a Rise in Brain Dopamine Metabolism, Glutamate Release in Frontal Cortex and Locomotor Hyperactivity Produced by MK-801 but not the Disruptions of Prepulse Inhibition, and Impairment of Working Memory in Rat

Pietraszek, Małgorzata ; Michaluk, Jerzy ; Romańska, Irena ; Wąsik, Agnieszka ; Gołembiowska, Krystyna ; Antkiewicz-Michaluk, Lucyna

Neurotoxicity research, 2009-11, Vol.16 (4), p.390-407 [Periódico revisado por pares]

New York: Springer-Verlag

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7
1-Methyl-4-Phenylpyridinium-Induced Cell Death via Autophagy Through a Bcl-2/Beclin 1 Complex-Dependent Pathway
1-Methyl-4-Phenylpyridinium-Induced Cell Death via Autophagy Through a Bcl-2/Beclin 1 Complex-Dependent Pathway
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1-Methyl-4-Phenylpyridinium-Induced Cell Death via Autophagy Through a Bcl-2/Beclin 1 Complex-Dependent Pathway

Nopparat, Chutikorn ; Porter, James E. ; Ebadi, Manuchair ; Govitrapong, Piyarat

Neurochemical research, 2014-02, Vol.39 (2), p.225-232 [Periódico revisado por pares]

Boston: Springer US

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8
1-Trichloromethyl-1,2,3,4-tetrahydro-beta-carboline (TaClo) Alters Cell Cycle Progression in Human Neuroblastoma Cell Lines
1-Trichloromethyl-1,2,3,4-tetrahydro-beta-carboline (TaClo) Alters Cell Cycle Progression in Human Neuroblastoma Cell Lines
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1-Trichloromethyl-1,2,3,4-tetrahydro-beta-carboline (TaClo) Alters Cell Cycle Progression in Human Neuroblastoma Cell Lines

Sharma, Rakesh Kumar ; Candelario-Jalil, Eduardo ; Feineis, Doris ; Bringmann, Gerhard ; Fiebich, Bernd L. ; Akundi, Ravi Shankar

Neurotoxicity research, 2017-11, Vol.32 (4), p.649-660 [Periódico revisado por pares]

New York: Springer US

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9
11β-HSD1, Inflammation, Metabolic Disease and Age-related Cognitive (dys)Function
11β-HSD1, Inflammation, Metabolic Disease and Age-related Cognitive (dys)Function
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11β-HSD1, Inflammation, Metabolic Disease and Age-related Cognitive (dys)Function

Chapman, Karen E. ; Seckl, Jonathan R.

Neurochemical research, 2008-04, Vol.33 (4), p.624-636 [Periódico revisado por pares]

Boston: Springer US

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10
14-3-3 Isoforms Differentially Regulate NFκB Signaling in the Brain After Ischemia-Reperfusion
14-3-3 Isoforms Differentially Regulate NFκB Signaling in the Brain After Ischemia-Reperfusion
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14-3-3 Isoforms Differentially Regulate NFκB Signaling in the Brain After Ischemia-Reperfusion

Zhou, Xin-yu ; Hu, Dian Xing ; Chen, Ruo Qiao ; Chen, Xiao Qian ; Dong, Wan-li ; Yi, Cheng-la

Neurochemical research, 2017-08, Vol.42 (8), p.2354-2362 [Periódico revisado por pares]

New York: Springer US

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Deste Autor:

  1. Cowan, W
  2. Schwartz, J
  3. Kandel, E
  4. Jessell, T
  5. Williams, S

Neste Assunto:

  1. Neurology
  2. Neurochemistry
  3. Neurology
  4. Neurochemistry
  5. Neurosciences

  • Realização: ABCD-USP USP   Logos de Redes Sociais: Freepik

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