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1
1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1H-pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion
1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1H-pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion
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1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1H-pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion

Klose, Angelika ; Huth, Tobias ; Alzheimer, Christian

Molecular pharmacology, 2008-11, Vol.74 (5), p.1203-1214 [Periódico revisado por pares]

United States: American Society for Pharmacology and Experimental Therapeutics

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2
1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1 H -pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion
1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1 H -pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion
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1-[6-[[(17β)-3-Methoxyestra-1,3,5(10)-trien-17-yl]amino]hexyl]-1 H -pyrrole-2,5-dione (U73122) Selectively Inhibits Kir3 and BK Channels in a Phospholipase C-Independent Fashion

Klose, Angelika ; Huth, Tobias ; Alzheimer, Christian

Molecular pharmacology, 2008-11, Vol.74 (5), p.1203-1214 [Periódico revisado por pares]

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3
1-beta-D-arabinofuranosylcytosine activates serine/threonine protein kinases and c-jun gene expression in phorbol ester-resistant myeloid leukemia cells
1-beta-D-arabinofuranosylcytosine activates serine/threonine protein kinases and c-jun gene expression in phorbol ester-resistant myeloid leukemia cells
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1-beta-D-arabinofuranosylcytosine activates serine/threonine protein kinases and c-jun gene expression in phorbol ester-resistant myeloid leukemia cells

Kharbanda, S ; Emoto, Y ; Kisaki, H ; Saleem, A ; Kufe, D

Molecular pharmacology, 1994-07, Vol.46 (1), p.67-72 [Periódico revisado por pares]

United States: American Society for Pharmacology and Experimental Therapeutics

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4
1-beta-D-arabinofuranosylcytosine enhancement of resistance to several antineoplastic drugs in mammalian tissue culture cells
1-beta-D-arabinofuranosylcytosine enhancement of resistance to several antineoplastic drugs in mammalian tissue culture cells
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1-beta-D-arabinofuranosylcytosine enhancement of resistance to several antineoplastic drugs in mammalian tissue culture cells

GOZ, B ; CARL, P. L ; TLSTY, T. D

Molecular pharmacology, 1989-09, Vol.36 (3), p.360-365 [Periódico revisado por pares]

Bethesda, MD: American Society for Pharmacology and Experimental Therapeutics

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5
1-beta-D-arabinosyl-5-azacytosine. Cytocidal activity and effects on the synthesis and methylation of DNA in human colon carcinoma cells
1-beta-D-arabinosyl-5-azacytosine. Cytocidal activity and effects on the synthesis and methylation of DNA in human colon carcinoma cells
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1-beta-D-arabinosyl-5-azacytosine. Cytocidal activity and effects on the synthesis and methylation of DNA in human colon carcinoma cells

GLAZER, R. I ; KNODE, M. C

Molecular pharmacology, 1984-09, Vol.26 (2), p.381-387 [Periódico revisado por pares]

Bethesda, MD: American Society for Pharmacology and Experimental Therapeutics

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6
1-Hydroxyethyl radical formation during NADPH- and NADH-dependent oxidation of ethanol by human liver microsomes
1-Hydroxyethyl radical formation during NADPH- and NADH-dependent oxidation of ethanol by human liver microsomes
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1-Hydroxyethyl radical formation during NADPH- and NADH-dependent oxidation of ethanol by human liver microsomes

Rao, D N ; Yang, M X ; Lasker, J M ; Cederbaum, A I

Molecular pharmacology, 1996-05, Vol.49 (5), p.814-821 [Periódico revisado por pares]

United States: American Society for Pharmacology and Experimental Therapeutics

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7
1-Methyl-4-Phenylpyridinium Induces Autocrine Excitotoxicity, Protease Activation, and Neuronal Apoptosis
1-Methyl-4-Phenylpyridinium Induces Autocrine Excitotoxicity, Protease Activation, and Neuronal Apoptosis
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1-Methyl-4-Phenylpyridinium Induces Autocrine Excitotoxicity, Protease Activation, and Neuronal Apoptosis

Leist, M ; Volbracht, C ; Fava, E ; Nicotera, P

Molecular pharmacology, 1998-11, Vol.54 (5), p.789-801 [Periódico revisado por pares]

