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Cellular and viral Fos proteins are degraded by different proteolytic systems
Cellular and viral Fos proteins are degraded by different proteolytic systems
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Cellular and viral Fos proteins are degraded by different proteolytic systems

ACQUAVIVA, Claire ; SALVAT, Cathy ; BROCKLY, Frédérique ; BOSSIS, Guillaume ; FERRARA, Patrizia ; PIECHACZYK, Marc ; JARIEL-ENCONTRE, Isabelle

Oncogene, 2001-02, Vol.20 (8), p.942-950 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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2
Mitogen regulated induction of FRA-1 proto-oncogene is controlled by the transcription factors binding to both serum and TPA response elements
Mitogen regulated induction of FRA-1 proto-oncogene is controlled by the transcription factors binding to both serum and TPA response elements
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Mitogen regulated induction of FRA-1 proto-oncogene is controlled by the transcription factors binding to both serum and TPA response elements

ADISESHAIAH, Pavan ; PEDDAKAMA, Suneetha ; QIN ZHANG ; KALVAKOLANU, Dhanunjaya V ; REDDY, Sekhar P

Oncogene, 2005-06, Vol.24 (26), p.4193-4205 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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3
Expression of a truncated 100 kDa HER2 splice variant acts as an endogenous inhibitor of tumour cell proliferation
Expression of a truncated 100 kDa HER2 splice variant acts as an endogenous inhibitor of tumour cell proliferation
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Expression of a truncated 100 kDa HER2 splice variant acts as an endogenous inhibitor of tumour cell proliferation

AIGNER, Achim ; JUHL, Hartmut ; MALERCZYK, Claudius ; TKYBUSCH, Anja ; BENZ, Christopher C ; CZUBAYKO, Frank

Oncogene, 2001-04, Vol.20 (17), p.2101-2111 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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4
The ability of Fos family members to produce phenotypic changes in epithelioid cells is not directly linked to their transactivation potentials
The ability of Fos family members to produce phenotypic changes in epithelioid cells is not directly linked to their transactivation potentials
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The ability of Fos family members to produce phenotypic changes in epithelioid cells is not directly linked to their transactivation potentials

ANDERSEN, Henriette ; MAHMOOD, Shaukat ; TKACH, Vadim ; COHN, Martin ; KUSTIKOVA, Olga ; GRIGORIAN, Mariam ; BEREZIN, Vladimir ; BOCK, Elisabeth ; LUKANIDIN, Eugene ; TULCHINSKY, Eugene

Oncogene, 2002-07, Vol.21 (31), p.4843-4848 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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5
Function and regulation of AP-1 subunits in skin physiology and pathology: AP-1
Function and regulation of AP-1 subunits in skin physiology and pathology: AP-1
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Function and regulation of AP-1 subunits in skin physiology and pathology: AP-1

ANGEL, Peter ; SZABOWSKI, Axel ; SCHORPP-KISTNER, Marina

Oncogene, 2001-04, Vol.20 (19), p.2413-2423 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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6
Functions of Fos phosphorylation in bone homeostasis, cytokine response and tumourigenesis
Functions of Fos phosphorylation in bone homeostasis, cytokine response and tumourigenesis
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Functions of Fos phosphorylation in bone homeostasis, cytokine response and tumourigenesis

BAKIRI, L ; RESCHKE, M. O ; GEFROH, H. A ; IDARRAGA, M. H ; POLZER, K ; ZENZ, R ; SCHETT, G ; WAGNER, E. F

Oncogene, 2011-03, Vol.30 (13), p.1506-1517 [Periódico revisado por pares]

Basingstoke: Nature Publishing Group

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7
Sustained MAP kinase activation is required for the expression of cyclin D1, p21Cip1 and a subset of AP-1 proteins in CCL39 cells
Sustained MAP kinase activation is required for the expression of cyclin D1, p21Cip1 and a subset of AP-1 proteins in CCL39 cells
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Sustained MAP kinase activation is required for the expression of cyclin D1, p21Cip1 and a subset of AP-1 proteins in CCL39 cells

BALMANNO, K ; COOK, S. J

Oncogene, 1999-05, Vol.18 (20), p.3085-3097 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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8
Increase in Ap-1 activity is a general event in thyroid cell transformation in vitro and in vivo
Increase in Ap-1 activity is a general event in thyroid cell transformation in vitro and in vivo
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Increase in Ap-1 activity is a general event in thyroid cell transformation in vitro and in vivo

BATTISTA, S ; DE NIGRIS, F ; VERDE, P ; FUSCO, A ; FEDELE, M ; CHIAPPETTA, G ; SCALA, S ; VALLONE, D ; PIERANTONI, G. M ; MEGAR, T ; SANTORO, M ; VIGLIETTO, G

Oncogene, 1998-07, Vol.17 (3), p.377-385 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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9
Changes in WT1 splicing are associated with a specific gene expression profile in Wilms' tumour
Changes in WT1 splicing are associated with a specific gene expression profile in Wilms' tumour
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Changes in WT1 splicing are associated with a specific gene expression profile in Wilms' tumour

BAUDRY, Dominique ; FAUSSILLON, Marine ; CABANIS, Marie-Odile ; RIGOLET, Muriel ; ZUCKER, Jean-Michel ; PATTE, Catherine ; SARNACKI, Sabine ; BOCCON-GIBOD, Liliane ; JUNIEN, Claudine ; JEANPIERRE, Cécile

Oncogene, 2002-08, Vol.21 (36), p.5566-5573 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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10
An unexpected role for FosB in activation-induced cell death of T cells
An unexpected role for FosB in activation-induced cell death of T cells
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An unexpected role for FosB in activation-induced cell death of T cells

BAUMANN, Sven ; HESS, Jochen ; EICHHORST, Sören T ; KRUEGER, Andreas ; ANGEL, Peter ; KRAMMER, Peter H ; KIRCHHOFF, Sabine

Oncogene, 2003-03, Vol.22 (9), p.1333-1339 [Periódico revisado por pares]

Basingstoke: Nature Publishing

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