United States: American Society for Pharmacology and Experimental Therapeutics

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8
1,1-Bis(3'-indolyl)-1-(p-substitutedphenyl)methanes are peroxisome proliferator-activated receptor gamma agonists but decrease HCT-116 colon cancer cell survival through receptor-independent activation of early growth response-1 and nonsteroidal anti-inflammatory drug-activated gene-1
1,1-Bis(3'-indolyl)-1-(p-substitutedphenyl)methanes are peroxisome proliferator-activated receptor gamma agonists but decrease HCT-116 colon cancer cell survival through receptor-independent activation of early growth response-1 and nonsteroidal anti-inflammatory drug-activated gene-1
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1,1-Bis(3'-indolyl)-1-(p-substitutedphenyl)methanes are peroxisome proliferator-activated receptor gamma agonists but decrease HCT-116 colon cancer cell survival through receptor-independent activation of early growth response-1 and nonsteroidal anti-inflammatory drug-activated gene-1

Chintharlapalli, Sudhakar ; Papineni, Sabitha ; Baek, Seung Joon ; Liu, Shengxi ; Safe, Stephen

Molecular pharmacology, 2005-12, Vol.68 (6), p.1782-1792 [Periódico revisado por pares]

United States

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9
1,1-bis(3'-indolyl)-1-(p-substitutedphenyl)methanes inhibit growth, induce apoptosis, and decrease the androgen receptor in LNCaP prostate cancer cells through peroxisome proliferator-activated receptor gamma-independent pathways
1,1-bis(3'-indolyl)-1-(p-substitutedphenyl)methanes inhibit growth, induce apoptosis, and decrease the androgen receptor in LNCaP prostate cancer cells through peroxisome proliferator-activated receptor gamma-independent pathways
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1,1-bis(3'-indolyl)-1-(p-substitutedphenyl)methanes inhibit growth, induce apoptosis, and decrease the androgen receptor in LNCaP prostate cancer cells through peroxisome proliferator-activated receptor gamma-independent pathways

Chintharlapalli, Sudhakar ; Papineni, Sabitha ; Safe, Stephen

Molecular pharmacology, 2007-02, Vol.71 (2), p.558-569 [Periódico revisado por pares]

United States

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10
1,1-Bis(3′-indolyl)-1-(p-substitutedphenyl)methanes Are Peroxisome Proliferator-Activated Receptor γ Agonists but Decrease HCT-116 Colon Cancer Cell Survival through Receptor-Independent Activation of Early Growth Response-1 and Nonsteroidal Anti-Inflammatory Drug-Activated Gene-1
1,1-Bis(3′-indolyl)-1-(p-substitutedphenyl)methanes Are Peroxisome Proliferator-Activated Receptor γ Agonists but Decrease HCT-116 Colon Cancer Cell Survival through Receptor-Independent Activation of Early Growth Response-1 and Nonsteroidal Anti-Inflammatory Drug-Activated Gene-1
Material Type:
Artigo 		Adicionar ao Meu Espaço

1,1-Bis(3′-indolyl)-1-(p-substitutedphenyl)methanes Are Peroxisome Proliferator-Activated Receptor γ Agonists but Decrease HCT-116 Colon Cancer Cell Survival through Receptor-Independent Activation of Early Growth Response-1 and Nonsteroidal Anti-Inflammatory Drug-Activated Gene-1

Sudhakar Chintharlapalli ; Sabitha Papineni ; Seung Joon Baek ; Shengxi Liu ; Stephen Safe

Molecular pharmacology, 2005-12, Vol.68 (6), p.1782 [Periódico revisado por pares]

American Society for Pharmacology and Experimental Therapeutics

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Deste Autor:

  1. Fontana, A
  2. Coutinho Netto, J
  3. Beleboni, R
  4. Santos, W
  5. Wojewodzic, M

Neste Assunto:

  1. Pharmacology & Pharmacy
  2. Life Sciences & Biomedicine
  3. Science & Technology
  4. Animals
  5. Humans

  • Realização: ABCD-USP USP   Logos de Redes Sociais: Freepik

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